01 NRDC Dyslexia 1-88 update - Texthelp
01 NRDC Dyslexia 1-88 update - Texthelp
01 NRDC Dyslexia 1-88 update - Texthelp
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42<br />
Research Report<br />
Part two<br />
Explanatory theories of <strong>Dyslexia</strong><br />
Caveat 1 ‘The history of dyslexia research, the well-known heterogeneity of dyslexic<br />
children and the very complexity of the reading process argue against any single<br />
unifying explanation for reading breakdown’ (page 432). Wolf, M. and Bowers, P. G.<br />
(1999). The double-deficit hypothesis for the developmental dyslexias. Journal of<br />
Educational Psychology, 91(3), 415–438.<br />
Caveat 2 ‘A difficulty in learning to read, or dyslexia, should not be viewed as a<br />
condition in itself, but as a symptom of a breakdown in one or more of the various<br />
processes involved’ (page 460). Farmer, M. E. and Klein, R. M. (1995). The evidence for<br />
a temporal processing deficit linked to dyslexia: a review. Psychonomic Bulletin &<br />
Review, 2(4), 460–493.<br />
Caveat 3 ‘Some of the seeming confusion in the study of dyslexia may simply reflect<br />
the fact that complex systems may break down in complex ways’ (page 260).<br />
Seidenberg, M. S. (1992). <strong>Dyslexia</strong> in a computational model of word recognition in<br />
reading. In P. B. Gough & L. C. Ehri & R. Treiman (Eds.), Reading Acquisition.<br />
Hillsdale, NJ: Laurence Erlbaum Associates.<br />
Introduction<br />
The theories reviewed here are attempts to describe what is going wrong for people who have<br />
difficulty in learning to read and write and to explain why it is going wrong for them. Not all of<br />
the theories attempt both tasks. Nevertheless, the different kinds of theory can be fitted into<br />
an inclusive causal model entailing three levels of analysis, one of which is descriptive, one of<br />
which is explanatory at the proximal level and one of which is explanatory at the distal level—<br />
behavioural, cognitive and biological explanations. The original model was developed at the<br />
Medical Research Council’s former Cognitive Development Unit (Morton & Frith, 1995) at<br />
about the same time that the comparable biobehavioural systems model was proposed in the<br />
USA (Fletcher et al., 1995). It has been adopted in accounts of dyslexia in adults (Lee, 2000,<br />
2002) and its usefulness as an explanatory framework is widely acknowledged (Nicolson et<br />
al., 20<strong>01</strong>a; Richards et al., 2002; Snowling, 2000). It has recently been elaborated in a general<br />
model of reading and the influences on reading development (Jackson & Coltheart, 20<strong>01</strong>). In<br />
this form (see Appendix 4), it offers useful points of reference for the present review.<br />
Reading, as most dyslexia theorists use the term, refers to the decoding of single words, but<br />
this is only one stage in the acquisition of literacy. A more elaborate account describes<br />
reading as a cognitive activity accomplished by a mental information-processing system made<br />
up of a number of distinct processing subsystems or component skills (Carr et al., 1990),<br />
where the input is print and the processes applied to the input yield output in the form of<br />
word meaning, syntactic representations of sequences of words and pronunciations (Jackson<br />
& Coltheart, 20<strong>01</strong>). This is not to suppose that anyone believes the ability to recognise and<br />
pronounce words—‘word calling’, or ‘barking at print’—is the final aim of reading instruction;<br />
if the basic skill is to derive meaning from print, then reasoning and discourse are ultimately<br />
what make literacy functional (Rayner et al., 20<strong>01</strong>). That being so, it may help to follow the