Womb as Paradise Lost
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Womb as Paradise Lost

Dissertation 2015. Womb as Paradise Lost - Regained by the Energy of Life. My name is Dr. Gideon Benavraham, professor-emeritus Clinical Hermeneutics. "What happens in a human being fundamentally during the proces of prenatal development (Fetal Programming) and what are the consequences to distortions and diseases later on life?" Research tools: Mindlink-Tesla-Transformation Technology (MTTT) as diagnosticum with PEMF and music frequencies as treatment methods. A RCT-double blind and placebo-controlled research, with statistics. Dissertation 2015. Womb as Paradise Lost - Regained by the Energy of Life.
My name is Dr. Gideon Benavraham, professor-emeritus Clinical Hermeneutics. "What happens in a human being fundamentally during the proces of prenatal development (Fetal Programming) and what are the consequences to distortions and diseases later on life?" Research tools: Mindlink-Tesla-Transformation Technology (MTTT) as diagnosticum with PEMF and music frequencies as treatment methods. A RCT-double blind and placebo-controlled research, with statistics.

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19.07.2020 Views

Womb as Paradise Lost – Foetal Programming544Practical Part – Appendicespression - results in altered epigenetics, with the next step as pre-procreation, tothe Foetal programming on the way to “gene-trauma interaction”: in other words:“intergenerational altered epigenetic inheritance”, expressive after the gametepreparation into the conception.Gene-environmental interaction has a cultural contextuality: here the Holocaustand Concentration Camp violence and hunger. In this matter of fact violence hasbeen the dominant factor, guided the process of trauma transmission. 41930.3 Dutch Hunger Winter (1944-1945)Dutch television 420 made a documentary about the hunger winter during the period1944-1945, a period in which over 15,000 people died of starvation. In thatperiod were also new people born. What happened to them? Under the headline"Hunger Winter haunts offspring" Elmar Veerman reports an interview withBastaan T. Heijmans, epigenetic researcher at the Leiden University MedicalCenter.Bastiaans in the publication of PNAS: “Here we show that individuals who wereprenatally exposed to famine during the Dutch Hunger Winter in 1944–45 had, 6decades later, less DNA methylation of the imprinted IGF2 gene compared withtheir unexposed, same-sex siblings. 421 (...) These data are the first to contribute419 Kellermann, Natan P.F. “Transmission of Holocaust Trauma”. In his model he includes four levels: 1. psychodynamic,2. socio-cultural, 3. family system and 4. biological points of view.420 NPO-science, 29th of October 2008.421 Heijmans, Bastiaan T., op. cit. “One of the best-characterized epigenetically regulated loci is insuline-like growthfactor II (IGF2). IGF2 is a key factor in human growth and development and is maternally imprinted. Imprinting is maintainedthrough the IGF2 differentially methylated region (DMR), the hypomethylation of which leads to bi-allelic expressionof IGF2. (...) IGF2 DMR methylation is a normally distributed quantitative trait that is largely determined by geneticfactors in both adolescence and middle age, indicating that the methylation mark is stable up to middle age. Thus, if affectedby environmental conditions early in human development, altered IGF2 DMR methylation may be detected manyyears later.

Womb as Paradise Lost – Foetal Programming545Practical Part – Appendicesempirical support for the hypothesis that early-life environmental conditions cancause epigenetic changes in humans that persist throughout life.” 422In a press release (2014) LUMC affirmed the research of 2008. We quote:“During the famine of 1944-1945 the availability of food fell to a quarter of whata man needs. Yet when children were conceived that were born with a normalbirth weight. Advanced DNA testing of famine children has now revealed that thegenes that promote growth are tuned differently by them. This probably explainswhy those in middle age have a higher risk of health problems.The modifications to the DNA that have occurred in famine children do seem tofollow a special pattern: groups of genes work together to promote growth in thewomb, are tuned differently to them than to their siblings, who were conceivedbefore or after the famine and born ”.42330.4 Conditions in the uterusThe investigated Hunger Winter Children were all around 60 years old at the timethey ceded DNA to the investigation. The growth genes are adjusted life, differentfor people who are conceived during the Hunger Winter."Adjusting genes occurs largely in the first weeks after conception. The operatingsystem determines which genes are on and off is then tuned. This operating system,also known as epigenetics, consists of molecular dimmers on the DNA.Conditions in the uterus, such as lack of food or stress of the mother, can cause422 Heijmans, Bastiaan T., op. cit. “... we used the ongoing Hunger Winter Families Study to investigate whether prenatalexposure to famine is associated with persistent differences in methylation of the IGF2 DMR (...) whether periconceptionalexposure to famine was associated with differences in IGF2 DMR methylation in adulthood (...) who were conceivedduring the famine 6 decades ago. The exposure period thus included the very early stages of development”.423 Press Release, LUMC 2014.

Womb as Paradise Lost – Foetal Programming

544

Practical Part – Appendices

pression - results in altered epigenetics, with the next step as pre-procreation, to

the Foetal programming on the way to “gene-trauma interaction”: in other words:

“intergenerational altered epigenetic inheritance”, expressive after the gamete

preparation into the conception.

Gene-environmental interaction has a cultural contextuality: here the Holocaust

and Concentration Camp violence and hunger. In this matter of fact violence has

been the dominant factor, guided the process of trauma transmission. 419

30.3 Dutch Hunger Winter (1944-1945)

Dutch television 420 made a documentary about the hunger winter during the period

1944-1945, a period in which over 15,000 people died of starvation. In that

period were also new people born. What happened to them? Under the headline

"Hunger Winter haunts offspring" Elmar Veerman reports an interview with

Bastaan T. Heijmans, epigenetic researcher at the Leiden University Medical

Center.

Bastiaans in the publication of PNAS: “Here we show that individuals who were

prenatally exposed to famine during the Dutch Hunger Winter in 1944–45 had, 6

decades later, less DNA methylation of the imprinted IGF2 gene compared with

their unexposed, same-sex siblings. 421 (...) These data are the first to contribute

419 Kellermann, Natan P.F. “Transmission of Holocaust Trauma”. In his model he includes four levels: 1. psychodynamic,

2. socio-cultural, 3. family system and 4. biological points of view.

420 NPO-science, 29th of October 2008.

421 Heijmans, Bastiaan T., op. cit. “One of the best-characterized epigenetically regulated loci is insuline-like growth

factor II (IGF2). IGF2 is a key factor in human growth and development and is maternally imprinted. Imprinting is maintained

through the IGF2 differentially methylated region (DMR), the hypomethylation of which leads to bi-allelic expression

of IGF2. (...) IGF2 DMR methylation is a normally distributed quantitative trait that is largely determined by genetic

factors in both adolescence and middle age, indicating that the methylation mark is stable up to middle age. Thus, if affected

by environmental conditions early in human development, altered IGF2 DMR methylation may be detected many

years later.

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