Womb as Paradise Lost
Dissertation 2015. Womb as Paradise Lost - Regained by the Energy of Life.
My name is Dr. Gideon Benavraham, professor-emeritus Clinical Hermeneutics. "What happens in a human being fundamentally during the proces of prenatal development (Fetal Programming) and what are the consequences to distortions and diseases later on life?" Research tools: Mindlink-Tesla-Transformation Technology (MTTT) as diagnosticum with PEMF and music frequencies as treatment methods. A RCT-double blind and placebo-controlled research, with statistics.
Dissertation 2015. Womb as Paradise Lost - Regained by the Energy of Life.
My name is Dr. Gideon Benavraham, professor-emeritus Clinical Hermeneutics. "What happens in a human being fundamentally during the proces of prenatal development (Fetal Programming) and what are the consequences to distortions and diseases later on life?" Research tools: Mindlink-Tesla-Transformation Technology (MTTT) as diagnosticum with PEMF and music frequencies as treatment methods. A RCT-double blind and placebo-controlled research, with statistics.
88Womb as Paradise Lost – Foetal ProgrammingThetic Part 1 – Philosophical Introductioneral processes of perceptual and motor skill learning, and more specifically resembleshuman speech learning. Song is sub served by circuitry that is specializedfor vocal learning and production, but that has strong similarities to mammalianbrain pathways (…) there are mechanistic parallels between birdsong andaspects of speech and social communion. (…) Songbirds therefore represent analmost unique animal model: song learning has remarkable parallels to human75 76speech learning.The speech disorders like auditory hallucinations of schizophrenia can be a mutationof FOXP2 as a genetic vulnerability, predisposed for auditory hallucinations.“Interestingly polymorphisms in FOXP2 have recently been associated with auditoryhallucinations (…) If hallucinations are misidentified perceptions originatingin the speech perception area of the left temporal lobe, then hallucinating patientsshould have problems identifying a simultaneously presented external speechsound, especially when the sound is presented and lateralized to the left hemisphere”.77Also the epigenetic research has formed an important part of the FOXP2 research.The question remains: “Do we have here an irreversible or a reversible distortionas a selection in the human lineage?” 7875 Michael S. Brainard a.o. “Translating Songbirds as a model for basic and applied medical research. Conclusion: “Nosingle animal model is likely to recapitulate all the features of complex neurological and psychiatric diseases, but we arguehere that songbirds, with their easily quantifiable learned behaviur and dedicated circuit, are a iniquely usefull addition tomore traditional model systems. (…) It is our hope that enhanced dialogue between songbird and human researchers, withattention to both the similarities and differences between systems, can provide new thrapeutic insights”.76 Ikuku Teramitsu, a.o. “Parallel FOXP1 and FOXP2 Expression in Songbird and Human Brain Predicts FunctionalInteraction”, The Journal of Neuroscience, 200477 Kenneth Hugdahl a.o. “Auditory hallucinations in schizophrenia: the role of cognitive, brain structural and geneticdisturbances in the left temporal lobe”, Frontiers in Human Neuroscience, 2008.78 Amporo Tolosa, a.o. “FOXP2 gene and language impairmaent in schizophrenia: association and epigenetic studies”,BioMed Central, 2010.
89Womb as Paradise Lost – Foetal ProgrammingThetic Part 1 – Philosophical Introduction3.7 ConclusionHeritage at the moment of procreation and the foetal programming as developmentcan become a monogenetic disorder. At the development of the epigeneticawe have to discern between the monogenetic disorders (irreversibly) and the epigeneticdisorders (reversibly). For instance: a long time the diagnosis ADHD wasa genetic distortion. Nowadays we know ADHD/ADD must be held for a epigeneticdistortion. The genetic distortion has a damage of the DNA-sequence, theepigenetic distortion depends on the environmental factors without damaging theDNA-sequences. That means: when we change the environmental factors, wehave a change to recover.May be we will come to the conclusion that “schizophrenia in a mild form” willbe a epigenetic disorder with view on recovery, instead of a rigid degenerativedisease. In that case the auditory hallucinations may be can stop by activation ofendorphins, memantine by an energetical treatment.Because of the universalistic matter “language and music” will form a core elementin the Tesla-technologic Mindlink research and treatment with a systematicsplit up between language and music, as positive energy. This energy can be usedfor a return to homeostasis by de-blockage of the negative energetic blockages.The treatment must be as deep as possible, so we have to look for the evolutionaryfoundations and energetic functional stimuli. Also for human beings musicand language have an individual signal function and group stimulation power,that bounds together. Although the investigations around the question “Is musican evolutionary adaptation, based on natural selection?” is an ongoing process.My research of “Maternal-Foetal-Distortion”-syndrome (MFD) connected withthe epigenetic and prenatal development disturbances, will prove the positiveeffects on these disorders (See the Practical Part of this dissertation).I will close this part going back to music and the brain, with a statement about theuniversal meaning of FOXP2 of Patel:
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Womb as Paradise Lost – Foetal Programming
Thetic Part 1 – Philosophical Introduction
eral processes of perceptual and motor skill learning, and more specifically resembles
human speech learning. Song is sub served by circuitry that is specialized
for vocal learning and production, but that has strong similarities to mammalian
brain pathways (…) there are mechanistic parallels between birdsong and
aspects of speech and social communion. (…) Songbirds therefore represent an
almost unique animal model: song learning has remarkable parallels to human
75 76
speech learning.
The speech disorders like auditory hallucinations of schizophrenia can be a mutation
of FOXP2 as a genetic vulnerability, predisposed for auditory hallucinations.
“Interestingly polymorphisms in FOXP2 have recently been associated with auditory
hallucinations (…) If hallucinations are misidentified perceptions originating
in the speech perception area of the left temporal lobe, then hallucinating patients
should have problems identifying a simultaneously presented external speech
sound, especially when the sound is presented and lateralized to the left hemisphere”.
77
Also the epigenetic research has formed an important part of the FOXP2 research.
The question remains: “Do we have here an irreversible or a reversible distortion
as a selection in the human lineage?” 78
75 Michael S. Brainard a.o. “Translating Songbirds as a model for basic and applied medical research. Conclusion: “No
single animal model is likely to recapitulate all the features of complex neurological and psychiatric diseases, but we argue
here that songbirds, with their easily quantifiable learned behaviur and dedicated circuit, are a iniquely usefull addition to
more traditional model systems. (…) It is our hope that enhanced dialogue between songbird and human researchers, with
attention to both the similarities and differences between systems, can provide new thrapeutic insights”.
76 Ikuku Teramitsu, a.o. “Parallel FOXP1 and FOXP2 Expression in Songbird and Human Brain Predicts Functional
Interaction”, The Journal of Neuroscience, 2004
77 Kenneth Hugdahl a.o. “Auditory hallucinations in schizophrenia: the role of cognitive, brain structural and genetic
disturbances in the left temporal lobe”, Frontiers in Human Neuroscience, 2008.
78 Amporo Tolosa, a.o. “FOXP2 gene and language impairmaent in schizophrenia: association and epigenetic studies”,
BioMed Central, 2010.