the-truth-about-cancer
the-truth-about-cancer the-truth-about-cancer
The Truth About Cancer work, NU is the abbreviation for neutrophils, on any given day they’ll account for anywhere from 50 to 70 percent of your total immune army, which basically your white blood cells. Ty, they can’t see cancer, can’t see it on your best day. The majority of your system cannot see cancer. In fact, the very narrow margin would be made up of natural killer cells and they’re constantly surveying all the cells in your body, healthy cell smiley face, healthy cell, smiley face. Hmm…this cell doesn’t have a smiley face—bada bing, bada boom. That’s the job that naturally kills it. There may be a virus hiding inside that cell. That cell could be mutating, it could be mutating and be benign, it could be mutating and in turning cancerous. It doesn’t matter. No smiley face. Ty: Fascinating way that you put that, very down to earth. You’re the opposite of Emeril. He kicks it up a notch. You pulled it down a notch so that I could understand that. I appreciate that. Now a couple things that you mentioned, you mentioned neutrophils, you mentioned natural NK cells which natural killer cells, you mentioned T-lymphocytes. AJ Lanigan: Cytotoxic T-lymphocytes… Ty: What does cytotoxic mean AJ Lanigan: Cytotoxic means—think of cyto—cytoplasm, that’s the guts of the cell, that’s the innerds. So if something is cytotoxic that means that when it hits a cell it basically blows it up and lets the guts come out. So cells, for example, your NK cells, their job is to keep ramming into that cell that doesn’t have a smiley face. And every time it The Quest for The Cures Page 38
Episode 2: Are You Immune rams into it it releases chemicals into the lining of that target cell until it finally bursts open or lyses—l-y-s-e. It’ll lyse that cell wall and a juice comes out, the cytoplasm. The same thing happens with cytotoxic T- lymphocytes. The difference is the natural killer cell is non-specific generally. It doesn’t care in any way whether or not there’s a herpes virus in there, it doesn’t care if it is herpes I, herpes II, in other words you got a variation of things that could be in there. It just generally doesn’t like anything that doesn’t have a smiley face. The cytotoxic T- lymphocyte, on the other hand, has been trained to look for one specific fingerprint. So that cytotoxic T-lymphocyte that attacks herpes I won’t attack cytomegalovirus, won’t attack roseola, won’t attack herpes II. In other words, one cell one target of attack. B-cells are lymphocytes, T- cells are lymphocytes. On the blood panels you’ve got a CD4. That’s a T4. You got CD8’s, that’s a T-8. They got subsets. Think of the T4 as a four star general. I mean he’s sitting up at the top of the hill and he’s watching what’s going on and troops are coming back and forth and giving him information. Well, he’s got a couple of two star generals. They’re called T-helper cells, also lymphocytes. T-helper subset one, T-helper subset two. Now those two generals take responsibility. The TH2, he goes over and takes care of all those B-cells and antibody production jobs. The TH1, he’s going to stay over here and help regulate all these cellular killing which will take place with those cytotoxic T-lymphocytes. It’ll also help with the lymphocytes that call for memory. So maybe you’ve have the measles. Maybe you’ve had the mumps. You’re never going to have those again. Why Because those T-cells that carry memory, you also need suppressor T-cells. You don’t need a immune system that just fights and wars and kills and destroys because there’s too much inflammation. You don’t want to be living in the middle of a war zone. So a suppressor cell says, hey, cool it down man. We’ve won this fight. Let’s go back, set up, have a good day, have a good rest, and fight again when there’s another challenge. So that TH1 is keeping track of all of that and largely keeps track also of those non-specific cells, again, the macrophages, the neutrophils, the natural killers. How do they regulate them Through what we call cytokines, chemical messengers. Think of them as chemical emails or faxes. And so that’s very important, not just the cells but also cellular communication. All of this working, by the way Ty, we don’t give one thought to it. There is more wisdom in one of these cells than there has been in all the libraries that have ever been or now or every will be. I think that’s pretty cool. I mean I’m old enough to remember Tricky Dick Nixon saying we’re going to have a cure for cancer in ten years. Well, obviously, he wasn’t a math major. So where is it Is there, in fact, even a cure for cancer Again, The Quest for The Cures Page 39
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Episode 2: Are You Immune<br />
rams into it it releases chemicals into <strong>the</strong> lining of that target cell until it<br />
finally bursts open or lyses—l-y-s-e. It’ll lyse that cell wall and a juice<br />
comes out, <strong>the</strong> cytoplasm. The same thing happens with cytotoxic T-<br />
lymphocytes. The difference is <strong>the</strong> natural killer cell is non-specific<br />
generally. It doesn’t care in any way whe<strong>the</strong>r or not <strong>the</strong>re’s a herpes<br />
virus in <strong>the</strong>re, it doesn’t care if it is herpes I, herpes II, in o<strong>the</strong>r words<br />
you got a variation of things that could be in <strong>the</strong>re. It just generally<br />
doesn’t like anything that doesn’t have a smiley face. The cytotoxic T-<br />
lymphocyte, on <strong>the</strong> o<strong>the</strong>r hand, has been trained to look for one specific<br />
fingerprint. So that cytotoxic T-lymphocyte that attacks herpes I won’t<br />
attack cytomegalovirus, won’t attack roseola, won’t attack herpes II. In<br />
o<strong>the</strong>r words, one cell one target of attack. B-cells are lymphocytes, T-<br />
cells are lymphocytes.<br />
On <strong>the</strong> blood panels you’ve got a CD4. That’s a T4. You got CD8’s,<br />
that’s a T-8. They got subsets. Think of <strong>the</strong> T4 as a four star general. I<br />
mean he’s sitting up at <strong>the</strong> top of <strong>the</strong> hill and he’s watching what’s going<br />
on and troops are coming back and forth and giving him information.<br />
Well, he’s got a couple of two star generals. They’re called T-helper<br />
cells, also lymphocytes. T-helper subset one, T-helper subset two. Now<br />
those two generals take responsibility. The TH2, he goes over and takes<br />
care of all those B-cells and antibody production jobs. The TH1, he’s<br />
going to stay over here and help regulate all <strong>the</strong>se cellular killing which<br />
will take place with those cytotoxic T-lymphocytes. It’ll also help with <strong>the</strong><br />
lymphocytes that call for memory.<br />
So maybe you’ve have <strong>the</strong> measles. Maybe you’ve had <strong>the</strong> mumps.<br />
You’re never going to have those again. Why Because those T-cells<br />
that carry memory, you also need suppressor T-cells. You don’t need a<br />
immune system that just fights and wars and kills and destroys because<br />
<strong>the</strong>re’s too much inflammation. You don’t want to be living in <strong>the</strong> middle<br />
of a war zone. So a suppressor cell says, hey, cool it down man. We’ve<br />
won this fight. Let’s go back, set up, have a good day, have a good rest,<br />
and fight again when <strong>the</strong>re’s ano<strong>the</strong>r challenge. So that TH1 is keeping<br />
track of all of that and largely keeps track also of those non-specific<br />
cells, again, <strong>the</strong> macrophages, <strong>the</strong> neutrophils, <strong>the</strong> natural killers.<br />
How do <strong>the</strong>y regulate <strong>the</strong>m Through what we call cytokines, chemical<br />
messengers. Think of <strong>the</strong>m as chemical emails or faxes. And so that’s<br />
very important, not just <strong>the</strong> cells but also cellular communication. All of<br />
this working, by <strong>the</strong> way Ty, we don’t give one thought to it. There is<br />
more wisdom in one of <strong>the</strong>se cells than <strong>the</strong>re has been in all <strong>the</strong> libraries<br />
that have ever been or now or every will be. I think that’s pretty cool. I<br />
mean I’m old enough to remember Tricky Dick Nixon saying we’re going<br />
to have a cure for <strong>cancer</strong> in ten years. Well, obviously, he wasn’t a math<br />
major. So where is it Is <strong>the</strong>re, in fact, even a cure for <strong>cancer</strong> Again,<br />
The Quest for The Cures Page 39