[libribook.com] Traumatic Scar Tissue Management 1st Edition
volume of fluid that accumulates or the degree of limb distortion (size).Four stages may be used to describe lymphedema (National Cancer Institute2014 – see Table 6.4).
Clinical ConsiderationEarly detection and treatment of lymphedema may limit the progressionand risks associated with more pronounced lymphedema. While subclinicallymphedema can be detected using methods such as perometry andbioimpedance spectroscopy, these technologies are not widely available.Alternatively, several studies indicate that patients’ self-reported symptomsare accurate indicators of early lymphedema. Early intervention (e.g.manual lymph treatment, compression garments) – during the more easilymanageable stage – has been shown to result in effective outcomes.As noted in Chapter 3, lymphedema is classified as primary or secondary.Secondary lymphedema – seen in conjunction with the surgical removal oflymph nodes or the use of radiation during cancer treatment – rarely happens inisolation. Scarring (soft tissue fibrosis), deficits in muscle strength, flexibilityand hypersensitivity are usually present. These symptoms are due to damagesustained to the supporting and organizational aspects of the fascial and CTstructures and their response to the healing process (Fourie 2008).There are a number of tissue composition changes in secondary lymphedemadue to the stagnation of the lymphatic fluid. The skin of the lymphatic arm canchange from soft and pitting to hard, heavy and fibrotic with time. Use oftomography and magnetic resonance imaging (MRI) has demonstrated a patternof circumferential edema and ‘honey combing’ with infiltration of the fibrosis inthe subcutaneous tissue region. An increase of adipose tissue was alsodiscovered around the lymphatic limb (Dylke et al. 2013).StageZeroDescriptionCommonly referred to as latent, subclinical or pre-lymphedema, this presentation is typically seen inconjunction with those that have undergone surgical lymph node dissection with subsequent disruption oflymphatic pathways. Although the transport capacity of the lymph system is reduced resulting in impairedlymph flow – swelling is typically not evident. Generally there are no visible changes to the affected area;however, the individual may notice a difference in feeling, such as fatigue, mild regional numbness,tingling, fullness or heaviness, which may be accompanied by low-grade discomfort. The individual maynotice difficulty or discomfort fitting into clothing, and jewelry may feel tight (e.g. rings, watch bands).This stage may last for months or many years, with eventual development of more obvious signs of lymphimpairment.
- Page 487 and 488: Clinical ConsiderationMechanical fo
- Page 489 and 490: Table 5.4Role of neuropeptides (NP)
- Page 491 and 492: Fitch P (2005) Scars of life. Journ
- Page 493 and 494: Langevin HM (2006) Connective tissu
- Page 495 and 496: active scars. Journal of Bodywork a
- Page 497 and 498: trauma.
- Page 499 and 500: Clinical ConsiderationPostsurgical
- Page 501 and 502: following burn injury,bacterial col
- Page 503 and 504: Table 6.1Comparison of scars (Ogawa
- Page 505 and 506: Pathophysiological ConsiderationAcc
- Page 507 and 508: BurnsA burn injury to the skin or o
- Page 510 and 511: Figure 6.1Depth of burn trauma and
- Page 512 and 513: • Stimulate ECM formation• Regu
- Page 514 and 515: Clinical ConsiderationIt has been i
- Page 516 and 517: Clinical ConsiderationMT may be a v
- Page 518 and 519: ThermoregulationThermoregulation (t
- Page 520 and 521: from the tissues and taken up by th
- Page 522 and 523: treatment strategies are difficult
- Page 524 and 525: Clinical ConsiderationSkin rolling
- Page 526 and 527: Sequelae and ComplicationsAdvances
- Page 528 and 529: • Paresthesia - 47%• Arm/should
- Page 530 and 531: breast or around the edge of the ar
- Page 532 and 533: Radiation scarringScar tissue as a
- Page 534 and 535: Implants and painPain of fluctuatin
- Page 536 and 537: LymphedemaBreast cancer treatment o
- Page 540 and 541: OneTwoThreeCommonly referred to as
- Page 542 and 543: myokinetic chain/myofascial meridia
- Page 544: • Loss of touch sensation• Clum
- Page 548 and 549: Figure 6.3Distribution of nerves in
- Page 550 and 551: include preservation of as much of
- Page 552 and 553: complication is present there is de
- Page 554 and 555: Clinical ConsiderationScar complica
- Page 556 and 557: BreastCancer.org (2015c) Side Effec
- Page 558 and 559: 323-9.Kania A (2012) Scars. In: Dry
- Page 560 and 561: Slemp AE, Kirschner RE (2006) Keloi
- Page 562 and 563: scar tissue, while being mindful of
- Page 564 and 565: Physiological ResponseAs discussed
- Page 566 and 567: • The realization that one is abo
- Page 568 and 569: Stress Response and Stress Hormones
- Page 570 and 571: dissociation and inner shakiness) (
- Page 572: Figure 7.1(A) Summary of the HPA me
- Page 575 and 576: Chronic Stress ResponseChronic stre
- Page 577 and 578: Pathophysiological ConsiderationChr
- Page 579 and 580: Clinical ConsiderationProlonged str
- Page 581 and 582: Psychological Stress and Wound Heal
- Page 583 and 584: Pathophysiological ConsiderationNor
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Clinical Consideration
Early detection and treatment of lymphedema may limit the progression
and risks associated with more pronounced lymphedema. While subclinical
lymphedema can be detected using methods such as perometry and
bioimpedance spectroscopy, these technologies are not widely available.
Alternatively, several studies indicate that patients’ self-reported symptoms
are accurate indicators of early lymphedema. Early intervention (e.g.
manual lymph treatment, compression garments) – during the more easily
manageable stage – has been shown to result in effective outcomes.
As noted in Chapter 3, lymphedema is classified as primary or secondary.
Secondary lymphedema – seen in conjunction with the surgical removal of
lymph nodes or the use of radiation during cancer treatment – rarely happens in
isolation. Scarring (soft tissue fibrosis), deficits in muscle strength, flexibility
and hypersensitivity are usually present. These symptoms are due to damage
sustained to the supporting and organizational aspects of the fascial and CT
structures and their response to the healing process (Fourie 2008).
There are a number of tissue composition changes in secondary lymphedema
due to the stagnation of the lymphatic fluid. The skin of the lymphatic arm can
change from soft and pitting to hard, heavy and fibrotic with time. Use of
tomography and magnetic resonance imaging (MRI) has demonstrated a pattern
of circumferential edema and ‘honey combing’ with infiltration of the fibrosis in
the subcutaneous tissue region. An increase of adipose tissue was also
discovered around the lymphatic limb (Dylke et al. 2013).
Stage
Zero
Description
Commonly referred to as latent, subclinical or pre-lymphedema, this presentation is typically seen in
conjunction with those that have undergone surgical lymph node dissection with subsequent disruption of
lymphatic pathways. Although the transport capacity of the lymph system is reduced resulting in impaired
lymph flow – swelling is typically not evident. Generally there are no visible changes to the affected area;
however, the individual may notice a difference in feeling, such as fatigue, mild regional numbness,
tingling, fullness or heaviness, which may be accompanied by low-grade discomfort. The individual may
notice difficulty or discomfort fitting into clothing, and jewelry may feel tight (e.g. rings, watch bands).
This stage may last for months or many years, with eventual development of more obvious signs of lymph
impairment.