1/2000 - Společnost pro pojivové tkáně

y the lengths of the neural skeleton, the deformity, usually discovered also inbony parts only "drive" by themselves (fig. ordinary achondroplasia.2, 3). A 7-year old boy sustained a trauma inthe dorsal interphalangeal lesion of theExperimental observationsthumb (fig. 9a). On the original film thereThe plan of development of may be just seen unevenness of the softmalformation (fig. 4) is the very same with parts. On the film 2 1/2-years later (fig. 9b)regard to the animal and man. The neural- we can see that the length of the digit hasextensive growth is somewhat s l o w e r not changed in spite of significantlythan the cellular-divisional and this makes, elongated first metacarpal and basalby way of cellulo-neural unevenness, a lot phalanx because the terminal phalanx wasof disorders of the congenital as well as almost at right angle bent dorsally. Whereasacquired lesions (the latter condition is this is hard to explain osteologically,treated by (9)). To disclose the nature of the neuroadaptivly it is quite easy tolesion one meticulously display to examine understand: the axis of the thumb is soin an extended form, it means with upper changed as too get into the neural axis, i.e.and lower ends far away since only in this into the axis of the visible nerves and thooseway we can recognize the cranio-caudal of invisible nervous skeleton (fig. 9b).way of production (fig. 5, 6).Congenital malformations are relativlyAlso various malformations of the frog common and most often are shortness ofhinterlimb frequently may be produced by a longitunidal axis or obliqity with mostlysimple amputation (fig. 7a-l). The grave modifications of structure.patophysiologic considerations will be in Neuroadaptive changes are in any caseessence the same as in the other types of documented: in the first instancedevelopmental defects.achondroplasia-artig shortening withthickening (fig. 10a, b), in the otherMedical observationsshowing (fig. 10c) slanting n e r v e with theA 22-years old man at the age of 2 1/2 skeleton disposed secondarily, is longeryears suffered an electrical trauma of 2.-4. than the nerve but the bone adapts itself tobasal phalange and metacarpi (fig. 8). The the nerv, not the oposite.trauma was such as to be not too light (only Cleft palate is a crucial example ofin form of blisters on the skin) neither not to total ignorance of neuroadaptiveserious in form of a far reaching destruction mechanism. All posible mechanisms haveof involved parts. The involment was in the been taken in question as possible cause ofinvisible nervous skeleton so that the parts the defect, an journal devoted to specialcould develop further but less in length and topic exists but neuroadaptive explanationmore in width, i.e. we encounter a clear-cut has remaind an unknown matter.achondroplasic picture. Moreover, the Neuroadaptive mechanism is, however, theneural lesion interferred also with more only and elegant explanation (fig. 11).proximal part of the nerve so that the Acromegalic phalangeal dysplasia.pertaining skeleton was afflicted to, with Phalanx of an adult, consisting in thiningcollapse and bowing of bones of the wrist, the diaphysis with thickening of epiviz.,it appeared the typical Madelung metaphysis (fig. 12). Osteological20LOCOMOTOR SYSTEM VOL. 7, 2000, No.1