[libribook.com] Traumatic Scar Tissue Management 1st Edition
rehabilitation and injury prevention. Journal of Orthopaedic and Sports Physical Therapy 40(2): 67–81.Henderson J, Terenghi G, McGrouther DA, Ferguson MW (2006) The reinnervation pattern of wounds andscars may explain their sensory symptoms. Journal of Plastic Reconstructive and Aesthetic Surgery 59(9):942–50.Henderson J, Ferguson MW, Terenghi G (2012). The feeling of healing. Plastic and Reconstructive Surgery129 (1): 223e–224e.Henry G, Garner, WL (2003) Inflammatory mediators in wound healing. Surgical Clinics of North America83: 483–507.Hinz B (2007) Formation and function of the myofibroblast during tissue repair. Journal of InvestigativeDermatology 127: 526–537.Hinz, B (2009) Tissue stiffness, latent TGF-beta1 activation, and mechanical signal transduction:implications for the pathogenesis and treatment of fibrosis. Current Rheumatology Reports 11(2): 120–126.Hinz B, Celetta G, Tomasek JJ et al (2001) Alpha-smooth muscle actin expression upregulates fibroblastcontractile activity. Molecular Biology of the Cell 12: 2730–41.Hinz B, Phan SH, Thannickal VJ et al (2012) Recent developments in myofibroblast biology: paradigms forconnective tissue remodeling. American Journal of Pathology 180 (4): 1340–1355.Hinz B (2013) It has to be the [alpha] v: myofibroblast integrins activate latent TGF-[beta] 1. NatureMedicine 19(12): 1567–1568.Hochman B, Nahas FX, Sobral CS et al (2008) Nerve fibres: a possible role in keloid pathogenesis. BritishJournal of Dermatology 158: 651–652.Huang C, Murphy GF, Akaish S, Ogawa R (2013) Keloids and hypertrophic scars: update and futuredirections. Plastic and Reconstructive Surgery Global Open 1(4).Ingber DE (1997) Tensegrity: the architectural basis of cellular mechanotransduction. Annual Review ofPhysiology 59(1): 575–599.Jing C, Jia-Han W, Hong-Xing Z (2010) Double-edged effects of neuropeptide substance P on repair ofcutaneous trauma. Wound Repair Regeneration 18: 319–324.Josenhans E (2007) Physiotherapeutic treatment for axillary cord formation following breast cancer surgery.ZVK Science Prize.Kania A (2012) Scars. In: Dryden T, Moyer C (eds) Massage therapy: Integrating research and practice.Human Kinetics Ch 15, pp 173–184.Karalaki M, Fili S, Philippou, A, Koutsilieris, M. (2009). Muscle regeneration: cellular and molecularevents. In vivo, 23(5): 779–796.Klingler W, Jurkat-Rott K, Lehmann-Horn F, Schleip R (2012) The role of fibrosis in Duchenne musculardystrophy. Acta Myologica 31(3): 184–95.Klingler W, Schleip R, Zorn A (2004) European Fascia Research Project Report. 5th World Congress LowBack and Pelvic Pain, Melbourne.Kobesova A et al (2007) Twenty-year-old pathogenic ‘active’ postsurgical scar: a case study of a patientwith persistent right lower quadrant pain. Journal of Manipulative and Physiological Therapeutics 30(3):234–238.Krammer A, Lu H, Isralewitz B et al (1999) Forced unfolding of the fibronectin type III module reveals atensile molecular recognition switch. Proceedings of the National Academy of Sciences in USA Feb 16;96(4): 1351–6.
Langevin HM (2006) Connective tissue: a body-wide signaling network? Med Hypotheses 66: 1074–1077.Langevin H (2010) Presentation: Ultrasound Imaging of Connective Tissue Pathology Associated withChronic Low Back Pain. 7th Interdisciplinary Congress on Low Back and Pelvic Pain, Los Angeles.Lawman MJ, Boyle MD, Gee AP, Young M (1985) Nerve growth factor accelerates the early cellular eventsassociated with wound healing. Experimental and Molecular Pathology 43(2): 274–281.Lee SS, Yosipovitch G, Chan YH, Goh CL (2004) Pruritus, pain, and small nerve fiber function in keloids:a controlled study. Journal of the American Academy of Dermatology 51: 1002–1006.Lewin GR, Mendell LM. (1993) Nerve growth factor and nociception. Trends in Neuroscience 16: 353–359.Li AK, Koroly MJ, Schattenkerk ME et al (1980) Nerve growth factor: acceleration of the rate of woundhealing in mice. Proceedings of the National Academy of Sciences in USA 77: 4379–81.Lowe JC (2009) Factors that induce fibrosis of fascial tissues. [accessed April 10, 2009 from HYPERLINK“http://www.drlowe.com” www.drlowe.com.]Marieb EN (2003) Human anatomy and physiology, 5th edn. Redwood City, CA: Benjamin CummingsPublishing, 5.Matsuda H, Koyama H, Sato H et al (1998) Role of nerve growth factor in cutaneous wound healing:accelerating effects in normal and healing-impaired diabetic mice. Journal of Experimental Medicine 187:297–306.Menke NB, Ward KR, Witten TM et al (2007). Impaired wound healing. Clinics in Dermatology 25: 19–25.Meyer-ter-Vehn T, Han H, Grehn F, Schlunck G (2011) Extracellular matrix elasticity modulates TGF-β–induced p38 activation and myofibroblast transdifferentiation in human tenon fibroblasts. InvestigativeOphthalmology and Visual Science 52(12): 9149–9155.Miyamoto J, Nagasao T, Miyamoto S et al (2009) Biomechanical analysis of stresses occurring in verticaland transverse scars on the lower leg. Plastic Reconstructive Surgery 124(6): 1974–1979.Moore ML, Dewey WS, Richard RL (2009) Rehabilitation of the burned hand. Hand Clinics 25(4): 529–541.Muangman P, Muffley LA, Anthony JP et al (2004) Nerve growth factor accelerates wound healing indiabetic mice. Wound Repair and Regeneration 12: 44–52.Ogawa R (2008) Keloid and hypertrophic scarring may result from a mechanoreceptor or mechanosensitivenociceptor disorder. Medical Hypotheses 71(4): 493–500.Ogawa R (2011) Mechanobiology of scarring. Wound Repair and Regeneration 19(s1), s2–s9.Ogawa R, Okai K, Tokumura F, Mori K et al (2012). The relationship between skin stretching/contractionand pathologic scarring: the important role of mechanical forces in keloid generation. Wound Repair andRegeneration, 20(2): 149–157.Profyris C, Tziotzios C, DoVale I (2012) Cutaneous scarring: pathophysiology, molecular mechanisms, andscar reduction therapeutics Part I. The molecular basis of scar formation. Journal of the American Academyof Dermatology 66: 1–10.Quinlan KL, Song IS, Bunnett NW et al (1998) Neuropeptide regulation of human dermal microvascularendothelial cell ICAM-1 expression and function. American Journal of Physiology 275: C1580–90.Quinlan KL, Naik SM, Cannon G et al. (1999) Substance P activates coincident NF-AT- and NF-kappa B-dependent adhesion molecule gene expression in microvascular endothelial cells through intracellularcalcium mobilization. Journal of Immunology 163(10): 5656–65.
- Page 441 and 442: fibroblast growth factor (FGF), epi
- Page 443 and 444: Clinical ConsiderationDuring wound
- Page 445 and 446: Clinical ConsiderationAlthough the
- Page 447 and 448: Pathophysiological ScarsPathophysio
- Page 450 and 451: Figure 5.3Adapted from Huang et al.
- Page 452 and 453: Pathophysiological considerationFib
- Page 454 and 455: Table 5.2Important pathophysiologic
- Page 456 and 457: According to Klingler (2012):… pa
- Page 458 and 459: Table 5.3Scar types and related ter
- Page 460 and 461: unyielding or pliable and mobile. R
- Page 462 and 463: Prolonged InflammationInflammation
- Page 464 and 465: ImmobilizationThe impact of immobil
- Page 467 and 468: Figure 5.4The fall-out associated w
- Page 469 and 470: Clinical ConsiderationHere we see t
- Page 471 and 472: Pathophysiological ConsiderationAcc
- Page 473 and 474: Pathophysiological ConsiderationNeu
- Page 475 and 476: The diverse biological effects of N
- Page 477 and 478: Clinical ConsiderationCareful appli
- Page 479 and 480: Clinical ConsiderationSome patholog
- Page 481 and 482: Pathophysiological ConsiderationSom
- Page 483 and 484: compressive effect in the keloidal
- Page 485 and 486: alterations in the mechanical envir
- Page 487 and 488: Clinical ConsiderationMechanical fo
- Page 489 and 490: Table 5.4Role of neuropeptides (NP)
- Page 491: Fitch P (2005) Scars of life. Journ
- Page 495 and 496: active scars. Journal of Bodywork a
- Page 497 and 498: trauma.
- Page 499 and 500: Clinical ConsiderationPostsurgical
- Page 501 and 502: following burn injury,bacterial col
- Page 503 and 504: Table 6.1Comparison of scars (Ogawa
- Page 505 and 506: Pathophysiological ConsiderationAcc
- Page 507 and 508: BurnsA burn injury to the skin or o
- Page 510 and 511: Figure 6.1Depth of burn trauma and
- Page 512 and 513: • Stimulate ECM formation• Regu
- Page 514 and 515: Clinical ConsiderationIt has been i
- Page 516 and 517: Clinical ConsiderationMT may be a v
- Page 518 and 519: ThermoregulationThermoregulation (t
- Page 520 and 521: from the tissues and taken up by th
- Page 522 and 523: treatment strategies are difficult
- Page 524 and 525: Clinical ConsiderationSkin rolling
- Page 526 and 527: Sequelae and ComplicationsAdvances
- Page 528 and 529: • Paresthesia - 47%• Arm/should
- Page 530 and 531: breast or around the edge of the ar
- Page 532 and 533: Radiation scarringScar tissue as a
- Page 534 and 535: Implants and painPain of fluctuatin
- Page 536 and 537: LymphedemaBreast cancer treatment o
- Page 538 and 539: volume of fluid that accumulates or
- Page 540 and 541: OneTwoThreeCommonly referred to as
rehabilitation and injury prevention. Journal of Orthopaedic and Sports Physical Therapy 40(2): 67–81.
Henderson J, Terenghi G, McGrouther DA, Ferguson MW (2006) The reinnervation pattern of wounds and
scars may explain their sensory symptoms. Journal of Plastic Reconstructive and Aesthetic Surgery 59(9):
942–50.
Henderson J, Ferguson MW, Terenghi G (2012). The feeling of healing. Plastic and Reconstructive Surgery
129 (1): 223e–224e.
Henry G, Garner, WL (2003) Inflammatory mediators in wound healing. Surgical Clinics of North America
83: 483–507.
Hinz B (2007) Formation and function of the myofibroblast during tissue repair. Journal of Investigative
Dermatology 127: 526–537.
Hinz, B (2009) Tissue stiffness, latent TGF-beta1 activation, and mechanical signal transduction:
implications for the pathogenesis and treatment of fibrosis. Current Rheumatology Reports 11(2): 120–126.
Hinz B, Celetta G, Tomasek JJ et al (2001) Alpha-smooth muscle actin expression upregulates fibroblast
contractile activity. Molecular Biology of the Cell 12: 2730–41.
Hinz B, Phan SH, Thannickal VJ et al (2012) Recent developments in myofibroblast biology: paradigms for
connective tissue remodeling. American Journal of Pathology 180 (4): 1340–1355.
Hinz B (2013) It has to be the [alpha] v: myofibroblast integrins activate latent TGF-[beta] 1. Nature
Medicine 19(12): 1567–1568.
Hochman B, Nahas FX, Sobral CS et al (2008) Nerve fibres: a possible role in keloid pathogenesis. British
Journal of Dermatology 158: 651–652.
Huang C, Murphy GF, Akaish S, Ogawa R (2013) Keloids and hypertrophic scars: update and future
directions. Plastic and Reconstructive Surgery Global Open 1(4).
Ingber DE (1997) Tensegrity: the architectural basis of cellular mechanotransduction. Annual Review of
Physiology 59(1): 575–599.
Jing C, Jia-Han W, Hong-Xing Z (2010) Double-edged effects of neuropeptide substance P on repair of
cutaneous trauma. Wound Repair Regeneration 18: 319–324.
Josenhans E (2007) Physiotherapeutic treatment for axillary cord formation following breast cancer surgery.
ZVK Science Prize.
Kania A (2012) Scars. In: Dryden T, Moyer C (eds) Massage therapy: Integrating research and practice.
Human Kinetics Ch 15, pp 173–184.
Karalaki M, Fili S, Philippou, A, Koutsilieris, M. (2009). Muscle regeneration: cellular and molecular
events. In vivo, 23(5): 779–796.
Klingler W, Jurkat-Rott K, Lehmann-Horn F, Schleip R (2012) The role of fibrosis in Duchenne muscular
dystrophy. Acta Myologica 31(3): 184–95.
Klingler W, Schleip R, Zorn A (2004) European Fascia Research Project Report. 5th World Congress Low
Back and Pelvic Pain, Melbourne.
Kobesova A et al (2007) Twenty-year-old pathogenic ‘active’ postsurgical scar: a case study of a patient
with persistent right lower quadrant pain. Journal of Manipulative and Physiological Therapeutics 30(3):
234–238.
Krammer A, Lu H, Isralewitz B et al (1999) Forced unfolding of the fibronectin type III module reveals a
tensile molecular recognition switch. Proceedings of the National Academy of Sciences in USA Feb 16;
96(4): 1351–6.