Advanced Trauma Life Support ATLS Student Course Manual 2018

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INITIAL PATIENT ASSESSMENT 47 A B C D n FIGURE 3-3 Assessment of circulation includes rapidly determining the site of blood loss. In addition to the floor, blood may be in four other places (“on the floor plus four more”): A. the chest; B. the abdomen; C. the pelvis and retroperitoneum; and D. major long bones and soft tissues. assessment with either FAST or diagnostic peritoneal lavage (DPL), and bladder catheterization may all be necessary to determine the source of blood loss (n FIGURES 3-2 and 3-3). Overview of Non-hemorrhagic Shock The category of non-hemorrhagic shock includes cardiogenic shock, cardiac tamponade, tension pneumothorax, neurogenic shock, and septic shock. Even without blood loss, most non-hemorrhagic shock states transiently improve with volume resuscitation. Cardiogenic Shock Myocardial dysfunction can be caused by blunt cardiac injury, cardiac tamponade, an air embolus, or, rarely, myocardial infarction. Suspect a blunt cardiac injury when the mechanism of injury to the thorax involves rapid deceleration. All patients with blunt thoracic trauma need continuous electrocardiographic (ECG) monitoring to detect injury patterns and dysrhythmias. (See Chapter 4: Thoracic Trauma.) The shock state may be secondary to myocardial infarction in the elderly and other high-risk patients, such as those with cocaine intoxication. Therefore, cardiac enzyme levels may assist in diagnosing and treating injured patients in the emergency department (ED), as acute myocardial ischemia may be the precipitating event. Cardiac Tamponade Although cardiac tamponade is most commonly encountered in patients with penetrating thoracic trauma, it can result from blunt injury to the thorax. Tachycardia, muffled heart sounds, and dilated, engorged neck veins with hypotension and insufficient response to fluid therapy suggest cardiac tamponade. n BACK TO TABLE OF CONTENTS
48 CHAPTER 3 n Shock However, the absence of these classic findings does not exclude the presence of this condition. Tension pneumothorax can mimic cardiac tamponade, with findings of distended neck veins and hypotension in both. However, absent breath sounds and hyperresonant percussion are not present with tamponade. Echocardiography may be useful in diagnosing tamponade and valve rupture, but it is often not practical or immediately available in the ED. FAST performed in the ED can identify pericardial fluid, which suggests cardiac tamponade as the cause of shock. Cardiac tamponade is best managed by formal operative intervention, as pericardiocentesis is at best only a temporizing maneuver. (See Chapter 4: Thoracic Trauma.) Tension Pneumothorax Tension pneumothorax is a true surgical emergency that requires immediate diagnosis and treatment. It develops when air enters the pleural space, but a flapvalve mechanism prevents its escape. Intrapleural pressure rises, causing total lung collapse and a shift of the mediastinum to the opposite side, with subsequent impairment of venous return and a fall in cardiac output. Spontaneously breathing patients often manifest extreme tachypnea and air hunger, while mechanically ventilated patients more often manifest hemodynamic collapse. The presence of acute respiratory distress, subcutaneous emphysema, absent unilateral breath sounds, hyperresonance to percussion, and tracheal shift supports the diagnosis of tension pneumothorax and warrants immediate thoracic decompression without waiting for x-ray confirmation of the diagnosis. Needle or finger decompression of tension pneumothorax temporarily relieves this lifethreatening condition. Follow this procedure by placing a chest tube using appropriate sterile technique. (See Appendix G: Breathing Skills and Chest Tube video on MyATLS mobile app.) Neurogenic Shock Isolated intracranial injuries do not cause shock, unless the brainstem is injured. Therefore, the presence of shock in patients with head injury necessitates the search for another cause. Cervical and upper thoracic spinal cord injuries can produce hypotension due to loss of sympathetic tone, which compounds the physiologic effects of hypovolemia. In turn, hypovolemia compounds the physiologic effects of sympathetic denervation. The classic presentation of neurogenic shock is hypotension without tachycardia or cutaneous vasoconstriction. A narrowed pulse pressure is not seen in neurogenic shock. Patients who have sustained a spinal cord injury often have concurrent torso trauma; therefore, patients with known or suspected neurogenic shock are treated initially for hypovolemia. The failure of fluid resuscitation to restore organ perfusion and tissue oxygenation suggests either continuing hemorrhage or neurogenic shock. Advanced techniques for monitoring intravascular volume status and cardiac output may be helpful in managing this complex problem. (See Chapter 7: Spine and Spinal Cord Trauma.) Septic Shock Shock due to infection immediately after injury is uncommon; however, it can occur when a patient’s arrival at the ED is delayed for several hours. Septic shock can occur in patients with penetrating abdominal injuries and contamination of the peritoneal cavity by intestinal contents. Patients with sepsis who also have hypotension and are afebrile are clinically difficult to distinguish from those in hypovolemic shock, as patients in both groups can have tachycardia, cutaneous vasoconstriction, impaired urinary output, decreased systolic pressure, and narrow pulse pressure. Patients with early septic shock can have a normal circulating volume, modest tachycardia, warm skin, near normal systolic blood pressure, and a wide pulse pressure. Hemorrhagic Shock Hemorrhage is the most common cause of shock in trauma patients. The trauma patient’s response to blood loss is made more complex by fluid shifts among the fluid compartments in the body, particularly in the extracellular fluid compartment. Soft tissue injury, even without severe hemorrhage, can result in shifts of fluid to the extracellular compartment. The response to blood loss must be considered in the context of these fluid shifts. Also consider the changes associated with severe, prolonged shock and the pathophysiologic results of resuscitation and reperfusion. Definition of Hemorrhage Hemorrhage is an acute loss of circulating blood volume. Although it can vary considerably, normal adult blood volume is approximately 7% of body weight. For example, a 70-kg male has a circulating blood volume of approximately 5 L. The blood volume n BACK TO TABLE OF CONTENTS
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TENTH EDITION ATLS ® Advanced Trau
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Chair of Committee on Trauma: Ronal
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FOREWORD My first exposure to Advan
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viii PREFACE MyATLS Mobile Applicat
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x PREFACE Gary A. Vercruysse, MD, F
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xii PREFACE Jacqueline Bustraan, MS
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ACKNOWLEDGMENTS It is clear that ma
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xvii ACKNOWLEDGMENTS Bertil Bouillo
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xix ACKNOWLEDGMENTS Oscar Guillamon
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xxi ACKNOWLEDGMENTS Mahesh Misra, M
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xxiii ACKNOWLEDGMENTS James Vosswin
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xxv ACKNOWLEDGMENTS James A. Geilin
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xxvii ACKNOWLEDGMENTS Tone Slåke R
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xxx COURSE OVERVIEW m. Protection o
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xxxii COURSE OVERVIEW Atls and Trau
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xxxiv COURSE OVERVIEW a systematize
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xxxvi COURSE OVERVIEW 69. Switzerla
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xxxviii COURSE OVERVIEW United Stat
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xl COURSE OVERVIEW 67. Hendrickson
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xlii COURSE OVERVIEW 122. Palusci V
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BRIEF CONTENTS Foreword Preface Ack
Page 49 and 50: xlviii DETAILED CONTENTS Teamwork 5
Page 51: l DETAILED CONTENTS Introduction 21
Page 55 and 56: 1 INITIAL ASSESSMENT AND MANAGEMENT
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Page 75 and 76: 2 AIRWAY AND VENTILATORY MANAGEMENT
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Page 95 and 96: 3 SHOCK The first step in the initi
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Page 115 and 116: 4 THORACIC TRAUMA Thoracic injury i
Page 117 and 118: 64 CHAPTER 4 n Thoracic Trauma Thor
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Page 127 and 128: 74 CHAPTER 4 n Thoracic Trauma A B
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Page 133 and 134: 80 CHAPTER 4 n Thoracic Trauma 18.
Page 135 and 136: ABDOMINAL AND 5 PELVIC TRAUMA When
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98 CHAPTER 5 n Abdominal and Pelvic
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100 CHAPTER 5 n Abdominal and Pelvi
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6 HEAD TRAUMA The primary goal of t
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104 CHAPTER 6 n Head Trauma Head in
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106 CHAPTER 6 n Head Trauma fibrous
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108 CHAPTER 6 n Head Trauma n FIGUR
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110 CHAPTER 6 n Head Trauma table 6
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112 CHAPTER 6 n Head Trauma covery.
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120 CHAPTER 6 n Head Trauma necessa
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122 CHAPTER 6 n Head Trauma Use 0.2
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124 CHAPTER 6 n Head Trauma not rea
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126 CHAPTER 6 n Head Trauma 18. Mar
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CHAPTER 7 Outline Objectives iNtrod
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ANATOMY AND PHYSIOLOGY 131 B A n FI
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RIGHT INTERNATIONAL STANDARDS FOR N
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DOCUMENTATION OF SPINAL CORD INJURI
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SPECIFIC TYPES OF SPINAL INJURIES 1
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RADIOGRAPHIC EVALUATION 139 Penetra
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GENERAL MANAGEMENT 141 When the low
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GENERAL MANAGEMENT 143 them to the
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BIBLIOGRAPHY 145 Bibliography 1. Bi
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8 MUSCULOSKELETAL TRAUMA Injuries t
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150 CHAPTER 8 n Musculoskeletal Tra
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9 THERMAL INJURIES The most signifi
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170 CHAPTER 9 n Thermal Injuries Th
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172 CHAPTER 9 n Thermal Injuries Ch
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174 CHAPTER 9 n Thermal Injuries Pi
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176 CHAPTER 9 n Thermal Injuries th
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178 CHAPTER 9 n Thermal Injuries ju
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180 CHAPTER 9 n Thermal Injuries Im
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182 CHAPTER 9 n Thermal Injuries lo
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184 CHAPTER 9 n Thermal Injuries 4.
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10 PEDIATRIC TRAUMA Injury remains
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188 CHAPTER 10 n Pediatric Trauma I
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190 CHAPTER 10 n Pediatric Trauma c
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192 CHAPTER 10 n Pediatric Trauma A
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194 CHAPTER 10 n Pediatric Trauma b
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196 CHAPTER 10 n Pediatric Trauma b
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198 CHAPTER 10 n Pediatric Trauma t
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200 CHAPTER 10 n Pediatric Trauma f
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202 CHAPTER 10 n Pediatric Trauma t
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204 CHAPTER 10 n Pediatric Trauma G
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206 CHAPTER 10 n Pediatric Trauma C
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208 CHAPTER 10 n Pediatric Trauma
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210 CHAPTER 10 n Pediatric Trauma 1
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212 CHAPTER 10 n Pediatric Trauma 6
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CHAPTER 11 Outline Objectives iNtro
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PRIMARY SURVEY WITH RESUSCITATION 2
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PRIMARY SURVEY WITH RESUSCITATION 2
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SPECIFIC INJURIES 221 table 11-6 ph
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BIBLIOGRAPHY 223 comprise only 12%
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12 TRAUMA IN PREGNANCY AND INTIMATE
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228 CHAPTER 12 n Trauma in Pregnanc
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240 CHAPTER 13 n Transfer to Defini
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Appendix A OCULAR TRAUMA OBJECTIVES
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259 APPENDIX A n Ocular Trauma In c
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261 APPENDIX A n Ocular Trauma to t
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Appendix B HYPOTHERMIA AND HEAT INJ
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267 APPENDIX B n Hypothermia and He
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Appendix C TRAUMA CARE IN MASS-CASU
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277 APPENDIX C n Trauma Care in Mas
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Appendix D DISASTER PREPAREDNESS AN
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291 APPENDIX D n Disaster Preparedn
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Pitfall Inadequate security Failed
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Appendix E ATLS AND TRAUMA TEAM RES
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305 APPENDIX E n ATLS and Trauma Te
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Appendix F TRIAGE SCENARIOS OBJECTI
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319 APPENDIX F n Triage Scenarios T
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333 APPENDIX F n Triage Scenarios 4
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Skill Station A AIRWAY Part 1: Basi
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339 APPENDIX G n Skills One-Person
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341 APPENDIX G n Skills STEP 5. Con
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343 APPENDIX G n Skills STEP 13. Co
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346 APPENDIX G n Skills STEP 1. D
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Skill Station C CIRCULATION LEARNIN
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351 APPENDIX G n Skills manual trac
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353 APPENDIX G n Skills STEP 3. Aft
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355 APPENDIX G n Skills STEP 2. If
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358 APPENDIX G n Skills A. Note fac
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360 APPENDIX G n Skills •• Flex
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362 APPENDIX G n Skills Utilization
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366 APPENDIX G n Skills n FIGURE G-
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368 APPENDIX G n Skills less than 1
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372 APPENDIX G n Skills G. Inspect
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374 APPENDIX G n Skills in the inju
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378 INDEX LTA for, 31-32, 32f Malla
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380 INDEX atlanto-occipital disloca
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382 INDEX Frostbite, 181-183, 182f
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384 INDEX Kussmaul’s sign, 69 Lac
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386 INDEX PEA. See Pulseless electr
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388 INDEX for musculoskeletal traum
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390 INDEX Tibial fractures, 163 Tou
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TRAUMA SCORES Correct triage is ess
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394 TRAUMA SCORES on Field Triage,
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396 INJURY PREVENTION Safety Admini
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398 INJURY PREVENTION providers to
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BIOMECHANICS OF INJURY Injuries occ
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402 BIOMECHANICS OF INJURY Ejection
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404 BIOMECHANICS OF INJURY •• A
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406 BIOMECHANICS OF INJURY in a 30-
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408 TETANUS IMMUNIZATION •• Wou
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410 TETANUS IMMUNIZATION Summary Gu
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SAMPLE TRAUMA FLOW SHEET Page 1 of
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414 SAMPLE TRAUMA FLOW SHEET Page 3
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420 SAMPLE TRAUMA FLOW SHEET Some h
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