[libribook.com] Traumatic Scar Tissue Management 1st Edition
According to Klingler (2012):… painful contractures resulting in limited/reduced range of motion are often associated withrigid collagenous tissue within and surrounding skeletal muscle, as well as other CTs involvedin force transmission (e.g. retinaculae, thoracolumbar fascia, tendon).HypertrophicscarKeloid scarAtrophic scarWidespreadscarAn elevated, excessive scar that is limited to the original boundaries of the incision or wound(Bordoni & Zanier 2014, Zhu et al. 2013). Common features include raised, thickened, red andsometimes ropelike scarring. Hypertrophic scars can lead to deformity, disfigurement and loss offunction and can take weeks to form. Scars formed at the fringe of skin grafts are prone tohypertrophy (e.g. those associated with deep second-degree burns and other scars requiring grafts).Both hypertrophic and keloid scars occur more commonly in the regions of the anterior chest,shoulder/periscapular, lower abdomen and ear lobes (Ogawa 2008, Bordoni & Zanier 2014) (seeFig. 5.1)An abnormal scar that forms at the site of an injury or an incision and spreads beyond the bordersof the original lesion. The scar is made up of a swirling mass of collagen fibers and fibroblasts.Keloid scars reach out like little fingers and extend well beyond the original injured area. Commonfeatures include thickened, raised, itchy clusters of scar tissue that grow beyond the edges of thewound or incision and are often red or darker in color than surrounding skin. Keloids occur whenthe body continues to produce the tough, fibrous collagen after a wound has healed. Keloids mostcommonly appear over the sternum, ears and shoulders and occur more in dark-skinned people.The tendency to develop keloid scars lessons with age. Some scars may start as hypertrophic andeventually develop into keloid (see Fig. 5.2)Common presentation is a fibrotic, cutaneous depression displaying a sunken or pitted appearance(Weiss et al. 2010)Can appear when the fine lines of surgical scars gradually become stretched and widened. Thesetypically flat, pale, soft, symptomless scars are more common following knee or shoulder surgery(Rudolph 1987). Stretch marks (abdominal striae) are variants of widespread scars involving injuryto the dermis and subcutaneous tissues without breaching the epidermis. There is no elevation,thickening, or nodularity in mature widespread scars, which distinguishes them from hypertrophicscars (Bayat et al. 2003)
Clinical ConsiderationAs discussed previously; if tensional homeostasis can be achieved/restored, thereby mediating superfluous collagenproliferation, it seems plausible that the risk of pathophysiological scarring can be reduced
- Page 406 and 407: Example 1Hypersensitized nerve fibe
- Page 408 and 409: Clinical ConsiderationIt is suggest
- Page 410 and 411: Clinical ConsiderationNeuropathic p
- Page 412 and 413: Wound HealingThe NS plays an import
- Page 414 and 415: Clinical ConsiderationNeural and ci
- Page 416 and 417: Compression SyndromesAlthough perip
- Page 418 and 419: Pathophysiological ConsiderationIf
- Page 420 and 421: Pathophysiological ConsiderationUni
- Page 422 and 423: Clinical ConsiderationAs is the cas
- Page 424 and 425: Pathophysiological ConsiderationFas
- Page 426 and 427: Damasio AR, Grabowski TJ, Bechara A
- Page 428 and 429: Magee DJ (2008) Orthopedic physical
- Page 430 and 431: Stecco C, Porzionato A, Macchi V et
- Page 432 and 433: CHAPTER 5Wound healing and scarsNev
- Page 434 and 435: Wound HealingWound healing, a compl
- Page 436 and 437: Table 5.1Stages of wound healing
- Page 438: Clinical ConsiderationBecause thera
- Page 441 and 442: fibroblast growth factor (FGF), epi
- Page 443 and 444: Clinical ConsiderationDuring wound
- Page 445 and 446: Clinical ConsiderationAlthough the
- Page 447 and 448: Pathophysiological ScarsPathophysio
- Page 450 and 451: Figure 5.3Adapted from Huang et al.
- Page 452 and 453: Pathophysiological considerationFib
- Page 454 and 455: Table 5.2Important pathophysiologic
- Page 458 and 459: Table 5.3Scar types and related ter
- Page 460 and 461: unyielding or pliable and mobile. R
- Page 462 and 463: Prolonged InflammationInflammation
- Page 464 and 465: ImmobilizationThe impact of immobil
- Page 467 and 468: Figure 5.4The fall-out associated w
- Page 469 and 470: Clinical ConsiderationHere we see t
- Page 471 and 472: Pathophysiological ConsiderationAcc
- Page 473 and 474: Pathophysiological ConsiderationNeu
- Page 475 and 476: The diverse biological effects of N
- Page 477 and 478: Clinical ConsiderationCareful appli
- Page 479 and 480: Clinical ConsiderationSome patholog
- Page 481 and 482: Pathophysiological ConsiderationSom
- Page 483 and 484: compressive effect in the keloidal
- Page 485 and 486: alterations in the mechanical envir
- Page 487 and 488: Clinical ConsiderationMechanical fo
- Page 489 and 490: Table 5.4Role of neuropeptides (NP)
- Page 491 and 492: Fitch P (2005) Scars of life. Journ
- Page 493 and 494: Langevin HM (2006) Connective tissu
- Page 495 and 496: active scars. Journal of Bodywork a
- Page 497 and 498: trauma.
- Page 499 and 500: Clinical ConsiderationPostsurgical
- Page 501 and 502: following burn injury,bacterial col
- Page 503 and 504: Table 6.1Comparison of scars (Ogawa
- Page 505 and 506: Pathophysiological ConsiderationAcc
Clinical Consideration
As discussed previously; if tensional homeostasis can be achieved/restored, thereby mediating superfluous collagen
proliferation, it seems plausible that the risk of pathophysiological scarring can be reduced