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[libribook.com] Traumatic Scar Tissue Management 1st Edition
ExampleHypersensitive nerves (associated with muscle) are prone to spontaneouselectrical impulses that trigger false pain signals or provoke involuntarymuscular activity such as increased tone or tension (Culp & Ochoa 1982).In turn, this increase in tone/tension can subsequently increase the risk ofsecondary injury (e.g. sprain/strain, tendinopathies, MTrPs).
Clinical ConsiderationHypersensitive afferents can display other forms of aberrant behavior.
- Page 354 and 355: Clinical ConsiderationSNS activatio
- Page 357 and 358: Figure 4.8Anterior and posterior di
- Page 360 and 361: Figure 4.9Neurofascial envelopes.
- Page 362 and 363: Pathophysiological ConsiderationA n
- Page 364 and 365: Clinical ConsiderationThere is an e
- Page 366 and 367: Clinical ConsiderationDiane Jacobs
- Page 368 and 369: Clinical ConsiderationIt appears th
- Page 370 and 371: Clinical ConsiderationAlthough musc
- Page 372 and 373: Clinical ConsiderationThe form of s
- Page 374 and 375: Example 2Ruffini stimulation result
- Page 376 and 377: Table 4.1Summary of receptor typolo
- Page 378 and 379: • Therapeutic outcome include enh
- Page 380 and 381: NS FunctionThe primary functions of
- Page 382 and 383: Clinical ConsiderationMechanorecept
- Page 384 and 385: Clinical ConsiderationIn addition t
- Page 386 and 387: Clinical ConsiderationManual techni
- Page 388 and 389: large enough, a voltage spike is pr
- Page 390 and 391: PathophysiologicalconsiderationUnde
- Page 392 and 393: Table 4.2Important pain terms. Vari
- Page 394 and 395: Clinical ConsiderationAccording to
- Page 396 and 397: disorders (diabetes mellitus), drug
- Page 398 and 399: forms of negative plasticity includ
- Page 400 and 401: Central and peripheral sensitizatio
- Page 402 and 403: Clinical ConsiderationFollowing per
- Page 406 and 407: Example 1Hypersensitized nerve fibe
- Page 408 and 409: Clinical ConsiderationIt is suggest
- Page 410 and 411: Clinical ConsiderationNeuropathic p
- Page 412 and 413: Wound HealingThe NS plays an import
- Page 414 and 415: Clinical ConsiderationNeural and ci
- Page 416 and 417: Compression SyndromesAlthough perip
- Page 418 and 419: Pathophysiological ConsiderationIf
- Page 420 and 421: Pathophysiological ConsiderationUni
- Page 422 and 423: Clinical ConsiderationAs is the cas
- Page 424 and 425: Pathophysiological ConsiderationFas
- Page 426 and 427: Damasio AR, Grabowski TJ, Bechara A
- Page 428 and 429: Magee DJ (2008) Orthopedic physical
- Page 430 and 431: Stecco C, Porzionato A, Macchi V et
- Page 432 and 433: CHAPTER 5Wound healing and scarsNev
- Page 434 and 435: Wound HealingWound healing, a compl
- Page 436 and 437: Table 5.1Stages of wound healing
- Page 438: Clinical ConsiderationBecause thera
- Page 441 and 442: fibroblast growth factor (FGF), epi
- Page 443 and 444: Clinical ConsiderationDuring wound
- Page 445 and 446: Clinical ConsiderationAlthough the
- Page 447 and 448: Pathophysiological ScarsPathophysio
- Page 450 and 451: Figure 5.3Adapted from Huang et al.
- Page 452 and 453: Pathophysiological considerationFib
Example
Hypersensitive nerves (associated with muscle) are prone to spontaneous
electrical impulses that trigger false pain signals or provoke involuntary
muscular activity such as increased tone or tension (Culp & Ochoa 1982).
In turn, this increase in tone/tension can subsequently increase the risk of
secondary injury (e.g. sprain/strain, tendinopathies, MTrPs).
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