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[libribook.com] Traumatic Scar Tissue Management 1st Edition
Clinical ConsiderationMechanoreceptors in skin gather and relay any external (touch)information being provided. In addition to this information being relayed tothe brain, the skin can communicate globally across its own expanse anddeeply as it is contiguous with the various layers of fascia, and all that thefascia is connected to (Abu-Hijleh & Harris 2007, Chaitow 2014). TheECM within the hypodermis or superficial fascia sends out projectionstowards the surface of the skin providing a communication route betweenthe deeper layers of fascia and the surface. Therefore movement, touch orother forms of stimuli can be communicated across all layers, fromsuperficial to deep, and from the subterranean to the surface (Paoletti2006). This connection pathway provides a plausible mechanism forvarious manual therapy approaches to achieve the ‘tissue release’ or tonuschanges we feel beneath our hands during treatment. Release can bemediated by the nervous system (responding to mechanoreceptorstimulation) or via the mechanotransduction pathway, as discussed inChapter 2 (Schleip 2003a, 2003b, Langevin 2006).
Pathophysiological ConsiderationTrauma, mechanoreceptors and proprioceptive disinformationTrauma, including pathophysiological scars, can result in aberrantmechanoreceptor function. Fibrosis and adhesions that occur withpathological scars can lead to innocuous mechanical stimuli beingperceived as noxious, resulting in the subsequent relay of incoherent ormisinformation to local, regional, or central nerve centers (i.e.proprioceptive disinformation). Under- or overestimation of the mechanicalstimuli received results in inappropriate muscular reactions, such asinterference with recruitment and timing of recruitment, which endangersequilibrium, coordination and stability. Consequently, proprioceptivedisinformation can adversely impact the rehabilitative process or increasethe incidence of comorbid injuries (e.g. falling with subsequent sprains orfracture).The interoceptive system, considered the substrate of recognition of self, plays afundamental role in the relationship between one’s subjective state of well-beingand physiological health. Volitional cortical control in humans can directlymodify homeostatic integration (Petrovic et al. 2002, Craig 2003, Critchley et al.2002, Damasio et al. 2000, Derbyshire et al. 2003). This supports that how wefeel about how we feel can influence how we function, right down to thephysiological level.
- Page 331 and 332: CHAPTER 4NeurologyEach human nervou
- Page 333: NS StructureThe human NS comprises
- Page 337 and 338: Figure 4.1B Functional composition
- Page 339 and 340: Pathophysiological ConsiderationWhe
- Page 341 and 342: Figure 4.2Dorsal and ventral compon
- Page 344 and 345: Figure 4.3Neuron anatomy: most neur
- Page 346 and 347: Figure 4.4Classic axon to dendrite
- Page 349: Figure 4.6PNS efferent (motor) and
- Page 352 and 353: Clinical ConsiderationPSNS afferent
- Page 354 and 355: Clinical ConsiderationSNS activatio
- Page 357 and 358: Figure 4.8Anterior and posterior di
- Page 360 and 361: Figure 4.9Neurofascial envelopes.
- Page 362 and 363: Pathophysiological ConsiderationA n
- Page 364 and 365: Clinical ConsiderationThere is an e
- Page 366 and 367: Clinical ConsiderationDiane Jacobs
- Page 368 and 369: Clinical ConsiderationIt appears th
- Page 370 and 371: Clinical ConsiderationAlthough musc
- Page 372 and 373: Clinical ConsiderationThe form of s
- Page 374 and 375: Example 2Ruffini stimulation result
- Page 376 and 377: Table 4.1Summary of receptor typolo
- Page 378 and 379: • Therapeutic outcome include enh
- Page 380 and 381: NS FunctionThe primary functions of
- Page 384 and 385: Clinical ConsiderationIn addition t
- Page 386 and 387: Clinical ConsiderationManual techni
- Page 388 and 389: large enough, a voltage spike is pr
- Page 390 and 391: PathophysiologicalconsiderationUnde
- Page 392 and 393: Table 4.2Important pain terms. Vari
- Page 394 and 395: Clinical ConsiderationAccording to
- Page 396 and 397: disorders (diabetes mellitus), drug
- Page 398 and 399: forms of negative plasticity includ
- Page 400 and 401: Central and peripheral sensitizatio
- Page 402 and 403: Clinical ConsiderationFollowing per
- Page 404 and 405: ExampleHypersensitive nerves (assoc
- Page 406 and 407: Example 1Hypersensitized nerve fibe
- Page 408 and 409: Clinical ConsiderationIt is suggest
- Page 410 and 411: Clinical ConsiderationNeuropathic p
- Page 412 and 413: Wound HealingThe NS plays an import
- Page 414 and 415: Clinical ConsiderationNeural and ci
- Page 416 and 417: Compression SyndromesAlthough perip
- Page 418 and 419: Pathophysiological ConsiderationIf
- Page 420 and 421: Pathophysiological ConsiderationUni
- Page 422 and 423: Clinical ConsiderationAs is the cas
- Page 424 and 425: Pathophysiological ConsiderationFas
- Page 426 and 427: Damasio AR, Grabowski TJ, Bechara A
- Page 428 and 429: Magee DJ (2008) Orthopedic physical
- Page 430 and 431: Stecco C, Porzionato A, Macchi V et
Clinical Consideration
Mechanoreceptors in skin gather and relay any external (touch)
information being provided. In addition to this information being relayed to
the brain, the skin can communicate globally across its own expanse and
deeply as it is contiguous with the various layers of fascia, and all that the
fascia is connected to (Abu-Hijleh & Harris 2007, Chaitow 2014). The
ECM within the hypodermis or superficial fascia sends out projections
towards the surface of the skin providing a communication route between
the deeper layers of fascia and the surface. Therefore movement, touch or
other forms of stimuli can be communicated across all layers, from
superficial to deep, and from the subterranean to the surface (Paoletti
2006). This connection pathway provides a plausible mechanism for
various manual therapy approaches to achieve the ‘tissue release’ or tonus
changes we feel beneath our hands during treatment. Release can be
mediated by the nervous system (responding to mechanoreceptor
stimulation) or via the mechanotransduction pathway, as discussed in
Chapter 2 (Schleip 2003a, 2003b, Langevin 2006).
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