26. Kay L, Jorgensen T. Epi<strong>de</strong>miology of upper dyspepsia in random population.Prevalence, inci<strong>de</strong>nce, natural history and risk factors. Scan J Gastroenterol 1994;29: 1-7.27. Yuyuan LI, Yuqiang NIE, Weihong SHA, Hua SU. The link between socialpsychosocial and functional dyspepsia: An epi<strong>de</strong>miological study. Chin Med J 2002;115: 1.082-1.084.28. Stanghellini V. Relationship between upper gastrointestinal symptoms andlifestyle, psychosocial factors and comorbidity in the general population: results fromthe Domestic/International Gastroenterology Surveillance Study (DIGEST). Scand JGastroenterol Suppl 1999; 231: 29-37.29. Pantoflickova D, Blum AL, Koelz HR. Helicobacter pylori and functionaldyspepsia: a real causal link? Bailleres Clin Gastroenterol 1998; 12: 503-532.30. Danesh J, Lawrence M, Murphy M, Roberts S, Collins R. Systematic review ofthe epi<strong>de</strong>miological evi<strong>de</strong>nce on Helicobacter pylori infection and nonulcer oruninvestigated dyspepsia. Arch Intern Med 2000; 160: 1.192-1.198.31. Bazzoli F, De Luca L, Pozzato P, Zagari RM, Fossi S, Ricciardiello L et al.Helicobacter pylori and functional dyspepsia: review of previous studies andcommentary on new data. Gut 2002; 50 (Supl. 4): 33-35.32. Elta GH, Behler EM, Colturi TJ. Comparison of coffee intake and coffee-inducedsymptoms in patients with duo<strong>de</strong>nal ulcer, nonulcer dyspepsia, and normal <strong>con</strong>trols.Am J Gastroenterol 1990; 85: 1.339-1.342.33. Nandurkar S, Talley NJ, Xia H, Mitchell H., Hazel S, Jones M. Dyspepsia in thecommunity is linked to smoking and aspirin use but not to Helicobacter pyloriinfection. Arch Intern Med 1998; 158: 1.427-1.433.34. Woodward M, Morrison CE, McColl Ke. The prevalence of dyspepsia and use ofantisecretory medication in North Glasgow: role of Helicobacter pylori vs. lifestylefactors. Aliment Pharmacology 1999; 13: 1.505-1.509.35. Biter P, Howell S, Lemon M, Young LJ, Jones MP, Talley NJ. Low socioe<strong>con</strong>omicclass is a risk factor for upper and lower gastrointestinal symptoms: a populationbased study in 15000 Australian adults. Gut 2001; 49: 66-67.36. Talley NJ, Weaver AL, Zinsmeister AR. Smoking, alcohol, and nonsteroidal antiinflammatorydrugs in outpatients with functional dyspepsia and among dyspepsiasubgroups. Am J Gastroenterol 1994; 89: 524-528.37. Talley NJ, McNeilD. Piper DW. Environmental factors and chronic unexplaineddyspepsia. Association with acetaminophen but not other analgesics, alcohol, coffee,tea, or smoking. Dig Dis Sci 1988; 33: 641-648.89
38. Kurata JH, Nogawa AN. Meta-analysis of risk factors for peptic ulcer.Nonsteroidal antiinflammatory drugs, Helicobacter pylori, and smoking. J ClinGastroenterol 1997; 24: 2-17. 39. Huang JQ, Sridhar S, Hunt RH. Role ofHelicobacter pylori infection and non-steroidal anti-inflammatory drugs in peptic-ulcerdisease: a meta-analysis. Lancet 2002; 359: 14-22.40. Chan FK, To KF, Wu JC, Yung MY, Leung WK, Kwok T et al. Eradication ofHelicobacter pylori and risk of peptic ulcers in patients starting long-term treatmentwith non-steroidal anti-inflammatory drugs: a randomised trial. Lancet 2002; 359: 9-13.41. Straus WL, Ofman JJ, MacLean C, Morton S, Berger ML, Roth EA et al. DoNSAIDs cause dyspepsia? A meta-analysis evaluating alternative dyspepsia<strong>de</strong>finitions. Am J Gastroenterol 2002; 97: 1.951-1.958.42. Aalykke C, Lauritsen K. Epi<strong>de</strong>miology of NSAID-related gastroduo<strong>de</strong>nal mucosalinjury. Best Pract Res Clin Gastroenterol 2001; 15: 705-722.43. The National Prescribing Center. Managing dyspepsia: the role of Helicobacterpylori. MeReC Bulletin 2001; 12 (1): 1-4.44. Moayyedi P, Axon AT, Feltbower R, Duffett S, Crocombe W, Braunholtz D et al.Relation of adult lifestiles and socioe<strong>con</strong>omic factors to the prevalence ofHelicobacter pylori infection. Int J Epi<strong>de</strong>miol 2002; 31: 624-631.45. Goodwin RD, Murray BS. Generalized Anxiety disor<strong>de</strong>r and peptic ulcer diseaseamong adults in the United States. Psychosomatic Medicine 2002; 64: 862-866.46. An<strong>de</strong>rsen IB, Jorgensen T, Bonnevie O, Gronbaek M, Sorensen TI. Smoking andalcohol intake as risk factors for bleeding and perforated peptic ulcers: a populationbasedcohort study. Epi<strong>de</strong>miology 2000; 11: 434-439.47. International Agency for Research on Cancer. Infection with Helicobacter pylori.IARC Monogr Eval Carcinog Risks Hum 1994; 61: 177-240.48. Correa P. Gastric neoplasia. Curr Gastroenterol Rep 2002; 4: 463-470.49. Priebe WM, DaCosta LR, Beck IT. Is epigastric ten<strong>de</strong>rness a sign of peptic ulcerdisease? Gastroenterology 1982; 82: 16-19.50. Numans ME, Van <strong>de</strong>r Graaf Y, <strong>de</strong> Wit NJ, Touw-Otten F, <strong>de</strong> Melker RA. Howmuch ulcer is ulcer-like? Diagnostic <strong>de</strong>terminants of peptic ulcer in open accessgastroscopy. Fam Pract 1994; 11: 382-388.51. Johannessen T, Petersen H, Kleveland PM, Dybdahl JH, Sandvik AK, Brenna Eet al. The predictive value of history in dyspepsia. Scand J Gastroenterol 1990; 25:689-697.52. Muris JW, Starmans R, Pop P, Crebol<strong>de</strong>r HF, Knottnerus JA. Discriminant valueof symptoms in patients with dyspepsia. J Fam Pract 1994; 38: 139-143.53. Bytzer P, Hansen JM, Havelund T, Malchow-Moller A, Schaffalitzky <strong>de</strong> Mucka<strong><strong>de</strong>l</strong>lOB. Predicting endoscopic diagnosis in the dyspeptic patient: the value of clinicaljudgement. Eur J Gastroenterol Hepatol 1996; 8: 359-363.90
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INSTITUTO GUATEMALTECO DE SEGURIDAD
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Los grados de recomendación son cr
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Las GPC ayudan a los profesionales
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2.11.3. Medidas higiénico-dietéti
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GUÍA DE BOLSILLO de Enfermedad Pé
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GUÍA DE BOLSILLO de Enfermedad Pé
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GUÍA DE BOLSILLO de Enfermedad Pé
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GUÍA DE BOLSILLO de Enfermedad Pé
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1 INTRODUCCIÓN1.1. ANTECEDENTESLa
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1.3. OBJETIVOS1.3.1. Proporcionar i
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2 CONTENIDO2.1. DEFINICIÓN Y TERMI
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Así mismo, la incidencia del cánc
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Los trastornos psicológicos y psiq
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2bA partir de la historia clínica,
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Estos modelos, construidos mediante
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2.8.1. TRATAMIENTO EMPÍRICO ANTISE
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2.8.3. INVESTIGACIÓN DE LA INFECCI
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Estos aspectos ya fueron señalados
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Es necesario evitar la toma de fár
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Algoritmo 2. Manejo del paciente co
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