By: Haydee Cantu and Jennifer Nichols - the UIC Department of ...

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Etiology - Neurochemistry• Dopamine hypothesis• Sx’s due primarily to hyperactive dopamine system inlimbic regions and hypoactive frontal regions• Reason why antipsychotics (D2 antagonist) worked sowell• Reason why dopamine agonists worsened schizophreniasymptoms• The newer atypical antipsychotics introduce a 5-HT role• Glutamate hypothesis• xs glutamate released causes a neurotoxic effect leadingto schizophrenia
“Cerebellar development in childhood onsetschizophrenia and non-psychotic siblings”(Greenstein et al, Psychiatry Research, 2010)• Longitudinal MRI study of regional and totalvolumetric cerebellar differences btwn 3 groups:• Pts dx with COS (COS)• Non-psychotic full siblings of those with COS (SIB)• Typical normal developing brain participants (TD)
Page 1 and 2: By: Haydee Cantu and Jennifer Nicho
Page 3 and 4: DSM-IV Criteria• Same criteria as
Page 5 and 6: Etiology - Genetics• Genetics and
Page 7: Etiology - Neuroanatomical Abnormal
Page 11 and 12: Results• COS diverged from TD in
Page 13 and 14: Clinical Presentation• Negative s
Page 15 and 16: Methods• Subjects: 117 patients w
Page 17 and 18: Psychosocial Factors• Interact wi
Page 19 and 20: Treatment• Combines medication wi

By: Haydee Cantu and Jennifer Nichols - the UIC Department of ...

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