[libribook.com] Traumatic Scar Tissue Management 1st Edition

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Figure 2.3Fascial membranes and rentinacula cutis fibers. Cross-section from the skin to musculature, showingfascial membranes and retinacula cutis fibers (RCF). (Adapted from Stecco et al. 2013.)Retinacula cutis fibers (RCF)Fibrous strands that invest and are continuous throughout the skin, superficialand deep fasciae layers. RCF play a role in tissue connectivity and mobility.

Clinical ConsiderationWhen thickened, as a result of injury or trauma, these vertical septa canrestrict and impact function. Any undue ‘tugging’ of bound tissue (e.g.during ‘normal’ movement) can lead to hypersensitization and consequentpain (Stecco 2004, 2009, Muscolino 2012).Deep/axial fascia (DF)Deeper scars (e.g. following surgery or due to penetrating puncture wounds) willimpact the deep fascia layer and, potentially, the underlying muscular fascia (epi,peri and endomysium).Generally speaking, DF presents throughout the body as a multilayerorganization, typically 2–3 dense collagen bundle layers interspersed with looseCT layers that contain collagen, adipocytes and are rich in HA. The dense layersserve to augment force transmission and the loose layers augment slide/glide.In each dense layer the collagen bundles are arranged in parallel with adjacent(above or below) layers arranged at a 78° angle to one another. Thisconfiguration (interspersed with sliding layers) allows for multidirectionalmovement and the ability of fascia to counter/resist tension multidirectionally(see Fig. 2.4).DF displays some distinct regional differences:

Clinical Consideration

When thickened, as a result of injury or trauma, these vertical septa can

restrict and impact function. Any undue ‘tugging’ of bound tissue (e.g.

during ‘normal’ movement) can lead to hypersensitization and consequent

pain (Stecco 2004, 2009, Muscolino 2012).

Deep/axial fascia (DF)

Deeper scars (e.g. following surgery or due to penetrating puncture wounds) will

impact the deep fascia layer and, potentially, the underlying muscular fascia (epi,

peri and endomysium).

Generally speaking, DF presents throughout the body as a multilayer

organization, typically 2–3 dense collagen bundle layers interspersed with loose

CT layers that contain collagen, adipocytes and are rich in HA. The dense layers

serve to augment force transmission and the loose layers augment slide/glide.

In each dense layer the collagen bundles are arranged in parallel with adjacent

(above or below) layers arranged at a 78° angle to one another. This

configuration (interspersed with sliding layers) allows for multidirectional

movement and the ability of fascia to counter/resist tension multidirectionally

(see Fig. 2.4).

DF displays some distinct regional differences:

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