Emotional inteligence

aygun.shukurova
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04.02.2022 Views

332/661Once heart disease develops, the mechanismstriggered by anger affect the very efficiency of the heartas a pump, as was shown in the study of angry memoriesin heart patients. The net effect is to make anger particularlylethal in those who already have heart disease.For instance, a Stanford University Medical Schoolstudy of 1,012 men and women who suffered from a firstheart attack and then were followed for up to eight yearsshowed that those men who were most aggressive andhostile at the outset suffered the highest rate of secondheart attacks. 12 There were similar results in a YaleSchool of Medicine study of 929 men who had survivedheart attacks and were tracked for up to ten years. 13Those who had been rated as easily roused to angerwere three times more likely to die of cardiac arrest thanthose who were more even-tempered. If they also hadhigh cholesterol levels, the added risk from anger wasfive times higher.The Yale researchers point out that it may not be angeralone that heightens the risk of death from heart disease,but rather intense negative emotionality of anykind that regularly sends surges of stress hormonesthrough the body. But overall, the strongest scientificlinks between emotions and heart disease are to anger: aHarvard Medical School study asked more than fifteenhundred men and women who had suffered heart

333/661attacks to describe their emotional state in the hours beforethe attack. Being angry more than doubled the riskof cardiac arrest in people who already had heart disease;the heightened risk lasted for about two hoursafter the anger was aroused. 14These findings do not mean that people should try tosuppress anger when it is appropriate. Indeed, there isevidence that trying to completely suppress such feelingsin the heat of the moment actually results in magnifyingthe body's agitation and may raise blood pressure.15 On the other hand, as we saw in Chapter 5, thenet effect of ventilating anger every time it is felt issimply to feed it, making it a more likely response to anyannoying situation. Williams resolves this paradox byconcluding that whether anger is expressed or not is lessimportant than whether it is chronic. An occasional displayof hostility is not dangerous to health; the problemarises when hostility becomes so constant as to definean antagonistic personal style—one marked by repeatedfeelings of mistrust and cynicism and the propensity tosnide comments and put-downs, as well as more obviousbouts of temper and rage. 16The hopeful news is that chronic anger need not be adeath sentence: hostility is a habit that can change. Onegroup of heart-attack patients at Stanford UniversityMedical School was enrolled in a program designed to

333/661

attacks to describe their emotional state in the hours before

the attack. Being angry more than doubled the risk

of cardiac arrest in people who already had heart disease;

the heightened risk lasted for about two hours

after the anger was aroused. 14

These findings do not mean that people should try to

suppress anger when it is appropriate. Indeed, there is

evidence that trying to completely suppress such feelings

in the heat of the moment actually results in magnifying

the body's agitation and may raise blood pressure.

15 On the other hand, as we saw in Chapter 5, the

net effect of ventilating anger every time it is felt is

simply to feed it, making it a more likely response to any

annoying situation. Williams resolves this paradox by

concluding that whether anger is expressed or not is less

important than whether it is chronic. An occasional display

of hostility is not dangerous to health; the problem

arises when hostility becomes so constant as to define

an antagonistic personal style—one marked by repeated

feelings of mistrust and cynicism and the propensity to

snide comments and put-downs, as well as more obvious

bouts of temper and rage. 16

The hopeful news is that chronic anger need not be a

death sentence: hostility is a habit that can change. One

group of heart-attack patients at Stanford University

Medical School was enrolled in a program designed to

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