[libribook.com] Traumatic Scar Tissue Management 1st Edition
Figure 6.1Depth of burn trauma and structures impacted.Dermal water plays an important role in maintaining the physical properties ofthe skin (e.g. moisturizing and elasticity) (Nakagawa et al. 2010). Specializedcells in the dermis assist with regulating temperature and fighting infection. Thedermis also houses vessels that supply blood and nutrients to the skin. Damageto the sweat glands, sebaceous glands, hair follicles, nerve receptors, bloodvessels, lymph nodes/vessels, muscle and connective tissue may occur.Partial-thickness burn injuries can be extremely painful, are often accompaniedby blistering and edema/swelling, and may require debridement (removal oflacerated, weaken, debilitated or contaminated tissue) or skin grafting(transplanted tissue). Depending on the type of skin graft needed, the donor sitemay lose the epidermis and part or all of the dermis.Third DegreeThird degree trauma involves destruction of all epidermal and dermal layers andis commonly referred to as a ‘full-thickness’ injury. Damage extends below hairfollicles and sweat glands, into the subcutaneous tissue (i.e. connective tissue(CT) layer that is seeded with adipocytes).With full-thickness trauma, there may be extensive edema and skincharacteristics can change dramatically (e.g. charred, leathery appearance). Thirddegree injuries are usually not painful because of the destruction of nerveendings. Skin grafting or other replacement options are often required.Fourth DegreeFourth degree trauma involves destruction of epidermis, dermis, subcutaneoustissue, ligaments, tendons, nerves, blood vessels, possibly down to the level ofbone. Fourth degree trauma often leads to amputation and significant functionalimpairment. Skin grafting and multistage reconstructive procedures arenecessary.
Sequelae and ComplicationsA critical burn injury is a unique trauma that often is accompanied by significantmetabolic disturbances as well as a change in the normal state of innate andadaptive immunity (Shankar et al. 2007). Human skin acts as a blockade againstenvironmental insults and against colonization of pathogenic microbes, and isalso an immune organ with significant surveillance and thermoregulatoryfunctions. This knowledge helps us to understand that the loss of large portionsof skin as the result of burns results in impaired immunity, metaboliccompromises, fluid shifts, and heat loss (Shankar et al. 2007).Burn injuries can cause severe muscle loss, muscle weakness, contractures, andtraumatic scars, leading to lifelong physical impairments (Diego et al. 2013).Severe burns may require the removal of the dermal layer and subsequent skingraft procedure(s) (Ganio et al. 2013).Burn injury sequelae can occur as a result of scarring associated with the burntrauma or graft proceedures.Traumatic ScarsHypertrophic scarring is the most common type of scar tissue formed after aburn injury. Hypertrophic scars are formed in 30–72% of burn survivors afterinjury. In addition to the above noted risk factors, hypertrophic scar developmentassociated with burn injuries is mainly influenced by wound healing time(delayed 3 weeks or more), the depth (degree of injury), extent (size/surface areaaffected) and if multiple surgical or graft procedures are performed (Deitch et al.1983, Cubison et al. 2006, Thompson et al. 2013, Cho et al. 2014).Unfortunately, most of the published information on post-burn scarring does notaccurately define these factors and the body’s whirlpool of physiologicalresponses to burn injury continue to be studied and researched (Spurr &Shakespeare 1990, Bombaro et al. 2003, Cho et al. 2014).What do we know? Fibroblasts play a key role in wound healing and scarformation as they:• Actively release cytokines
- Page 458 and 459: Table 5.3Scar types and related ter
- Page 460 and 461: unyielding or pliable and mobile. R
- Page 462 and 463: Prolonged InflammationInflammation
- Page 464 and 465: ImmobilizationThe impact of immobil
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- Page 483 and 484: compressive effect in the keloidal
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- Page 489 and 490: Table 5.4Role of neuropeptides (NP)
- Page 491 and 492: Fitch P (2005) Scars of life. Journ
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- Page 495 and 496: active scars. Journal of Bodywork a
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- Page 503 and 504: Table 6.1Comparison of scars (Ogawa
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- Page 512 and 513: • Stimulate ECM formation• Regu
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- Page 520 and 521: from the tissues and taken up by th
- Page 522 and 523: treatment strategies are difficult
- Page 524 and 525: Clinical ConsiderationSkin rolling
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- Page 528 and 529: • Paresthesia - 47%• Arm/should
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- Page 538 and 539: volume of fluid that accumulates or
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- Page 542 and 543: myokinetic chain/myofascial meridia
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- Page 550 and 551: include preservation of as much of
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Figure 6.1
Depth of burn trauma and structures impacted.
Dermal water plays an important role in maintaining the physical properties of
the skin (e.g. moisturizing and elasticity) (Nakagawa et al. 2010). Specialized
cells in the dermis assist with regulating temperature and fighting infection. The
dermis also houses vessels that supply blood and nutrients to the skin. Damage
to the sweat glands, sebaceous glands, hair follicles, nerve receptors, blood
vessels, lymph nodes/vessels, muscle and connective tissue may occur.
Partial-thickness burn injuries can be extremely painful, are often accompanied
by blistering and edema/swelling, and may require debridement (removal of
lacerated, weaken, debilitated or contaminated tissue) or skin grafting
(transplanted tissue). Depending on the type of skin graft needed, the donor site
may lose the epidermis and part or all of the dermis.
Third Degree
Third degree trauma involves destruction of all epidermal and dermal layers and
is commonly referred to as a ‘full-thickness’ injury. Damage extends below hair
follicles and sweat glands, into the subcutaneous tissue (i.e. connective tissue
(CT) layer that is seeded with adipocytes).
With full-thickness trauma, there may be extensive edema and skin
characteristics can change dramatically (e.g. charred, leathery appearance). Third
degree injuries are usually not painful because of the destruction of nerve
endings. Skin grafting or other replacement options are often required.
Fourth Degree
Fourth degree trauma involves destruction of epidermis, dermis, subcutaneous
tissue, ligaments, tendons, nerves, blood vessels, possibly down to the level of
bone. Fourth degree trauma often leads to amputation and significant functional
impairment. Skin grafting and multistage reconstructive procedures are
necessary.