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Utantio-PJK_CAD [Compatibility Mode].pdf - E-Lib FK UWKS
Utantio-PJK_CAD [Compatibility Mode].pdf - E-Lib FK UWKS Utantio-PJK_CAD [Compatibility Mode].pdf - E-Lib FK UWKS
Stage A : Endothelial injury Endotel intak : • Berfungsi sbg “BARRIER” menjamin aliran darah koroner lancar Faktor Risiko; akan memudahkan masuknya lipoprotein (LDL teroksidasi) ke dinding arteri maupun makrophag. Interaksi antara endothelial injury dg platelet, monocyte dan jaringan ikat (collagen) menyebabkan terjadi penempelan platelet (platelet adherence) dan agregrasi trombosit (platelet aggregation)
Stage B : Fatty Streak Formation CIRCULATING : TOTAL CHOLEST TOTAL CHOLEST MONOCYTE SUBENDOTHELIAL SPACE : FREE CHOLESTEROL OXIDIZED LDL FATTY STREAK MACROPHAGE FOAM CELL MONOCYTE
- Page 1 and 2: Penyakit Jantung Koroner = PJK Coro
- Page 3 and 4: CAB. ANT ATRIUM KA A. KOR. KA A. DE
- Page 5 and 6: Penyempitan P. Darah - Bertugas men
- Page 7 and 8: Aterosklerosis Koroner Arteri ~ pro
- Page 9 and 10: 4. Kurang bergerak (Lack of Exercis
- Page 11 and 12: Skema evolusi aterosklerosis korone
- Page 13: Teori : Proses aterosklerosis 1. Re
- Page 17 and 18: Stage D : Unstable Plaque Formation
- Page 19 and 20: Struktur Plak Stabil “FIBROUS PLA
- Page 21 and 22: PJK : FATTY STREAK FIBROUS PLAQUE T
- Page 23 and 24: Obat Trombus-Intrakoroner 1. Antitr
- Page 25 and 26: • Obat Trombolisis : - Streptokin
- Page 27 and 28: Infark Miokard Akut (IMA) • Kerus
- Page 29 and 30: Kriteria Diagnosis : (modified WHO
- Page 31 and 32: 3. Lama : 0,5 - 30 menit Spesifitas
- Page 34 and 35: a b c e f d EKG IMA
- Page 36 and 37: LOKASI INFARK : • Anteroseptal :
- Page 38 and 39: DIAGNOSIS & PENANGANAN DINI • Seg
- Page 40 and 41: Penanganan di Rumah Sakit • Penan
- Page 42 and 43: Indikasi Terapi Trombolitik • 1.
- Page 44 and 45: Terapi trombolitik : Kontraindikasi
- Page 46 and 47: Prinsip Terapi IMA : (Non Invasif)
- Page 48 and 49: PROGNOSIS : Tergantung 3 Faktor : 1
Stage A : Endothelial injury<br />
Endotel intak :<br />
• Berfungsi sbg “BARRIER”<br />
menjamin aliran darah koroner lancar<br />
Faktor Risiko; akan memudahkan masuknya lipoprotein<br />
(LDL teroksidasi) ke dinding arteri maupun makrophag.<br />
Interaksi antara endothelial injury dg platelet, monocyte<br />
dan jaringan ikat (collagen) menyebabkan terjadi<br />
penempelan platelet (platelet adherence) dan agregrasi<br />
trombosit (platelet aggregation)
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