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[libribook.com] Traumatic Scar Tissue Management 1st Edition
Clinical ConsiderationPSNS afferent myelinated fibers travel from the viscera to their cell bodieslocated either in the sensory ganglia of the cranial nerves or in the posteriorroot ganglia of the sacrospinal nerves. The central axons then enter theCNS and take part in the formation of local reflex arcs or pass to highercenters of the ANS. The location of the sensory ganglia gives rise to thebasic anatomical principle of craniosacral work – with intended outcomesmediated by the PSNS (Upledger 1987, Frymann 1988, Schleip 2003a,2003b, Dorko 2003, Minasny 2009).]SNSThe SNS mediates activity associated with emergency or stress response,commonly referred to as the fight, flight or freeze aspect of the nervous system.The SNS facilitates activity such as accelerated heart rate, elevated bloodpressure and constriction of peripheral blood vessels when the need arises. Bloodfrom the skin and intestines is redirected to the brain, heart and skeletal muscleto enhance chances of survival (e.g. fight or flight). The primaryneurotransmitter associated with the SNS is norepinephrine. Norepinephrine canstimulate the adrenal medulla, triggering the release of noradrenalin andadrenalin, which are known to prolong the effects of sympathetic stimulation.The impact of prolonged sympathetic stimulation associated with trauma will becovered in greater detail in Chapter 7.
Clinical ConsiderationStress and the myofascial system – psychological distress or anxiety – hasclearly been identified as a source of ‘unnecessary’ muscular tension: theconfusing intermediate between a non-voluntary muscle contraction(spasm) and viscoelastic tension (a fascial property) showing noelectromyography (EMG) activity (Simon & Mense 2007). According toChaitow (2007):the shortened fibers of the soft tissues may be the result of acombination of structural anomalies, trauma, and/or physical oremotional stress, and are always influenced by underlying nutritionaland behavioral elements. Some of these shortened fibers and tenderspots (i.e. trigger points) may be the source of reflex symptoms andpain. Such soft tissue dysfunctions respond to manual pressure in theform of modalities like MT.
- Page 302 and 303: Lymphatic Drainage and TransportIn
- Page 304 and 305: SuperficialThe superficial layer is
- Page 306: Primary upper lymphatic structuresT
- Page 309 and 310: Primary lower lymphatic structuresT
- Page 311 and 312: Left side lymphatic drainageThe lef
- Page 313 and 314: Lymphatic System FunctionsA brief o
- Page 316 and 317: Fig 3.5Brain lymphatic vessels.Give
- Page 318: ImmunityLymph nodes play an importa
- Page 321 and 322: Wound HealingAs with the blood vasc
- Page 323 and 324: Clinical ConsiderationFour continuo
- Page 325 and 326: Lymphatic InadequacyInadequacy in t
- Page 327 and 328: EdemaEdema - the medical term for s
- Page 329 and 330: Lymphatic treatment protocols will
- Page 331 and 332: CHAPTER 4NeurologyEach human nervou
- Page 333: NS StructureThe human NS comprises
- Page 337 and 338: Figure 4.1B Functional composition
- Page 339 and 340: Pathophysiological ConsiderationWhe
- Page 341 and 342: Figure 4.2Dorsal and ventral compon
- Page 344 and 345: Figure 4.3Neuron anatomy: most neur
- Page 346 and 347: Figure 4.4Classic axon to dendrite
- Page 349: Figure 4.6PNS efferent (motor) and
- Page 354 and 355: Clinical ConsiderationSNS activatio
- Page 357 and 358: Figure 4.8Anterior and posterior di
- Page 360 and 361: Figure 4.9Neurofascial envelopes.
- Page 362 and 363: Pathophysiological ConsiderationA n
- Page 364 and 365: Clinical ConsiderationThere is an e
- Page 366 and 367: Clinical ConsiderationDiane Jacobs
- Page 368 and 369: Clinical ConsiderationIt appears th
- Page 370 and 371: Clinical ConsiderationAlthough musc
- Page 372 and 373: Clinical ConsiderationThe form of s
- Page 374 and 375: Example 2Ruffini stimulation result
- Page 376 and 377: Table 4.1Summary of receptor typolo
- Page 378 and 379: • Therapeutic outcome include enh
- Page 380 and 381: NS FunctionThe primary functions of
- Page 382 and 383: Clinical ConsiderationMechanorecept
- Page 384 and 385: Clinical ConsiderationIn addition t
- Page 386 and 387: Clinical ConsiderationManual techni
- Page 388 and 389: large enough, a voltage spike is pr
- Page 390 and 391: PathophysiologicalconsiderationUnde
- Page 392 and 393: Table 4.2Important pain terms. Vari
- Page 394 and 395: Clinical ConsiderationAccording to
- Page 396 and 397: disorders (diabetes mellitus), drug
- Page 398 and 399: forms of negative plasticity includ
- Page 400 and 401: Central and peripheral sensitizatio
Clinical Consideration
Stress and the myofascial system – psychological distress or anxiety – has
clearly been identified as a source of ‘unnecessary’ muscular tension: the
confusing intermediate between a non-voluntary muscle contraction
(spasm) and viscoelastic tension (a fascial property) showing no
electromyography (EMG) activity (Simon & Mense 2007). According to
Chaitow (2007):
the shortened fibers of the soft tissues may be the result of a
combination of structural anomalies, trauma, and/or physical or
emotional stress, and are always influenced by underlying nutritional
and behavioral elements. Some of these shortened fibers and tender
spots (i.e. trigger points) may be the source of reflex symptoms and
pain. Such soft tissue dysfunctions respond to manual pressure in the
form of modalities like MT.
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