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Effects and clinical potential of very-low-calorie diets (VLCDs) in ...

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DIAB-4541; No <strong>of</strong> Pages 8diabetes research <strong>and</strong> <strong>cl<strong>in</strong>ical</strong> practice xxx (2009) xxx–xxx 7[42] M. Halle, A. Berg, U. Garwers, D. Grathwohl, W. Knisel, J.Keul, Concurrent reductions <strong>of</strong> serum lept<strong>in</strong> <strong>and</strong> lipidsdur<strong>in</strong>g weight loss <strong>in</strong> obese men with type II diabetes, Am.J. Physiol. 277 (1999) E277–282.[43] S. Chearskul, E. Delbridge, A. Shulkes, J. Proietto, A.Kriketos, Effect <strong>of</strong> weight loss <strong>and</strong> ketosis on postpr<strong>and</strong>ialcholecystok<strong>in</strong><strong>in</strong> <strong>and</strong> free fatty acid concentrations, Am. J.Cl<strong>in</strong>. Nutr. 87 (2008) 1238–1246.[44] D.E. Cumm<strong>in</strong>gs, D.S. Weigle, R.S. Frayo, P.A. Breen, M.K. Ma,E.P. Dell<strong>in</strong>ger, et al., Plasma ghrel<strong>in</strong> levels after diet<strong>in</strong>ducedweight loss or gastric bypass surgery, N. Engl. J.Med. 346 (2002) 1623–1630.[45] W.A. Bauman, E. Schwartz, H.G. Rose, H.N. Eisenste<strong>in</strong>, D.W.Johnson, Early <strong>and</strong> long-term effects <strong>of</strong> acute caloricdeprivation <strong>in</strong> obese diabetic patients, Am. J. Med. 85 (1988)38–46.[46] B. Vasquez, E.V. Flock, P.J. Savage, M. Nagulesparan, L.J.Bennion, H.R. Baird, et al., Susta<strong>in</strong>ed reduction <strong>of</strong>prote<strong>in</strong>uria <strong>in</strong> type 2 (non-<strong>in</strong>sul<strong>in</strong>-dependent) diabetesfol<strong>low</strong><strong>in</strong>g diet-<strong>in</strong>duced reduction <strong>of</strong> hyperglycaemia,Diabetologia 26 (1984) 127–133.[47] J.W. Anderson, C.W. Kendall, D.J. Jenk<strong>in</strong>s, Importance <strong>of</strong>weight management <strong>in</strong> type 2 diabetes: review with metaanalysis<strong>of</strong> <strong>cl<strong>in</strong>ical</strong> studies, J. Am. Coll. Nutr. 22 (2003)331–339.[48] T.P. Markovic, A.B. Jenk<strong>in</strong>s, L.V. Campbell, S.M. Furler, E.W.Kraegen, D.J. Chisholm, The determ<strong>in</strong>ants <strong>of</strong> glycemicresponses to diet restriction <strong>and</strong> weight loss <strong>in</strong> obesity <strong>and</strong>NIDDM, Diabetes Care 21 (1998) 687–694.[49] B.H. Goodpaster, D.E. Kelley, R.R. W<strong>in</strong>g, A. Meier, F.L.Thaete, <strong>Effects</strong> <strong>of</strong> weight loss on regional fat distribution<strong>and</strong> <strong>in</strong>sul<strong>in</strong> sensitivity <strong>in</strong> obesity, Diabetes 48 (1999)839–847.[50] R.R. Henry, P. Wallace, J.M. Olefsky, <strong>Effects</strong> <strong>of</strong> weightloss on mechanisms <strong>of</strong> hyperglycemia <strong>in</strong> obesenon-<strong>in</strong>sul<strong>in</strong>-dependent diabetes mellitus, Diabetes 35(1986) 990–998.[51] F. Capstick, B.A. Brooks, C.M. Burns, R.R. Zilkens, K.S.Ste<strong>in</strong>beck, D.K. Yue, Very <strong>low</strong> <strong>calorie</strong> diet (VLCD): a usefulalternative <strong>in</strong> the treatment <strong>of</strong> the obese NIDDM patient,Diabetes Res. Cl<strong>in</strong> Pract. 36 (1997) 105–111.[52] D.K. Yue, Author’s reply: VLCD <strong>in</strong> the treatment <strong>of</strong> obesepatients with Type 2 diabetes: does it make sense? Int.Diabetes Monit. 10 (1998) 11.[53] R.R. W<strong>in</strong>g, M.D. Marcus, R. Salata, L.H. Epste<strong>in</strong>, S.Miaskiewicz, E.H. Blair, <strong>Effects</strong> <strong>of</strong> a <strong>very</strong>-<strong>low</strong>-<strong>calorie</strong>diet on long-term glycemic control <strong>in</strong> obese type 2diabetic subjects, Arch. Intern. Med. 151 (1991)1334–1340.[54] R.R. W<strong>in</strong>g, E. Blair, M. Marcus, L.H. Epste<strong>in</strong>, J. Harvey,Year-long weight loss treatment for obese patients withtype II diabetes: does <strong>in</strong>clud<strong>in</strong>g an <strong>in</strong>termittent <strong>very</strong>-<strong>low</strong><strong>calorie</strong>diet improve outcome? Am. J. Med. 97 (1994)354–362.[55] R.B. Paisey, J. Frost, P. Harvey, A. Paisey, L. Bower, R.M.Paisey, et al., Five year results <strong>of</strong> a prospective <strong>very</strong><strong>low</strong> <strong>calorie</strong> diet or conventional weight loss programme<strong>in</strong> type 2 diabetes, J. Hum. Nutr. Diet. 15 (2002)121–127.[56] R.R. Henry, B. Gumb<strong>in</strong>er, Benefits <strong>and</strong> limitations <strong>of</strong> <strong>very</strong><strong>low</strong>-<strong>calorie</strong>diet therapy <strong>in</strong> obese NIDDM, Diabetes Care 14(1991) 802–823.[57] J.M. Amatruda, J.F. Richeson, S.L. Welle, R.G. Brodows, D.H.Lockwood, The safety <strong>and</strong> efficacy <strong>of</strong> a controlled <strong>low</strong>energy(‘<strong>very</strong>-<strong>low</strong>-<strong>calorie</strong>’) diet <strong>in</strong> the treatment <strong>of</strong> non<strong>in</strong>sul<strong>in</strong>-dependentdiabetes <strong>and</strong> obesity, Arch. Intern. Med.148 (1988) 873–877.[58] B. Gumb<strong>in</strong>er, K.S. Polonsky, W.F. Beltz, K. Griver, P.Wallace, G. Brechtel, et al., <strong>Effects</strong> <strong>of</strong> weight loss <strong>and</strong>reduced hyperglycaemia on the k<strong>in</strong>etics <strong>of</strong> <strong>in</strong>sul<strong>in</strong>secretion <strong>in</strong> obese non-<strong>in</strong>sul<strong>in</strong> dependent diabetesmellitus, J. Cl<strong>in</strong>. Endocr. Metab. 70 (1990) 1594–1602.[59] B. Gumb<strong>in</strong>er, J.A. Wendel, M.P. McDermott, <strong>Effects</strong> <strong>of</strong> dietcomposition <strong>and</strong> ketosis on glycemia dur<strong>in</strong>g <strong>very</strong>-<strong>low</strong>energy-diettherapy <strong>in</strong> obese patients with non-<strong>in</strong>sul<strong>in</strong>dependentdiabetes mellitus, Am. J. Cl<strong>in</strong>. Nutr. 63 (1996)110–115.[60] E.O. Balasse, H.A. Ooms, J.P. Lambilliotte, Evidence for astimulatory effect <strong>of</strong> ketone bodies on <strong>in</strong>sul<strong>in</strong> secretion <strong>in</strong>man, Horm. Metab. Res. 2 (1970) 371–372.[61] R.R. Henry, G. Brechtel, K.H. Lim, <strong>Effects</strong> <strong>of</strong> ketone bodieson carbohydrate metabolism <strong>in</strong> non-<strong>in</strong>sul<strong>in</strong>-dependent(type II) diabetes mellitus, Metabolism 39 (1990) 853–858.[62] R.L. Veech, The therapeutic implications <strong>of</strong> ketone bodies:the effects <strong>of</strong> ketone bodies <strong>in</strong> pathological conditions:ketosis, ketogenic diet, redox states, <strong>in</strong>sul<strong>in</strong> resistance, <strong>and</strong>mitochondrial metabolism, Prostagl<strong>and</strong><strong>in</strong>s Leukot. Essent.Fatty Acids 70 (2004) 309–319.[63] A. Pelletier, A. Tardif, M.H. G<strong>in</strong>gras, J.L. Chiasson, L.Coderre, Chronic exposure to ketone bodies impairsglucose uptake <strong>in</strong> adult cardiomyocytes <strong>in</strong> response to<strong>in</strong>sul<strong>in</strong> but not vanadate: the role <strong>of</strong> PI3-K, Mol. CellBiochem. 296 (2007) 97–108.[64] Z. Liu, W. Long, T. Hillier, L. Saffer, E.J. Barrett, Insul<strong>in</strong>regulation <strong>of</strong> prote<strong>in</strong> metabolism <strong>in</strong> vivo, Diabetes Nutr.Metab. 12 (1999) 421–428.[65] S.M. Nickols-Richardson, M.D. Coleman, J.J. Volpe, K.W.Hosig, Perceived hunger is <strong>low</strong>er <strong>and</strong> weight loss is greater<strong>in</strong> overweight premenopausal women consum<strong>in</strong>g a <strong>low</strong>carbohydrate/high-prote<strong>in</strong>vs high-carbohydrate/<strong>low</strong>-fatdiet, J. Am. Diet Assoc. 105 (2005) 1433–1437.[66] M.I. Uusitupa, M. Laakso, H. Sarlund, H. Maj<strong>and</strong>er, J. Takala,I. Penttila, <strong>Effects</strong> <strong>of</strong> a <strong>very</strong>-<strong>low</strong>-<strong>calorie</strong> diet on metaboliccontrol <strong>and</strong> cardiovascular risk factors <strong>in</strong> the treatment <strong>of</strong>obese non-<strong>in</strong>sul<strong>in</strong>-dependent diabetics, Am. J. Cl<strong>in</strong>. Nutr.51 (1990) 768–773.[67] P. Poirier, T.D. Giles, G.A. Bray, Y. Hong, J.S. Stern, F.X. Pi-Sunyer, et al., Obesity <strong>and</strong> cardiovascular disease:pathophysiology, evaluation, <strong>and</strong> effect <strong>of</strong> weight loss:an update <strong>of</strong> the 1997 American Heart AssociationScientific Statement on Obesity <strong>and</strong> Heart Disease fromthe Obesity Committee <strong>of</strong> the Council on Nutrition,Physical Activity, <strong>and</strong> Metabolism, Circulation 113 (2006)898–918.[68] C. Piantadosi, R. Duncan, A. Nelson, G. Wittert, B. Dundon,M. Worthley, et al., Weight loss <strong>in</strong> type 2 diabetics improvesvascular <strong>and</strong> cardiac contractile function, Heart Lung Circ.17 (2008) S132–S133.[69] R.W. van der Meer, S. Hammer, J.W. Smit, M. Frolich, J.J.Bax, M. Diamant, et al., Short-term caloric restriction<strong>in</strong>duces accumulation <strong>of</strong> myocardial triglycerides <strong>and</strong>decreases left ventricular diastolic function <strong>in</strong> healthysubjects, Diabetes 56 (2007) 2849–2853.[70] S. Hammer, R.W. van der Meer, H.J. Lamb, H.H. de Boer, J.J.Bax, A. de Roos, et al., Short-term flexibility <strong>of</strong> myocardialtriglycerides <strong>and</strong> diastolic function <strong>in</strong> patients with type 2diabetes mellitus, Am. J. Physiol. Endocr<strong>in</strong>ol. Metab. 295(2008) E714–718.[71] S. Hammer, M. Snel, H.J. Lamb, I.M. Jazet, R.W. van derMeer, H. Pijl, et al., Prolonged caloric restriction <strong>in</strong> obesepatients with type 2 diabetes mellitus decreasesmyocardial triglyceride content <strong>and</strong> improvesmyocardial function, J. Am. Coll. Cardiol. 52 (2008)1006–1012.[72] M.C. Lewis, M.L. Phillips, J.P. Slavot<strong>in</strong>ek, L. Kow, C.H.Thompson, J. Toouli, Change <strong>in</strong> liver size <strong>and</strong> fat contentafter treatment with Optifast <strong>very</strong> <strong>low</strong> <strong>calorie</strong> diet, Obes.Surg. 16 (2006) 697–701.Please cite this article <strong>in</strong> press as: S. Baker, et al., <strong>Effects</strong> <strong>and</strong> <strong>cl<strong>in</strong>ical</strong> <strong>potential</strong> <strong>of</strong> <strong>very</strong>-<strong>low</strong>-<strong>calorie</strong> <strong>diets</strong> (<strong>VLCDs</strong>) <strong>in</strong> type 2 diabetes,Diab. Res. Cl<strong>in</strong>. Pract. (2009), doi:10.1016/j.diabres.2009.06.002

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