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[libribook.com] Traumatic Scar Tissue Management 1st Edition
NeuroplasticityAn adaptation trait of the nervous system (NS) that enables modification offunction and structure in response to demands via the strengthening, weakening,pruning, or adding of synaptic connections and by promoting neurogenesis. Akey principle of neuroplasticity is that brain activity (increased or decreased)promotes brain adaptation and reorganization.
NeuropeptidesSignaling molecules that influence neuronal and brain activity. Neuropeptidesreleased during wound healing modulate a number of important aspects of theprocess such as cell proliferation, cytokine and growth factor production, antigenpresentation, mast cell degradation, increased vascular permeability andneovascularization.
- Page 99 and 100: HomeostasisState of physiological e
- Page 101 and 102: HyperalgesiaHeightened pain sensati
- Page 103 and 104: HyperexcitationNeurons firing too e
- Page 105 and 106: Hypertrophic scarA thickened, red a
- Page 107 and 108: HysteresisA property of systems (ti
- Page 109 and 110: Indirect release techniqueMechanica
- Page 111 and 112: Informed consentProcess involving t
- Page 113 and 114: Intense versus invasiveAn important
- Page 115 and 116: InteroceptionIncludes a wide range
- Page 117 and 118: KeratinA structural protein that pr
- Page 119 and 120: KininsPeptides (e.g. bradykinin) pr
- Page 121 and 122: Linking fasciaPlays a role in augme
- Page 123 and 124: Lymph nodesFilter and clean the lym
- Page 125 and 126: LymphangiogenesisFormation of new l
- Page 127 and 128: Lymphatic drainageThe movement of f
- Page 129 and 130: Lymphatic returnVolume of lymph ret
- Page 131 and 132: Lymphatic transportThe movement of
- Page 133 and 134: Lymphatic vesselsTri-laminar vessel
- Page 135 and 136: LymphocyteThe main cell type found
- Page 137 and 138: MacrophageA type of phagocytic whit
- Page 139 and 140: MastectomySurgical removal of all b
- Page 141 and 142: MechanotransductionThe mechanism by
- Page 143 and 144: MucopolysaccharidesSee GAGs.Myofasc
- Page 145 and 146: Nerve receptorsNerve terminations t
- Page 147 and 148: NeurofasciaTri-laminar, continuous
- Page 149: Neuropathic painPain due to damage
- Page 153 and 154: NociceptionThreatening stimuli acti
- Page 155 and 156: Nociceptive painPain that arises fr
- Page 157 and 158: OxytocinHormone that plays role in
- Page 159 and 160: ParesthesiaAltered sensations; e.g.
- Page 161 and 162: Peripheral sensitizationIncreased r
- Page 163 and 164: PerineuriumFascia surrounding/inves
- Page 165 and 166: Pitting edemaAn edematous region wh
- Page 167 and 168: Proprioceptive disinformationRecept
- Page 169 and 170: Provisional matrixDuring the early
- Page 171 and 172: Pumping techniqueA lymphatic techni
- Page 173 and 174: Referred painPain perceived at a lo
- Page 175 and 176: RetinaculaeDense fascial bands that
- Page 177 and 178: ScarMark left in various tissues or
- Page 179 and 180: Secondary lymphedemaOccurs as a res
- Page 181 and 182: Self careThose practices and activi
- Page 183 and 184: Selfcare managementResponse to sens
- Page 185 and 186: SensitizationChanges in the PNS or
- Page 187 and 188: SerotoninNeurotransmitter involved
- Page 189 and 190: Shear techniqueA therapeutic loadin
- Page 191 and 192: Skin graftMedical procedure where a
- Page 193 and 194: SomatizationA tendency to experienc
- Page 195 and 196: State anxietyThe experience of unpl
- Page 197 and 198: Stress response adaptationAny of th
- Page 199 and 200: Stress responseAny cognitive, physi
Neuropeptides
Signaling molecules that influence neuronal and brain activity. Neuropeptides
released during wound healing modulate a number of important aspects of the
process such as cell proliferation, cytokine and growth factor production, antigen
presentation, mast cell degradation, increased vascular permeability and
neovascularization.
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