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[libribook.com] Traumatic Scar Tissue Management 1st Edition
Clinical ConsiderationAs noted above, undue mechanical tension is considered the mostsignificant causative factor associated with pathophysiological scarformation. Therefore it is suggested that stabilizing a scar during formationvia the control of mechanotransduction-mediated mechanical tension –involving MFBs – appears to be a primary target to prevent or reduce theincidence of pathophysiological scars. Additionally, sedation of exposednerve endings and attenuation of neurogenic inflammation are alsoimportant scar management considerations, as well as proper wounddressings to prevent infection and maintain proper wound hydration. Amultimodal approach to attenuating scar tensional stress and undue sensorystimulation will likely yield the best possible outcomes (Widgerow 2013).
Clinical ConsiderationCareful application of massage therapy during the repair process candecrease undue tissue tension. A decrease in mechanical tension canattenuate tension-driven responses. In addition to the influence ofmechanoreceptors on pain (via both ascending and descending pathways),mechanical stretching of fibroblasts can alter interstitial osmotic pressureas well as increase blood flow, thereby reducing concentrations of proinflammatorycytokines and reduce sensitization of peripheral nociceptors,by diluting the concentration of sensitizing agents (Fryer & Fossum 2009).
- Page 426 and 427: Damasio AR, Grabowski TJ, Bechara A
- Page 428 and 429: Magee DJ (2008) Orthopedic physical
- Page 430 and 431: Stecco C, Porzionato A, Macchi V et
- Page 432 and 433: CHAPTER 5Wound healing and scarsNev
- Page 434 and 435: Wound HealingWound healing, a compl
- Page 436 and 437: Table 5.1Stages of wound healing
- Page 438: Clinical ConsiderationBecause thera
- Page 441 and 442: fibroblast growth factor (FGF), epi
- Page 443 and 444: Clinical ConsiderationDuring wound
- Page 445 and 446: Clinical ConsiderationAlthough the
- Page 447 and 448: Pathophysiological ScarsPathophysio
- Page 450 and 451: Figure 5.3Adapted from Huang et al.
- Page 452 and 453: Pathophysiological considerationFib
- Page 454 and 455: Table 5.2Important pathophysiologic
- Page 456 and 457: According to Klingler (2012):… pa
- Page 458 and 459: Table 5.3Scar types and related ter
- Page 460 and 461: unyielding or pliable and mobile. R
- Page 462 and 463: Prolonged InflammationInflammation
- Page 464 and 465: ImmobilizationThe impact of immobil
- Page 467 and 468: Figure 5.4The fall-out associated w
- Page 469 and 470: Clinical ConsiderationHere we see t
- Page 471 and 472: Pathophysiological ConsiderationAcc
- Page 473 and 474: Pathophysiological ConsiderationNeu
- Page 475: The diverse biological effects of N
- Page 479 and 480: Clinical ConsiderationSome patholog
- Page 481 and 482: Pathophysiological ConsiderationSom
- Page 483 and 484: compressive effect in the keloidal
- Page 485 and 486: alterations in the mechanical envir
- Page 487 and 488: Clinical ConsiderationMechanical fo
- Page 489 and 490: Table 5.4Role of neuropeptides (NP)
- Page 491 and 492: Fitch P (2005) Scars of life. Journ
- Page 493 and 494: Langevin HM (2006) Connective tissu
- Page 495 and 496: active scars. Journal of Bodywork a
- Page 497 and 498: trauma.
- Page 499 and 500: Clinical ConsiderationPostsurgical
- Page 501 and 502: following burn injury,bacterial col
- Page 503 and 504: Table 6.1Comparison of scars (Ogawa
- Page 505 and 506: Pathophysiological ConsiderationAcc
- Page 507 and 508: BurnsA burn injury to the skin or o
- Page 510 and 511: Figure 6.1Depth of burn trauma and
- Page 512 and 513: • Stimulate ECM formation• Regu
- Page 514 and 515: Clinical ConsiderationIt has been i
- Page 516 and 517: Clinical ConsiderationMT may be a v
- Page 518 and 519: ThermoregulationThermoregulation (t
- Page 520 and 521: from the tissues and taken up by th
- Page 522 and 523: treatment strategies are difficult
- Page 524 and 525: Clinical ConsiderationSkin rolling
Clinical Consideration
Careful application of massage therapy during the repair process can
decrease undue tissue tension. A decrease in mechanical tension can
attenuate tension-driven responses. In addition to the influence of
mechanoreceptors on pain (via both ascending and descending pathways),
mechanical stretching of fibroblasts can alter interstitial osmotic pressure
as well as increase blood flow, thereby reducing concentrations of proinflammatory
cytokines and reduce sensitization of peripheral nociceptors,
by diluting the concentration of sensitizing agents (Fryer & Fossum 2009).
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