[libribook.com] Traumatic Scar Tissue Management 1st Edition
HyperarousalTerm generally used to describe when the ANS is in a state of hypervigilance orhyperactivation.
HyperexcitationNeurons firing too easily or too often, which in turn can make somethingperceived as painful feel even worse (hyperalgesia), or it can make things hurtthat should not (allodynia). Injury-induced hyperexcitability is not limited tonociceptors. Hyperexcitability can also develop in myelinated afferents thatnormally convey innocuous information (e.g. normal movement and touch) andunder neuropathic conditions, mechanical allodynia can occur.
- Page 51 and 52: Compassion fatigueThe profound emot
- Page 53 and 54: Compression techniqueA therapeutic
- Page 55 and 56: Consolidated edemaChronic stage ede
- Page 57 and 58: CortisolHormone released in respons
- Page 59 and 60: CreepA time-dependent response of v
- Page 61 and 62: DebridementRemoval of foreign mater
- Page 63 and 64: Degloving injuryA form of avulsion
- Page 65 and 66: DermatomeAreas of skin supplied by
- Page 67 and 68: Direct release techniqueCommonly us
- Page 69 and 70: Dry needlingA procedure involving i
- Page 71 and 72: Empathy strainA healthcare provider
- Page 73 and 74: EndoneuriumFascia surrounding indiv
- Page 75 and 76: Endothelial cellsSimple squamous ce
- Page 77 and 78: EndoscopeAn instrument that is used
- Page 79 and 80: EpidermisThe tough, outermost layer
- Page 81 and 82: EpineuriumOutermost covering of fas
- Page 83 and 84: Epithelial cellsCells that line the
- Page 85 and 86: FasciaFibrocollagenous CT, its morp
- Page 87 and 88: FibrinFibrous protein involved bloo
- Page 89 and 90: FibroblastsSynthesize the component
- Page 91 and 92: FibrosisA process culminating in th
- Page 93 and 94: Fluid techniquesTechniques employed
- Page 95 and 96: Gentle circles techniqueCircular mo
- Page 97 and 98: Gross stretch techniqueCombination
- Page 99 and 100: HomeostasisState of physiological e
- Page 101: HyperalgesiaHeightened pain sensati
- Page 105 and 106: Hypertrophic scarA thickened, red a
- Page 107 and 108: HysteresisA property of systems (ti
- Page 109 and 110: Indirect release techniqueMechanica
- Page 111 and 112: Informed consentProcess involving t
- Page 113 and 114: Intense versus invasiveAn important
- Page 115 and 116: InteroceptionIncludes a wide range
- Page 117 and 118: KeratinA structural protein that pr
- Page 119 and 120: KininsPeptides (e.g. bradykinin) pr
- Page 121 and 122: Linking fasciaPlays a role in augme
- Page 123 and 124: Lymph nodesFilter and clean the lym
- Page 125 and 126: LymphangiogenesisFormation of new l
- Page 127 and 128: Lymphatic drainageThe movement of f
- Page 129 and 130: Lymphatic returnVolume of lymph ret
- Page 131 and 132: Lymphatic transportThe movement of
- Page 133 and 134: Lymphatic vesselsTri-laminar vessel
- Page 135 and 136: LymphocyteThe main cell type found
- Page 137 and 138: MacrophageA type of phagocytic whit
- Page 139 and 140: MastectomySurgical removal of all b
- Page 141 and 142: MechanotransductionThe mechanism by
- Page 143 and 144: MucopolysaccharidesSee GAGs.Myofasc
- Page 145 and 146: Nerve receptorsNerve terminations t
- Page 147 and 148: NeurofasciaTri-laminar, continuous
- Page 149 and 150: Neuropathic painPain due to damage
- Page 151 and 152: NeuropeptidesSignaling molecules th
Hyperexcitation
Neurons firing too easily or too often, which in turn can make something
perceived as painful feel even worse (hyperalgesia), or it can make things hurt
that should not (allodynia). Injury-induced hyperexcitability is not limited to
nociceptors. Hyperexcitability can also develop in myelinated afferents that
normally convey innocuous information (e.g. normal movement and touch) and
under neuropathic conditions, mechanical allodynia can occur.