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[libribook.com] Traumatic Scar Tissue Management 1st Edition
Pathophysiological ConsiderationUnimpeded nerve conduction is essential to trophic effect (i.e. control andmaintenance of cellular function). When conduction is impeded or blocked,innervated structures become atrophic. Atrophic structures can becomehighly irritable and develop hypersensitivity (Cannon & Rosenblueth1949).Double-crush injuryWith double-crush injury, an axon compressed in one region can becomesusceptible to injury at another site (e.g. development of a cervical radiculopathyin conjunction with carpal tunnel syndrome) (Upton & McComas 1973,Moghtaderi & Izadi 2008, Coppieters & Nee 2012). Mechanisms similar to thoseassociated with sensitization are implicated.
Clinical ConsiderationWith double-crush injuries, failure to treat at multiple levels will result infailure to relieve the patient’s symptoms (Mackinnon 2002). This supportsthe importance of local and global treatment considerations (e.g. along thenerve path, corresponding myofascial meridians/myokinetic chains andneighboring tissue).
- Page 370 and 371: Clinical ConsiderationAlthough musc
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- Page 374 and 375: Example 2Ruffini stimulation result
- Page 376 and 377: Table 4.1Summary of receptor typolo
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- Page 380 and 381: NS FunctionThe primary functions of
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- Page 392 and 393: Table 4.2Important pain terms. Vari
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- Page 396 and 397: disorders (diabetes mellitus), drug
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- Page 404 and 405: ExampleHypersensitive nerves (assoc
- Page 406 and 407: Example 1Hypersensitized nerve fibe
- Page 408 and 409: Clinical ConsiderationIt is suggest
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- Page 412 and 413: Wound HealingThe NS plays an import
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- Page 418 and 419: Pathophysiological ConsiderationIf
- Page 422 and 423: Clinical ConsiderationAs is the cas
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- Page 426 and 427: Damasio AR, Grabowski TJ, Bechara A
- Page 428 and 429: Magee DJ (2008) Orthopedic physical
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- Page 432 and 433: CHAPTER 5Wound healing and scarsNev
- Page 434 and 435: Wound HealingWound healing, a compl
- Page 436 and 437: Table 5.1Stages of wound healing
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- Page 450 and 451: Figure 5.3Adapted from Huang et al.
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- Page 454 and 455: Table 5.2Important pathophysiologic
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- Page 458 and 459: Table 5.3Scar types and related ter
- Page 460 and 461: unyielding or pliable and mobile. R
- Page 462 and 463: Prolonged InflammationInflammation
- Page 464 and 465: ImmobilizationThe impact of immobil
- Page 467 and 468: Figure 5.4The fall-out associated w
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Pathophysiological Consideration
Unimpeded nerve conduction is essential to trophic effect (i.e. control and
maintenance of cellular function). When conduction is impeded or blocked,
innervated structures become atrophic. Atrophic structures can become
highly irritable and develop hypersensitivity (Cannon & Rosenblueth
1949).
Double-crush injury
With double-crush injury, an axon compressed in one region can become
susceptible to injury at another site (e.g. development of a cervical radiculopathy
in conjunction with carpal tunnel syndrome) (Upton & McComas 1973,
Moghtaderi & Izadi 2008, Coppieters & Nee 2012). Mechanisms similar to those
associated with sensitization are implicated.
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