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Untitled - D Ank Unlimited

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acute cellular rejection 10 acute disseminated encephalomyelitis<br />

Detectable antibodies with specificity for HIV constituents<br />

gp120, gp160, p24, and p41 are not detectable until at least<br />

6 months following infection. Approximately 33% of the<br />

infected subjects manifest acute AIDS syndrome.<br />

acute cellular rejection<br />

Acute graft rejection mediated by recipient cytotoxic T<br />

lymphocytes and delayed-type hypersensitivity reactions<br />

against allogeneic graft cells.<br />

acute disseminated encephalomyelitis<br />

Brain inflammation that may be a sequela of certain acute<br />

viral infections such as measles in children or following<br />

vaccination. It was reported in some subjects following<br />

smallpox vaccination and in early recipients of rabies<br />

vaccine containing nervous system tissues. Symptoms<br />

include neck stiffness, headache, disorientation, and coma.<br />

Elevated quantities of protein and lymphocytes appear in<br />

the cerebrospinal fluid. Histopathologically, lymphocytes,<br />

plasma cells, and polymorphonuclear leukocytes may form<br />

perivascular infiltrates. The pathological changes are probably<br />

attributable to immune reactivity against the central<br />

nervous system constituent myelin basic protein and may<br />

represent the human equivalent of experimental allergic<br />

encephalomyelitis produced in animals. Refer also to the<br />

animal model of this condition, termed experimental allergic<br />

encephalomyelitis (EAE).<br />

Diffuse erythematous to morbilliform rash in a child with acute graft-vs.host<br />

disease (GVHD).<br />

Diffuse erythematous skin rash in a patient with acute graft-vs.-host<br />

disease (GVHD).<br />

Acute graft-vs.-host reaction. Histologically, there is an intense interface<br />

dermatitis with destruction of basilar cells, particular at the tips of the<br />

rete ridges, incontinence of melanin pigment, and necrosis of individual<br />

epithelial cells, referred to as apoptosis.<br />

Histological appearance of the skin in graft-vs.-host disease with disruption<br />

of the basal cell layer, hyperkeratosis, and beginning sclerotic<br />

change.<br />

Papulosquamous rash in graft-vs.-host disease.

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