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Quale forma di IRA è frequentemente associata a MOF/Sepsi? • L’intensità e la durata dell’ischemia è la causa del danno cellulare dell’epitelio del tubulo distale,reversibile o letale • Fattori emodinamici : sistema nervoso simpatico, angiotensina II, trombossano A2, leucotrieni, endotelina, adenosina • Danno del microcircolo: infiltrazione leucocitaria, selectine, integrine, ICAM,VCAM, IL-1, TNF-a • Aumento dell’escrezione frazionale del sodio
Sepsi: Stadi e definizioni Infezione Trauma SIRS • Risposta clinica a seguito di un insulto aspecifico, comprendente ≥ 2 dei seguenti criteri: – Temperatura > 38 o C o < 36 o C; – Frequenza Cardiaca > 90 bpm; – Frequenza respiratoria > 20/min o PaCO 2 < 32 mmHg; – Conta dei leucociti > 12.000/mm 3 o < 4.000/mm 3 o forme immature (cellule “a banda”) > 10%. Sepsi • SIRS con segni di infezione documentata o presunta. Sepsi Grave SIRS = sindrome della risposta infiammatoria sistemica. Bone e coll. Chest. 1992;101:1644.
Page 1 and 2: CORSO AVANZATO PER MEDICI Trattamen
Page 3: → Il flusso ematico renale rappre
Page 6 and 7: Factors that render the kidney susc
Page 8 and 9: IRA: definizione L’IRA IRA è una
Page 10 and 11: Incidenza dell’IRA dell IRA (Vinc
Page 12 and 13: August 28-30, 28 30, 2000 New York
Page 14 and 15: ORIGINAL INVESTIGATIONS The Outcome
Page 16 and 17: Hospital Mortality Rate in RRT Pati
Page 18 and 19: • IRA,frequente complicanza in IC
Page 20 and 21: DIAGNOSES IN 544 PATIENTS WITH ARF
Page 22 and 23: Acute Renal Failure in critically i
Page 24 and 25: M.E. Ostermann, D. Taube, C.J. Morg
Page 26 and 27: A higher level of knowledge needs t
Page 28 and 29: Vivino G, Antonelli M, Moro ML, Cot
Page 30 and 31: Risk factors increased by • Age
Page 32 and 33: Agents and Events That Ameliorate o
Page 34 and 35: Drugs Associated with Acute Renal F
Page 36 and 37: RI=0.63 Renal Protection through st
Page 38 and 39: A.de Mendonça, J.L. Vincent, P.M.
Page 40 and 41: • Infection … they concluded th
Page 42 and 43: De Mendonça A et al. Acute renal f
Page 44 and 45: • Advanced age • Altered previo
Page 48 and 49: Sepsi: Stadi e definizioni Infezion
Page 50 and 51: Arterial Vasodilatation and Renal V
Page 52 and 53: Effect of Membrane Potential on the
Page 54 and 55: good and bad effects of nitric oxid
Page 56 and 57: FORMATION OF LPS/PROTEIN COMPLEXES
Page 58 and 59: Tubular-Cell Injury and Repair in I
Page 60 and 61: ACTIVATION OF HUMORAL PATHWAY L P S
Page 62 and 63: A disorder due to incontrolled infl
Page 64 and 65: The pathogenesis of septic acute re
Page 66 and 67: Jo e colleghi hanno recentemente di
Page 68 and 69: APOPTOSIS
Page 70 and 71: Acute renal failure Norbert Lameire
Page 73: 600 500 400 300 200 100 0 PaO2/FiO2
Page 77 and 78: ml/die 8000 7000 6000 5000 4000 300
Page 79 and 80: mg/dL 700 600 500 400 300 200 100 0
Page 81 and 82: Lasciato a se stesso e non contrast
Page 83 and 84: Strategie di protezione
Page 85 and 86: SEPSI E IRA Rilascio citochine,atti
Page 87 and 88: Renal Perfusion Pressure: Pressure
Page 89 and 90: • Riempimento volemico per manten
Page 91 and 92: Acute phase: Patients who have dila
Page 93 and 94: Cardiovascular changes associated w
Page 95 and 96: Dellinger RP. Crit Care Med 2003;31
Page 97 and 98:
Edema removal by a dialysis techniq
Page 100:
“independent independent from an
Page 103 and 104:
…..guardando gli effetti biologic
Page 105 and 106:
APOPTOSIS IN SEPSIS PHVHF decreased
Page 107 and 108:
Terapia anti apoptosica? The Acute
Page 109 and 110:
(%) Survival Rate of Early Vs Late
Page 112 and 113:
EARLY ISOVOLEMIC HAEMOFILTRATION IN
Page 114 and 115:
- Trattamento extracorporeo del san
Page 116 and 117:
MOST Brain Liver mRNA/NfKb Cytokine
Page 118 and 119:
Early detection of ARF,especially i
Page 120 and 121:
. . Grazie per l’attenzione l att
Page 122 and 123:
The Response to Pathogens, Involvin
Page 125 and 126:
Immunologic Response of Three Hypot
Page 127 and 128:
Dal momento che la sepsi è un proc
Page 129:
IMMUNOMODULAZIONE : Il goal della M
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