y the lengths of the neural skeleton, the deformity, usually discovered also inbony parts only "drive" by themselves (fig. ordinary achondroplasia.2, 3). A 7-year old boy sustained a trauma inthe dorsal interphalangeal lesion of theExperimental observationsthumb (fig. 9a). On the original film thereThe plan of development of may be just seen unevenness of the softmalformation (fig. 4) is the very same with parts. On the film 2 1/2-years later (fig. 9b)regard to the animal and man. The neural- we can see that the length of the digit hasextensive growth is somewhat s l o w e r not changed in spite of significantlythan the cellular-divisional and this makes, elongated first metacarpal and basalby way of cellulo-neural unevenness, a lot phalanx because the terminal phalanx wasof disorders of the congenital as well as almost at right angle bent dorsally. Whereasacquired lesions (the latter condition is this is hard to explain osteologically,treated by (9)). To disclose the nature of the neuroadaptivly it is quite easy tolesion one meticulously display to examine understand: the axis of the thumb is soin an extended form, it means with upper changed as too get into the neural axis, i.e.and lower ends far away since only in this into the axis of the visible nerves and thooseway we can recognize the cranio-caudal of invisible nervous skeleton (fig. 9b).way of <strong>pro</strong>duction (fig. 5, 6).Congenital malformations are relativlyAlso various malformations of the frog common and most often are shortness ofhinterlimb frequently may be <strong>pro</strong>duced by a longitunidal axis or obliqity with mostlysimple amputation (fig. 7a-l). The grave modifications of structure.patophysiologic considerations will be in Neuroadaptive changes are in any caseessence the same as in the other types of documented: in the first instancedevelopmental defects.achondroplasia-artig shortening withthickening (fig. 10a, b), in the otherMedical observationsshowing (fig. 10c) slanting n e r v e with theA 22-years old man at the age of 2 1/2 skeleton disposed secondarily, is longeryears suffered an electrical trauma of 2.-4. than the nerve but the bone adapts itself tobasal phalange and metacarpi (fig. 8). The the nerv, not the oposite.trauma was such as to be not too light (only Cleft palate is a crucial example ofin form of blisters on the skin) neither not to total ignorance of neuroadaptiveserious in form of a far reaching destruction mechanism. All posible mechanisms haveof involved parts. The involment was in the been taken in question as possible cause ofinvisible nervous skeleton so that the parts the defect, an journal devoted to specialcould develop further but less in length and topic exists but neuroadaptive explanationmore in width, i.e. we encounter a clear-cut has remaind an unknown matter.achondroplasic picture. Moreover, the Neuroadaptive mechanism is, however, theneural lesion interferred also with more only and elegant explanation (fig. 11).<strong>pro</strong>ximal part of the nerve so that the Acromegalic phalangeal dysplasia.pertaining skeleton was afflicted to, with Phalanx of an adult, consisting in thiningcollapse and bowing of bones of the wrist, the diaphysis with thickening of epiviz.,it appeared the typical Madelung metaphysis (fig. 12). Osteological20LOCOMOTOR SYSTEM VOL. 7, <strong>2000</strong>, No.1
Fig. 10. Congenital malformations. Achondroplasic shortening (a, b), slanting directed driftof the nerve and nervous skeleton (c) is the truth cause of the condition.Fig. 11. Cleft palate is a crucial example of total ignorance of neuroadaptive mechanism.All posible mechanisms have been taken in question, as possible cause of the defect, anjournal devoted to special topic exists but neuroadaptive explanation has remaind anunknown matter. Neuroadaptive mechanism is, however, the only and elegant explanation.Fig. 12a, b,c. Aged acromegalic with displastic alteration of basal phalangae, viz., thinningof diaphysis and thickening of epi-metaphysis. Survey view (a) and detail, normal andpathological view (b,c).1112aPOHYBOVÉ ÚSTROJÍ, ročník 7, <strong>2000</strong>, č. 1 21
- Page 1 and 2: POHYBOVÉ ÚSTROJÍročník 7, 2000
- Page 3 and 4: POHYBOVÉ LOCOMOTORÚSTROJÍSYSTEM1
- Page 5 and 6: SLOVO ČTENÁŘŮMVážení čtená
- Page 7 and 8: Putnama, aby se připojil k expedic
- Page 9 and 10: názoru, že pleistocénní fauna m
- Page 11 and 12: Bristolský záliv a ostrov Kodiak.
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- Page 15 and 16: PŮVODNÍ PRÁCE * ORIGINAL PAPERNE
- Page 17 and 18: Fig. 4a-d.Succession of diagrammati
- Page 19: Fig. 9a-c. Trauma of the thumb in a
- Page 23 and 24: Copeia, 1987/2, p. 489-491.I.Band.
- Page 25 and 26: leukograms were estimated. From the
- Page 27 and 28: Obr. 1. Leukogram 3., 5. a 11. den
- Page 29 and 30: Obr. 4. Polotenký řez synoviáln
- Page 31 and 32: Tabulka č.2. Hladiny sledovaných
- Page 33 and 34: Z těchto skutečností vychází i
- Page 35 and 36: implanted into the cartilage defect
- Page 37 and 38: electrophoresis: collagen of a part
- Page 39 and 40: Fig.5. The defect extendedinto the
- Page 41 and 42: 41. 911-15.7. Hascall V C, Sajdera
- Page 43 and 44: some object, for example pliers wit
- Page 45 and 46: amena jsou pro flexor (počínaje o
- Page 47 and 48: Po ukončení iteračního vypočtu
- Page 49 and 50: Obr. 2. Poloha os článků prstů
- Page 51 and 52: 4. Síly v kloubech a šlachách ro
- Page 53 and 54: sx1=N +Ar1MzIr1Obr.4. Řez kostí
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- Page 57 and 58: zdravotní nakladatelství, Praha 1
- Page 59 and 60: KONFERENCE * CONFERENCESYMPOSIUM
- Page 61 and 62: postižených trvalými následky,
- Page 63 and 64: ZPRÁVYZPRÁVA O ČINNOSTI SPOLEČN
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- Page 67 and 68: (Revmatologický ústav, Praha).( U
- Page 69 and 70: RECENZE * NEW BOOKSSmrčka V, Dylev
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potřeby. Kdo potřeboval skutečn
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SMĚRNICE PRO AUTORY PŘÍSPĚVKŮT
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INSTRUCTIONS FOR AUTHORSSubject Mat
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A5 (188x120mm)- zadní strana obál
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- ortopedická protetikaVysokoúči