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Episode 6: What Would Doc Do<br />

cells. What we think is <strong>the</strong> proteolytic, which are <strong>the</strong> protein digesting<br />

pancreatic enzymes like trypsin and chymotrypsin actually tear apart <strong>the</strong><br />

cell membranes. You know, cell membranes are lipid, fatty, but <strong>the</strong>y<br />

also have protein molecules that are receptors and pores that allow<br />

nutrients to get in and waste products to get out. That’s how cells<br />

survive with <strong>the</strong>se protein pores in <strong>the</strong> membrane. And <strong>the</strong>se are<br />

proteins like any o<strong>the</strong>r protein. We think <strong>the</strong> enzymes chew <strong>the</strong>m up.<br />

Now <strong>the</strong>y don’t affect normal tissue and Beard said a hundred—in his<br />

book, a 100—in 2011, more than 100 years ago, that <strong>the</strong>re’s a reason<br />

that <strong>cancer</strong> cells have <strong>the</strong> opposite electrical charge than normal cells.<br />

Everyone laughed at him at that time. We now know that it’s true. And<br />

normal cells repulse <strong>the</strong> pancreatic enzymes in <strong>the</strong> blood stream but<br />

<strong>cancer</strong> cells attract <strong>the</strong>m. And <strong>the</strong> enzymes go right to <strong>the</strong> <strong>cancer</strong> cell<br />

and we believe chew up <strong>the</strong> proteins on <strong>the</strong> cell membrane <strong>the</strong> cell<br />

does. So we think it’s that simple. We haven’t had <strong>the</strong> trillions of dollars<br />

of funding to substantiate that. I mean Pour wanted to do that but Nestle<br />

was willing to fund more but <strong>the</strong> key with Nestle is I would have had to<br />

turnover my intellectual property, it would have been a co-ownership.<br />

They wanted—<strong>the</strong>y had <strong>the</strong>ir patent lawyers in <strong>the</strong> US filing patents but<br />

it would have been Nestle and me as co-owners. The problem with that<br />

is Pierre retires and <strong>the</strong> next person may think it’s all quackery. And<br />

Nestle owns my work and I can’t do anything with it. So I said no thank<br />

you, gracious, we’re still friends, Pierre and I. And he understood why.<br />

Ty: Mr. Griffin tell us a little bit <strong>about</strong> <strong>the</strong> role that pancreatic<br />

enzymes might play in prevention and/or treatment of <strong>cancer</strong>.<br />

G. Edward Griffin: Yeah, very important. Thanks for asking that. a<br />

moment ago I said <strong>the</strong>re were two factors, <strong>the</strong> extrinsic factor and <strong>the</strong><br />

intrinsic factor. And we’ve been talking <strong>about</strong> <strong>the</strong> extrinsic factor so far,<br />

<strong>the</strong> food that comes in from outside but <strong>the</strong>re are certain things that are<br />

generated within <strong>the</strong> body that are very, very important. And one of<br />

those happens to be <strong>the</strong> pancreatic enzymes. We’ve got trypsin and<br />

chymotrypsin and o<strong>the</strong>r digestive enzymes that have a special mission<br />

of digesting meat protein. Why is that important It’s a fascinating story.<br />

They had known for a long time that <strong>the</strong>—let me back up a little bit by<br />

saying one of <strong>the</strong> questions that puzzled scientists for a long time is why<br />

doesn’t <strong>the</strong> immune system attack <strong>cancer</strong> cells If you’re working on <strong>the</strong><br />

assumption that <strong>cancer</strong> is something that’s foreign to <strong>the</strong> body, which is<br />

not our assumption, of course. Our assumption is that <strong>cancer</strong> is natural<br />

to <strong>the</strong> body that’s gone array. But <strong>the</strong> orthodox view of <strong>cancer</strong> is<br />

something foreign to <strong>the</strong> body. it’s not supposed to be <strong>the</strong>re. Why<br />

The Quest for The Cures Page 157

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