[libribook.com] Traumatic Scar Tissue Management 1st Edition
LymphedemaBreast cancer treatment often impacts upper body function. Dysfunction may bedue to fibrosis, lack of muscle strength, lack of flexibility, lymphaticinsufficiency (lymphedema) and neural hypersensitivity (Fourie 2008).Lymphedema is caused by an inefficiency in the lymphatic system. Hydrophilicprotein becomes congested in the interstitial spaces of the extremities or trunk,which causes swelling in the area.Incidence statistics of lymphedema range from 5% to 60%, with the onset ofsymptoms occurring immediately following surgery to up to 30 years postsurgery(Poage 2008). Statistics for development of lymphedema for breastcancer patients range from 2.4% to 56%. Once lymphedema is established it canbe managed but not cured (Fourie 2008).Grade I Grade II Grade III Grade IVBreastappearance isnatural/normal,feel is of normalsoftness and thecapsule ispliable/suppleBreast appearance isnatural/normal butfeels somewhat hardto the touch andpliability/supplenessis somewhatdiminishedThe breast feels hard,pliability/suppleness is morenoticeably diminished and somedistortion is evident (e.g. presents asan atypical rounded shape, or theimplant is generally tilted upwards)Similar to grade III butwith greater distortionand hardening of thecapsule –pain/discomfort mayaccompany this grade ofcontracture
Table 6.3Baker scaleCancer treatment is the leading cause of lymphedema in developed countries.Development and severity of lymphedema have a significant impact on comfort,psychological distress and overall quality of life. Development of lymphedemaand at what point in a patient’s recovery stage it may manifest are wide ranging.Some research studies indicate factors such as variations in anatomy, surgicalprocedures and radiotherapy may play a part in lymphedema development(Fourie 2008).Quantitative lymphoscintigraphy – a method used to check the lymph system fordisease (e.g. lymphoma, lymphedema) – reveals that lymph drainage is slowedin the subcutis (deeper part of the dermis), where most of the edema lies, and inthe subfascial muscle compartment, which normally has much higher lymphflows than the subcutis.Although the associated musculature does not swell significantly, the impaireddrainage correlates with the severity of arm subfascial swelling, indicating animportant role for muscle lymphatic function (Stanton et al. 2009). The farreachingconsequences of chronic lymph stasis is not completely understood;however, what is known is that the accumulation of the protein-rich interstitialfluid lends to early and progressive predisposition to tissue fibrosis. Componentsof the epidermal and dermal layers thicken significantly which, in turn, increasesthe thickness of the subcutaneous adipose layer (Rockson 2013).Early indications of lymphedema include self-reported sensations of heaviness inthe affected limb, edema, tingling, fatigue, or aching. Lymphedema may initiallybe dismissed as edema, discomfort and inflammation after surgery. Axillaryparesthesia and pain in the breast, chest and arm have been reported assymptoms of lymphedema (Poage et al. 2008).Lymphedema is generally defined by the consistency of the tissues, not the
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Table 6.3
Baker scale
Cancer treatment is the leading cause of lymphedema in developed countries.
Development and severity of lymphedema have a significant impact on comfort,
psychological distress and overall quality of life. Development of lymphedema
and at what point in a patient’s recovery stage it may manifest are wide ranging.
Some research studies indicate factors such as variations in anatomy, surgical
procedures and radiotherapy may play a part in lymphedema development
(Fourie 2008).
Quantitative lymphoscintigraphy – a method used to check the lymph system for
disease (e.g. lymphoma, lymphedema) – reveals that lymph drainage is slowed
in the subcutis (deeper part of the dermis), where most of the edema lies, and in
the subfascial muscle compartment, which normally has much higher lymph
flows than the subcutis.
Although the associated musculature does not swell significantly, the impaired
drainage correlates with the severity of arm subfascial swelling, indicating an
important role for muscle lymphatic function (Stanton et al. 2009). The farreaching
consequences of chronic lymph stasis is not completely understood;
however, what is known is that the accumulation of the protein-rich interstitial
fluid lends to early and progressive predisposition to tissue fibrosis. Components
of the epidermal and dermal layers thicken significantly which, in turn, increases
the thickness of the subcutaneous adipose layer (Rockson 2013).
Early indications of lymphedema include self-reported sensations of heaviness in
the affected limb, edema, tingling, fatigue, or aching. Lymphedema may initially
be dismissed as edema, discomfort and inflammation after surgery. Axillary
paresthesia and pain in the breast, chest and arm have been reported as
symptoms of lymphedema (Poage et al. 2008).
Lymphedema is generally defined by the consistency of the tissues, not the