nara lins meira quintão análise dos mecanismos envolvidos na ...

PAUL, A.T.; GOHIL, V.M.; BHUTANI, K.K.. Modulating TNF-α signaling with natural
products. Drug Disc. Today. v. 11, n. 15, p. 725-732, 2006.
PEREZ, C.; ALBERT, I.; DEFAY, K.; ZACHARIADES, N.; GOODING, L.; KRIEGLER,
M.. A nonsecretable cell surface mutant of tumor necrosis factor (TNF) kills by cellto-cell
contact. Cell. v. 63, p. 251-258, 1990.
PERKINS, M.N.; CAMPBELL, E.; DRAY, A.. Antinociceptive activity of the bradykinin
B1 and B2 receptor antagonists, des-arg 9 ,[leu 8 ]-bk and hoe140, in two models of
persistent hyperalgesia in the rat. Pain. v. 51, p. 191-197, 1993.
PERKINS, N.M. e TRACEY, D.J.. Hyperalgesia due to nerve injury: role of neutrophils.
Neurosci. v. 101, n. 3, p. 745-757, 2000.
PERRY, D.C. e SNIDER, S.H.. Identification of bradykinin in mammalian brain. J.
Neurochem. v. 43, p. 1072-1080, 1984.
PESQUERO, J.B.; ARAUJO, R.C.; HEPPENSTALL, P.A.; STUCKY, C.L.; SILVA, J.A.
JR; WALTHER, T.; OLIVEIRA, S.M.; PESQUERO, J.L.; PAIVA, A.C.; CALIXTO,
J.B.; LEWIN, G.R.; BADER, M.. Hypoalgesia and altered inflammatory responses in
mice lacking kinin B1 receptors. Proc. Natl. Acad. Sci. USA. v. 97, p. 8140–8145,
2000.
PETERSEN, M.; ECKERT, A.S.; SEGOND VON BANCHET, G.; HEPPELMANN, B.;
KLUSCH, A.; KNIFFKI, K.-D.. Plasticity in the expression of bradykinin binding sites
in the sensory neurons after mechanical nerve injury. Neurosci. v. 83, n. 3, p. 949-
959, 1998.
PETERSEN, M.; SEGOND VON BANCHET, G.; HEPPELMANN, B.; KOLTZENBURG,
M.. NGF regulates the expression of bradykinin biding sites on adult sensory
neurons via the neurotrophin receptor p75. Neurosci. v. 83, p. 161-168, 1996.
PEZET, S.; MALCANGIO, M.; MCMAHON, S.B.. BDNF: a neuromodulator in
nociceptive pathways? Brain Res. Rev. v. 40, p. 240-249, 2002.
PITCHER, G.M.; RITCHIE, J.; HENRY, J.L.. Nerve constriction in the rat: model of
neuropathic, surgical and central pain. Pain. v. 83, p. 37-46, 1999.
PRADO, G.N.; TAYLOR, L.; ZHOU, X.; RICUPERO, D.; MIERKE, D.F.; POLGAR, P..
Mechanisms regulating the expression, self-maintenance, and signaling-finction of
the bradykinin B2 and B1 receptors. J. Cell. Physiol. v. 193, p. 275-286, 2002.