njit-etd2003-081 - New Jersey Institute of Technology

njit-etd2003-081 - New Jersey Institute of Technology njit-etd2003-081 - New Jersey Institute of Technology

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189 The purpose of this is to show that, although the output for different wavelets varies, the fundamental characteristics of the wavelet transform are the same. Figure 5.29 — 5.33 show the results of these transforms. A band of cones of influence corresponding to each minima and maxima on the BP IIBI signal at the frequency of respiration (16 bpm, 0.266667 Hz) appears in each wavelet transform representation. In addition, the power intensity of the band is as high as the similar band shown in the normal subject case investigated in section 5.2.2. Another band of cones of influence in the very low frequency range (0.01 — 0.15 Hz) also exists with high energy and in longer time (from 10 seconds to 120 seconds, to 180 seconds, to 250 seconds). This matches the trend of the blood pressure IIBI signal that can be seen in figure 5.28 (top panel, signal in green). Among the wavelets, the Morlet wavelet again provides the best representation in terms of time, frequency resolution and meaningful details. The band of the cones of influence at 0.266667 Hz modulated slightly indicating that the BPV is also dominated by the respiration signal and is also not a stationary signal. There are also many interesting observations unveiled from the data and the experimental setup of the normal and COPD subjects. The first was the interplay of the two opposing physiological factors that were simultaneously revealed in the timefrequency analysis of the heart rate variability signal during stressful exercise conditions for normal subjects and during rest for COPD patients. Another was the compensation of the HR in COPD by having a cyclic modulation at about 4 to 5 minutes while normal subjects took longer to have these HR modulations and in a more random nature.

190 Figure 5.29 3D and contour plot of blood pressure spectrum using Morlet wavelet. Figure 5.30 3D and contour plot of blood pressure spectrum using Meyer wavelet.

189<br />

The purpose <strong>of</strong> this is to show that, although the output for different wavelets<br />

varies, the fundamental characteristics <strong>of</strong> the wavelet transform are the same. Figure 5.29<br />

— 5.33 show the results <strong>of</strong> these transforms. A band <strong>of</strong> cones <strong>of</strong> influence corresponding<br />

to each minima and maxima on the BP IIBI signal at the frequency <strong>of</strong> respiration (16<br />

bpm, 0.266667 Hz) appears in each wavelet transform representation. In addition, the<br />

power intensity <strong>of</strong> the band is as high as the similar band shown in the normal subject<br />

case investigated in section 5.2.2. Another band <strong>of</strong> cones <strong>of</strong> influence in the very low<br />

frequency range (0.01 — 0.15 Hz) also exists with high energy and in longer time (from 10<br />

seconds to 120 seconds, to 180 seconds, to 250 seconds). This matches the trend <strong>of</strong> the<br />

blood pressure IIBI signal that can be seen in figure 5.28 (top panel, signal in green).<br />

Among the wavelets, the Morlet wavelet again provides the best representation in terms<br />

<strong>of</strong> time, frequency resolution and meaningful details. The band <strong>of</strong> the cones <strong>of</strong> influence<br />

at 0.266667 Hz modulated slightly indicating that the BPV is also dominated by the<br />

respiration signal and is also not a stationary signal.<br />

There are also many interesting observations unveiled from the data and the<br />

experimental setup <strong>of</strong> the normal and COPD subjects. The first was the interplay <strong>of</strong> the<br />

two opposing physiological factors that were simultaneously revealed in the timefrequency<br />

analysis <strong>of</strong> the heart rate variability signal during stressful exercise conditions<br />

for normal subjects and during rest for COPD patients. Another was the compensation <strong>of</strong><br />

the HR in COPD by having a cyclic modulation at about 4 to 5 minutes while normal<br />

subjects took longer to have these HR modulations and in a more random nature.

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