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PublishedAbstractsPublished Abstracts_________________________________________________________________Akada H, Yan D, Zou H, Hutchison RE, Mohi MG. Conditional expression ofheterozygous or homozygous Jak2V617F from its endogenous promoter induces apolycythemia vera-like disease.Geneva II, Calvert PD. High resolution FRAP of the cilium-localized somatostatin 3receptor reveals the presence of a lateral diffusion barrier at the cilium base.Biophysical Journal 2010; 98(3):376aNajafi M, Calvert PD. 3D-FRAP of PAGFP reveals inhomogeneity in cytoplasmicstructures between the major rod photoreceptor compartments. Biophysical Journal2010; 98(3): 397aWalker BF, French SD, Grant W, Green S, Esterman AJ. Chiropractic interventionsfor low-back pain. Cochrane Database of Systematic Reviews 2010 April, Issue 4Holland MG, Marraffa J, Rosano T.Elevated glycolate levels after unintentionalpediatric ethylene glycol ingestion. Clin Tox 2009Holz GG, Chepurny OG, Leech C, Dzhura I, Kelley GG, Roe MW, Dzhura E, Li X,Schwede F, Genieser HG. A permissive role for protein kinase A in supportof Epac agonist-stimulated human islet insulin secretion. BiophysicalJournal 2010; 98 (3), 680aKnight LD, Fredrickson LA, Stueve MM, Dudley MH. Young drivers overrepresentedin traffic fatalities in Missouri: Retrospective review of young driverdeaths in a large metropolitan jurisdiction, 2006-2008, with an eye toward prevention.Amer J Forensic Medicine and Pathology, March 2010. 31: 1Ko P, Scott J, Mihai A, Barus CE, Grant WD. Comparison of simulation-basedACLS versus traditional ACLS curriculum in third-year medical students. Simulationin Healthcare Winter 2009; 4: 270Liptak GS, Dosa NP, Kennedy J. Youth with Spina Bifida and transitions: Using theWho ICF Model in a nationally representative sample. American Academy of CerebralPalsy and Developmental Medicine, abstracts with programs, Phoenix, Arizona,September 23-26, 2009Liptak GS, Kennedy J, Dosa NP. Transitions in youth with autism in a nationallyrepresentative sample using the Who ICF Model. American Academy of CerebralPalsy and Developmental Medicine, abstracts with programs, Phoenix, Arizona,September 23-26, 2009Roy SK, Vieau C, Gatto L, Snyder K, Maier K, Vodovotz Y, Nieman GF.Histopathology of intestinal mucosa reflects etiology of shock. Critical Care Medicine2009; 37(12); A214- 26 -
Roy SK, Kubiak B, Albert SP, Gatto L, Snyder KP, Vieau C, Maier KG, Nieman GF,Vodovotz Y. Col-3 prevents histologic intestine damage in a clinically applicable largeanimal model of multiple organ dysfunction syndrome (MODS). Critical CareMedicine; 2009; 37(12); A417Roy SK, Vieau CJ, Gatto L, Snyder KP, Maier KG, Nieman GF, Vodovotz Y.Mesenteric lymph node histology reflects similarities in immune response followingshock induced by either ischemia/reperfusion or sepsis. Critical Care Medicine 2009;37(12); A135Roy SK, Vieau CJ, Snyder KP, Maier KG, Gatto L, Vodovotz Y, Nieman GF.Unexpected similarities in severity and acuity of lactic acidosis between sepsis andischemia/reperfusion induced multiple organ dysfunction syndrome. Critical CareMedicine 2009; 37(12); A81Kubiak B, Albert S, Gatto L, Snyder K, Vieau C, Nieman G. A clinically applicableporcine model for the study of the Abdominal Compartment Syndrome (ACS) andMultiple Organ Dysfunction Syndrome (MODS). Acta Clinica Belgica 2009; 4(3):259Kubiak B, Albert S, Gatto L, Snyder K, Maier K, Vieau C, Nieman G.Peritoneal negative pressure therapy reduces both peritoneal and systemicinflammation and prevents Abdominal Compartment Syndrome. Acta ClinicaBelgica 2009; 64(3):260Gatto L, Kubiak B, Albert S, Snyder K, Maier K, Vieau C, Nieman G. Reductionof intestinal histopathology with Peritoneal Negative Pressure Therapy (NPT)in a porcine model of Abdominal Compartment Syndrome (ACS). Acta ClinicaBelgica 2009; 64(3):260Nieman G, Gatto L, Kubiak B, Albert S, Snyder K, Vieau C, Maier K.Peritoneal Ascites, Cytokines, and ACS. Acta Clinica Belgica 2009; 64(3):250Vieau CJ, Roy SK, Gatto L, Kubiak BD, Vodovotz Y, Nieman GF.Ischemia/reperfusion reduces circulating lymphocytes in response toPeritonitis. Critical Care Medicine 2009; 37(12);A208Vieau CJ, Roy SK, Gatto L, Kubiak BD, Vodovotz Y, Nieman GF. Elevatedfibrinogen levels following COL-3 treatment in a 48-hour porcine model ofsepsis and ischemia/reperfusion induced multiple organ dysfunctionsyndrome. Critical Care Medicine 2009; 37(12);A209Roy SK, Vieau C, Gatto L, Snyder K, Maier K, Vodovotz Y, Nieman GF.Histopathology of intestinal mucosa reflects etiology of shock.² CriticalCare Medicine 2009; 37(12); A214Roy SK, Kubiak B, Albert SP, Gatto L, Snyder KP, Vieau C, Maier KG, NiemanGF, Vodovotz Y. Col-3 prevents histologic intestine damage in a clinicallyapplicable large animal model of multiple organ dysfunction syndrome(MODS). Critical Care Medicine 2009;3 7(12);A417- 27 -
Page 1 and 2: THE FACULTYCOMMONS“commons”: (n
Page 3 and 4: Superb EntertainmentDrs. Ron Salets
Page 5: John Andrake, MDPediatricsR. Eugene
Page 8 and 9: National LeadershipRan Anbar, MD, M
Page 10 and 11: P Sebastian Thomas, MD, Anesthesiol
Page 12: Sandra M. Mooney, PhD (PI), Neurosc
Page 15: Biederman J, Petty CR, Wilens TE, S
Page 18 and 19: Faraone SV. Using meta-analysis to
Page 20: Cavender F, Phillips S, Holland MG.
Page 23 and 24: Perl A. Overview of signal processi
Page 25: Procida K, Jørgensen L, Schmitt N,
Page 29 and 30: Books andBook Chapters Books and Bo
Page 31 and 32: PresentationsPresentations_________
Page 33 and 34: NationalNATIONAL___________________
Page 35 and 36: Melissa Ko, MD and Luis Mejico, MD,
Page 37 and 38: Rebecca Garden, PhD, Bioethics & Hu
Page 39 and 40: Lucian Dinu, MD, Anesthesiology, wa
Page 41 and 42: His work has also been published in

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