njit-etd2003-081 - New Jersey Institute of Technology

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147 Table 4.3 Principal Components from Normal and COPD Data Set P1 P2 P3 Categories 0.0351 -0.3625 0.3099 RSP 0.2960 -0.1943 0.0928 HR 0.3800 0.0271 -0.0793 BP 0.2729 -0.2367 0.1637 LF_coh_HR rsp 0.2725 -0.0886 -0.3849 LF_coh_HR_BP 0.3349 0.2178 -0.0244 LF_coh_BP_rsp 0.3416 0.0691 -0.0556 HF_coh_HR_rsp 0.1389 0.1252 -0.5128 HF _ coh _ HR _ BP 0.1924 0.4961 0.0194 HF_coh_BP_rsp 0.3386 -0.2607 0.0482 LF_pcoh_HR_rsp 0.0449 0.0540 -0.0351 LF_pcoh_HR_BP 0.3341 0.1433 0.0701 LF_pcoh_BP_rsp 0.2954 -0.2978 0.2039 HF_pcoh_HR_rsp -0.0170 -0.2281 -0.5558 HF_pcoh_HR_BP 0.1308 0.4691 0.2969 HF_pcoh_BP_rsp
148 4.2.11 Cluster Analysis The same data used for PCA in the previous section were also used for cluster analysis in separating the normal and COPD subjects and severity classification. The data set was entered into the Matlab clustering program. The program executed according to the following syntax: T = clusterdata(X,cutoff) where: X was a matrix of size m by n, interpreted as m observations of n variables. cutoff was a threshold value that determines how the cluster function creates clusters. The value of cutoff determined how clusterdata interpreted it. If 0 < cutoff < 1, cutoff was interpreted as the threshold for the inconsistency coefficient. The inconsistency coefficient quantified the degree of difference between objects in the hierarchical cluster tree. If the inconsistency coefficient of a link was greater than the threshold, the cluster function would use the link as a boundary for a cluster grouping. If cutoff >= 1 cutoff was interpreted as the maximum number of clusters to retain in the hierarchical tree. The output, T, was a vector of size m that identifies, by number, the cluster in which each object was grouped. The program used the clusterdata function to compute the distances between items in the dataset and create a hierarchical cluster tree from the dataset. Finally, the clusterdata function grouped the items in the dataset into the number of cluster specified by the value "cutoff'.
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Copyright Warning & Restrictions Th
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ABSTRACT TIME-FREQUENCY INVESTIGATI
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TIME-FREQUENCY INVESTIGATION OF HEA
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APPROVAL PAGE TIME-FREQUENCY INVEST
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BIOGRAPHICAL SKETCH (Continued) D.A
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ACKNOWLEDGMENT The author wishes to
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TABLE OF CONTENTS Chapter Page 1 IN
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TABLE OF CONTENTS (Continued) Chapt
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LIST OF FIGURES Figure Page 2.1 The
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LIST OF FIGURES (Continued) Figure
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LIST OF FIGURES (Continued) Figure
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ABBREVIATIONS A ABP Arterial Blood
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ABBREVIATIONS (Continued) P PID Pro
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2 mortality and sudden death. [2] B
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4 1) Patients with severe pulmonary
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6 examined for determining the inte
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8 identified using principal compon
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1 0 1. Present the application of t
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12 1.3 Outline of the Dissertation
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CHAPTER 2 PHYSIOLOGY BACKGROUND Bio
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16 Figure 2.2 The systemic and pulm
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18 illustrated in Figure 2.3. The i
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20 2.2 Blood Pressure The force tha
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22 2.4 The Nervous System Human beh
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24 The sympathetic nerve fibers lea
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26 Without these sympathetic and pa
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28 center in the medulla, which con
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30 Figure 2.6 Autonomic innervation
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32 average heart rate was measured
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34 However, they do note that there
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Figure 2.9 The placement of the pos
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38 female. While more men suffer fr
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40 Stage II: Moderate COPD - Worsen
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CHAPTER 3 ENGINEERING BACKGROUND Th
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44 Two common types of time-frequen
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46 STFT: Short-Time Fourier Transfo
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48 3.3 The Analytic Signal and Inst
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50 The advantage of using equation
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52 3.5 Covariance and Invariance Th
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where H(f), S(f) are Fourier transf
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56 Another shortcoming of the spect
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58 should take the kernel of the WD
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60 called the cross Wigner distribu
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62 3.6.3 The Choi-Williams (Exponen
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64 Figure 3.3 Performance of the Ch
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66 [-Ω,Ω ], then its STFT will be
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68 This condition forces that the w
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70 where c is a constant. Thus, the
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Figure 3.5 The time-frequency plane
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74 The measure dadb used in the tra
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76 and the wavelet transform repres
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78 Figure 3.6 Figure depicting the
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80 The final step to obtain the pow
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82 It should be noted that if the w
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84 The normal respiration rate can
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Figure 3.12 Power spectrum of BP II
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RR similar manner to give: When com
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90 when there is significant correl
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92 3.12 Partial Coherence Analysis
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94 after removal of the effects of
Page 125 and 126: 96 The bulk of the theory and appli
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Page 133 and 134: 104 eXogenous input (ARX) was used
Page 135 and 136: 106 The baroreflex, an autonomic re
Page 137 and 138: 108 the principal components are no
Page 139 and 140: 110 The mathematical solution for t
Page 141 and 142: 112 3.15 Cluster Analysis The term
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Page 145 and 146: 116 3.15.5 Squared Euclidian Distan
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Page 149 and 150: 120 Sneath and Sokal used the abbre
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Page 153 and 154: CHAPTER 4 METHODS The purpose of th
Page 155 and 156: 126 4.1.2.1 Autonomic Testing. HR V
Page 157 and 158: 128 of heart rate, blood pressure,
Page 159 and 160: 130 The patients who underwent LVRS
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Page 163 and 164: 134 4.2.3 Power Spectrum Analysis o
Page 165 and 166: 136 weighted-average value of the c
Page 167 and 168: 138 For each given scale a within t
Page 169 and 170: 140 frequency F to the wavelet func
Page 171 and 172: 142 4.2.8 System Identification Ana
Page 173 and 174: 144 In this study a simpler approac
Page 175: 146 Table 4.2 Parameters That Make
Page 179 and 180: 150 viewing the time series of sequ
Page 181 and 182: Figure 5.2 BPV analysis of a COPD s
Page 183 and 184: Figure 5.3 HRV analysis of a normal
Page 185 and 186: Figure 5.4.1 Comparison of the HRV
Page 187 and 188: 158 5.2 Time Frequency Analysis One
Page 189 and 190: Figure 5.5 Test signal with 3 sine
Page 191 and 192: 162 Figure 5.6 (c) CWD of a signal
Page 193 and 194: 164 Figure 5.7 (c) WT (dB4 wavelet)
Page 195 and 196: 166 HRV more information about HRV
Page 197 and 198: 168 Figure 5.9 (c) CWD plots of a n
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Page 201 and 202: 172 The following figures show the
Page 203 and 204: 174 Figure 5.15 CWT (Mexican Hat) H
Page 205 and 206: 176 5.2.5 Best Wavelet Selection fo
Page 207 and 208: 178 Table 5.1 Correlation Indices o
Page 209 and 210: 180 5.2.6 Vagal Tone and Sympathova
Page 211 and 212: 182 These figures basically show th
Page 213 and 214: 184 Figure 5.20 Sympathetic and par
Page 217 and 218: 188 5.2.7 Time-Frequency Analysis (
Page 219 and 220: 190 Figure 5.29 3D and contour plot
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198 Figure 5.42 Sympathetic and par
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Figure 5.44 Plot of raw respiration
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Figure 5.46 The LF partial coherenc
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Figure 5.48 HF partial coherence pl
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Table 5.2 Cross-Spectral Analysis o
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Table 5.3 Cross-Spectral Analysis o
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Figure 5.50 HF coherence of COPD (1
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212 For better presentation of the
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Figure 5.53 Coherence and partial c
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216 2. Interpretation of the transf
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218 covariances of the parameters,
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220 deviations are interpreted as A
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222 Figure 5.58 Bode plot of the HR
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224 In this section a simple model
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226 The data for all 47 COPD subjec
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228 Figure 5.60 Normal and COPD cla
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230 Figure 5.61 Normal and COPD cla
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232 Figure 5.62 Normal classificati
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234 5.7 Cluster Analysis The purpos
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236 Figure 5.64 Severity classifica
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238 both the normal and COPD subjec
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240 In summary, COPD subjects had h
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APPENDIX A EXERCISE PHYSIOLOGY A.1
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244 A.3 Figure Out Your Target Hear
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APPENDIX B ANALYSIS PROGRAM LISTING
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248 4) Click on file, close to exit
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250 • TN 11
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252 B.1.2 Partial Coherence Between
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254
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256 Block Diagram !rime of record K
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258
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260 B.2.2 Time — Frequency Analys
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262 This program provides the STFT
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264 G(:j+1)=G(:,j+1)/(2*sum(G(:j+1)
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266 T=(length(Signa)/sample)/(Times
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268 subplot(3, 1,3), plot(T,E); xla
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270 4. The program creates five out
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272 B.2.3.4 Program to Generate Sym
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274 ylabel('frequency'); title('Ins
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276 The program will run and output
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278 axis([0 1 0 2]); grid on; xlabe
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280 vagal=sum(TFDs(HFC,1:k)); symto
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282 plot(J,symtopar); %plot(A,symto
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284 4. Remove the constant levels a
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286 Make sure the agreement is quit
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288 B.2.6 Principal Components Anal
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290 Columns 12 through 15 'LF_pcoh_
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292 I= 1.0000 -0.0000 -0.0000 -0.00
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294 variances = 3.4083 1.2140 1.141
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296 B.2.7 Cluster Analysis Program
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298 end [R,C]=size(Data); if length
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300 B.2.8 Cross-correlation Program
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302 C.3 Partial coherence of HR and
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304 [13] Madwed, J., and R. Cohen.
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306 [41] Mallat, S. G., "A Theory f
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[70] Tazebay, M.V., R.T. Saliba and
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