[libribook.com] Traumatic Scar Tissue Management 1st Edition

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rehabilitation and injury prevention. Journal of Orthopaedic and Sports Physical Therapy 40(2): 67–81.Henderson J, Terenghi G, McGrouther DA, Ferguson MW (2006) The reinnervation pattern of wounds andscars may explain their sensory symptoms. Journal of Plastic Reconstructive and Aesthetic Surgery 59(9):942–50.Henderson J, Ferguson MW, Terenghi G (2012). The feeling of healing. Plastic and Reconstructive Surgery129 (1): 223e–224e.Henry G, Garner, WL (2003) Inflammatory mediators in wound healing. Surgical Clinics of North America83: 483–507.Hinz B (2007) Formation and function of the myofibroblast during tissue repair. Journal of InvestigativeDermatology 127: 526–537.Hinz, B (2009) Tissue stiffness, latent TGF-beta1 activation, and mechanical signal transduction:implications for the pathogenesis and treatment of fibrosis. Current Rheumatology Reports 11(2): 120–126.Hinz B, Celetta G, Tomasek JJ et al (2001) Alpha-smooth muscle actin expression upregulates fibroblastcontractile activity. Molecular Biology of the Cell 12: 2730–41.Hinz B, Phan SH, Thannickal VJ et al (2012) Recent developments in myofibroblast biology: paradigms forconnective tissue remodeling. American Journal of Pathology 180 (4): 1340–1355.Hinz B (2013) It has to be the [alpha] v: myofibroblast integrins activate latent TGF-[beta] 1. NatureMedicine 19(12): 1567–1568.Hochman B, Nahas FX, Sobral CS et al (2008) Nerve fibres: a possible role in keloid pathogenesis. BritishJournal of Dermatology 158: 651–652.Huang C, Murphy GF, Akaish S, Ogawa R (2013) Keloids and hypertrophic scars: update and futuredirections. Plastic and Reconstructive Surgery Global Open 1(4).Ingber DE (1997) Tensegrity: the architectural basis of cellular mechanotransduction. Annual Review ofPhysiology 59(1): 575–599.Jing C, Jia-Han W, Hong-Xing Z (2010) Double-edged effects of neuropeptide substance P on repair ofcutaneous trauma. Wound Repair Regeneration 18: 319–324.Josenhans E (2007) Physiotherapeutic treatment for axillary cord formation following breast cancer surgery.ZVK Science Prize.Kania A (2012) Scars. In: Dryden T, Moyer C (eds) Massage therapy: Integrating research and practice.Human Kinetics Ch 15, pp 173–184.Karalaki M, Fili S, Philippou, A, Koutsilieris, M. (2009). Muscle regeneration: cellular and molecularevents. In vivo, 23(5): 779–796.Klingler W, Jurkat-Rott K, Lehmann-Horn F, Schleip R (2012) The role of fibrosis in Duchenne musculardystrophy. Acta Myologica 31(3): 184–95.Klingler W, Schleip R, Zorn A (2004) European Fascia Research Project Report. 5th World Congress LowBack and Pelvic Pain, Melbourne.Kobesova A et al (2007) Twenty-year-old pathogenic ‘active’ postsurgical scar: a case study of a patientwith persistent right lower quadrant pain. Journal of Manipulative and Physiological Therapeutics 30(3):234–238.Krammer A, Lu H, Isralewitz B et al (1999) Forced unfolding of the fibronectin type III module reveals atensile molecular recognition switch. Proceedings of the National Academy of Sciences in USA Feb 16;96(4): 1351–6.

Langevin HM (2006) Connective tissue: a body-wide signaling network? Med Hypotheses 66: 1074–1077.Langevin H (2010) Presentation: Ultrasound Imaging of Connective Tissue Pathology Associated withChronic Low Back Pain. 7th Interdisciplinary Congress on Low Back and Pelvic Pain, Los Angeles.Lawman MJ, Boyle MD, Gee AP, Young M (1985) Nerve growth factor accelerates the early cellular eventsassociated with wound healing. Experimental and Molecular Pathology 43(2): 274–281.Lee SS, Yosipovitch G, Chan YH, Goh CL (2004) Pruritus, pain, and small nerve fiber function in keloids:a controlled study. Journal of the American Academy of Dermatology 51: 1002–1006.Lewin GR, Mendell LM. (1993) Nerve growth factor and nociception. Trends in Neuroscience 16: 353–359.Li AK, Koroly MJ, Schattenkerk ME et al (1980) Nerve growth factor: acceleration of the rate of woundhealing in mice. Proceedings of the National Academy of Sciences in USA 77: 4379–81.Lowe JC (2009) Factors that induce fibrosis of fascial tissues. [accessed April 10, 2009 from HYPERLINK“http://www.drlowe.com” www.drlowe.com.]Marieb EN (2003) Human anatomy and physiology, 5th edn. Redwood City, CA: Benjamin CummingsPublishing, 5.Matsuda H, Koyama H, Sato H et al (1998) Role of nerve growth factor in cutaneous wound healing:accelerating effects in normal and healing-impaired diabetic mice. Journal of Experimental Medicine 187:297–306.Menke NB, Ward KR, Witten TM et al (2007). Impaired wound healing. Clinics in Dermatology 25: 19–25.Meyer-ter-Vehn T, Han H, Grehn F, Schlunck G (2011) Extracellular matrix elasticity modulates TGF-β–induced p38 activation and myofibroblast transdifferentiation in human tenon fibroblasts. InvestigativeOphthalmology and Visual Science 52(12): 9149–9155.Miyamoto J, Nagasao T, Miyamoto S et al (2009) Biomechanical analysis of stresses occurring in verticaland transverse scars on the lower leg. Plastic Reconstructive Surgery 124(6): 1974–1979.Moore ML, Dewey WS, Richard RL (2009) Rehabilitation of the burned hand. Hand Clinics 25(4): 529–541.Muangman P, Muffley LA, Anthony JP et al (2004) Nerve growth factor accelerates wound healing indiabetic mice. Wound Repair and Regeneration 12: 44–52.Ogawa R (2008) Keloid and hypertrophic scarring may result from a mechanoreceptor or mechanosensitivenociceptor disorder. Medical Hypotheses 71(4): 493–500.Ogawa R (2011) Mechanobiology of scarring. Wound Repair and Regeneration 19(s1), s2–s9.Ogawa R, Okai K, Tokumura F, Mori K et al (2012). The relationship between skin stretching/contractionand pathologic scarring: the important role of mechanical forces in keloid generation. Wound Repair andRegeneration, 20(2): 149–157.Profyris C, Tziotzios C, DoVale I (2012) Cutaneous scarring: pathophysiology, molecular mechanisms, andscar reduction therapeutics Part I. The molecular basis of scar formation. Journal of the American Academyof Dermatology 66: 1–10.Quinlan KL, Song IS, Bunnett NW et al (1998) Neuropeptide regulation of human dermal microvascularendothelial cell ICAM-1 expression and function. American Journal of Physiology 275: C1580–90.Quinlan KL, Naik SM, Cannon G et al. (1999) Substance P activates coincident NF-AT- and NF-kappa B-dependent adhesion molecule gene expression in microvascular endothelial cells through intracellularcalcium mobilization. Journal of Immunology 163(10): 5656–65.

Langevin HM (2006) Connective tissue: a body-wide signaling network? Med Hypotheses 66: 1074–1077.

Langevin H (2010) Presentation: Ultrasound Imaging of Connective Tissue Pathology Associated with

Chronic Low Back Pain. 7th Interdisciplinary Congress on Low Back and Pelvic Pain, Los Angeles.

Lawman MJ, Boyle MD, Gee AP, Young M (1985) Nerve growth factor accelerates the early cellular events

associated with wound healing. Experimental and Molecular Pathology 43(2): 274–281.

Lee SS, Yosipovitch G, Chan YH, Goh CL (2004) Pruritus, pain, and small nerve fiber function in keloids:

a controlled study. Journal of the American Academy of Dermatology 51: 1002–1006.

Lewin GR, Mendell LM. (1993) Nerve growth factor and nociception. Trends in Neuroscience 16: 353–

359.

Li AK, Koroly MJ, Schattenkerk ME et al (1980) Nerve growth factor: acceleration of the rate of wound

healing in mice. Proceedings of the National Academy of Sciences in USA 77: 4379–81.

Lowe JC (2009) Factors that induce fibrosis of fascial tissues. [accessed April 10, 2009 from HYPERLINK

“http://www.drlowe.com” www.drlowe.com.]

Marieb EN (2003) Human anatomy and physiology, 5th edn. Redwood City, CA: Benjamin Cummings

Publishing, 5.

Matsuda H, Koyama H, Sato H et al (1998) Role of nerve growth factor in cutaneous wound healing:

accelerating effects in normal and healing-impaired diabetic mice. Journal of Experimental Medicine 187:

297–306.

Menke NB, Ward KR, Witten TM et al (2007). Impaired wound healing. Clinics in Dermatology 25: 19–25.

Meyer-ter-Vehn T, Han H, Grehn F, Schlunck G (2011) Extracellular matrix elasticity modulates TGF-β–

induced p38 activation and myofibroblast transdifferentiation in human tenon fibroblasts. Investigative

Ophthalmology and Visual Science 52(12): 9149–9155.

Miyamoto J, Nagasao T, Miyamoto S et al (2009) Biomechanical analysis of stresses occurring in vertical

and transverse scars on the lower leg. Plastic Reconstructive Surgery 124(6): 1974–1979.

Moore ML, Dewey WS, Richard RL (2009) Rehabilitation of the burned hand. Hand Clinics 25(4): 529–

541.

Muangman P, Muffley LA, Anthony JP et al (2004) Nerve growth factor accelerates wound healing in

diabetic mice. Wound Repair and Regeneration 12: 44–52.

Ogawa R (2008) Keloid and hypertrophic scarring may result from a mechanoreceptor or mechanosensitive

nociceptor disorder. Medical Hypotheses 71(4): 493–500.

Ogawa R (2011) Mechanobiology of scarring. Wound Repair and Regeneration 19(s1), s2–s9.

Ogawa R, Okai K, Tokumura F, Mori K et al (2012). The relationship between skin stretching/contraction

and pathologic scarring: the important role of mechanical forces in keloid generation. Wound Repair and

Regeneration, 20(2): 149–157.

Profyris C, Tziotzios C, DoVale I (2012) Cutaneous scarring: pathophysiology, molecular mechanisms, and

scar reduction therapeutics Part I. The molecular basis of scar formation. Journal of the American Academy

of Dermatology 66: 1–10.

Quinlan KL, Song IS, Bunnett NW et al (1998) Neuropeptide regulation of human dermal microvascular

endothelial cell ICAM-1 expression and function. American Journal of Physiology 275: C1580–90.

Quinlan KL, Naik SM, Cannon G et al. (1999) Substance P activates coincident NF-AT- and NF-kappa B-

dependent adhesion molecule gene expression in microvascular endothelial cells through intracellular

calcium mobilization. Journal of Immunology 163(10): 5656–65.

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