EFS12- Book of abstracts - Contact

More documents

Recommendations

Info

SESSION 4: GENETICS OF HOSTS – PLANT RESISTANCE TO FUSARIUM, VARIETY DEVELOPMENT P108 - Effect of phlobaphene accumulation in maize kernel pericarp on Fusarium ear rot levels in Lombardia G. Venturini, G. Assante, L. Babazadeh, D. Salomoni, S. L. Toffolatti, R. Pilu, A. Vercesi Dipartimento di Scienze Agrarie e Ambientali - Produzione Territorio Agroenergia (DISAA), Università degli Studi di Milano,Via G. Celoria 2, 20133 Milano-Italy E-mail: giovanni.venturini@unimi.it Fusarium ear rot (FER) is one the most serious maize fungal diseases especially in Lombardia, the most important maize producer region in Italy. The main aetiological agents of FER are Fusarium species belonging to Gibberella fujikuroi species complex, in particular F. verticillioides and F. proliferatum. FER usually does not lead to significant yield losses, but its causal agents are well known producers of mycotoxins such as fumonisins. Control measures in Italy consist of one - two treatments against the European corn borer, but additional preventive means are required in order to assure a more effective protection. Breeding efforts have been undertaken to increase resistance to FER and in particular phlobaphenes seem to be able to reduce the fumonisin accumulation in kernels. The aim of this study was to assess the effect of pericarp phlobaphenes on FER rating under field conditions. Two hybrids, one with P1-rr allele providing pigmentation in pericarp and the other carrying P1-wr allele without phlobaphenes accumulation in pericarp, were sown in 2011 and 2012 in four different locations in Lombardia. The frequency and severity of FER, together with the incidence of latent infections caused by G. fujikuroi clade and the fumonisin content, were assessed in each genotype at the four field trials. Significant but not univocal differences in FER frequency and severity between the two genotypes were found only in 2012. Latent infections were significantly more frequent on P1-wr genotype in two fields and on P1-rr in the same location for two years. A similar distribution pattern was also found in the fumonisin level. Statistical analysis showed that FER indexes are positively correlated with both latent infections and fumonisin accumulation and significantly influenced not only by the presence of phlobaphenes, but also by the field location and the year of cultivation Keywords: Maize, Phlobaphenes, Fusarium ear rot, Gibberella fujikuroi clade 201
SESSION 4: GENETICS OF HOSTS – PLANT RESISTANCE TO FUSARIUM, VARIETY DEVELOPMENT P109 - Comparative analysis of resistance in Fusarium wilt-resistant and -susceptible watermelons: reinforcement of the structure barrier P. F. Chang 1,2,5 , T. H. Chang 1 , Y. H. Lin 1 , K. S. Chen 3 , J. W. Huang 1 , S. C. Hsiao 4 E-mail: pfchang@nchu.edu.tw Watermelon (Citrullus lanatus) is an economically important crop and was reportedly the third largest vegetable cultivated worldwide. However, the soilborne fungal disease, Fusarium wilt of watermelon, caused by Fusarium oxysporum f. sp. niveum (Fon), is one of the most important restricting factors for watermelon production. A well understanding of the interactions between plant and pathogen can lead to the establishment of more efficient management strategies. The aims of this research were to study mechanisms contributed to Fusarium resistance. The comparative analysis of resistance in the resistant JSB (Chen et al. 2003. Plant Pathology Bulletin 12: 247-254) and susceptible Sugar Baby (SB) watermelon lines was examined to elucidate the resistance mechanism of watermelon to Fon. Results showed that Fon colonization was limited to the area below the shoot base of JSB suggesting that the shoot base is important for Fusarium resistance in JSB. A significant increase in the enzyme activities of phenylalanine ammonia lyase (PAL) and peroxidase (POD), and high amounts of soluble and cell wall-bound phenolics were found in the shoot bases of the resistant JSB line after Fon inoculation. Furthermore, a high level of lignin deposition in the cell wall of a vascular bundle was observed in the shoot bases of Fon-inoculated JSB. In addition, using whole transcriptome sequencing, transcripts of ligase (EC 6.2.1.12) involved in phenylpropanoid biosynthesis was highly expressed in the shoot bases of the resistant JSB than in that of the susceptible SB. The gene for this specific ligase was significantly up-regulated after Fon inoculation in JSB but not in SB. Results strongly suggest that the accumulation of wall-bound phenolics, lignin deposition, the increase of PAL and POD activities, and up-regulated expression of phenylpropanoid pathwayassociated ligase in shoot base of JSB seedlings may reinforce the structure barrier in resistant JSB to resist Fon invasion. Keywords: F. oxysporum f. sp. niveum, Citrullus lanatus, Fusarium resistance, structure barrier 202
Page 1 and 2:
Bordeaux EFS12 12 th European Fusar
Page 4:
TABLE OF CONTENTS Welcome from the
Page 7:
4 SPONSORSHIP The 12 th European Fu
Page 11 and 12:
8 13:00 - 14:00 Lunch 14:00 - 15:00
Page 13 and 14:
10 Session 4: Genetics of Hosts - P
Page 15 and 16:
12 Thursday, May 16 th Session 6: F
Page 17 and 18:
Evidence for birth-and-death evolut
Page 19 and 20:
Future challenges of Fusarium and m
Page 21 and 22:
P35 - Screening deoxynivalenol in o
Page 23 and 24:
P76 - Fusarium verticillioides and
Page 25 and 26:
P113 - Agronomic practices and risk
Page 28:
ORAL PRESENTATIONS 25
Page 32 and 33:
KEYNOTE LECTURE SESSION 1: FUSARIUM
Page 34 and 35:
SESSION 1: FUSARIUM - GENETICS, GEN
Page 36 and 37:
Page 38 and 39:
Page 40 and 41:
Page 42 and 43:
KEYNOTE LECTURE SESSION 2: SECONDAR
Page 44 and 45:
SESSION 2: SECONDARY METABOLITES -
Page 46 and 47:
Page 48 and 49:
Page 50:
Page 53 and 54:
SESSION 3: PATHOGENESIS - EPIDEMIOL
Page 55 and 56:
Page 57 and 58:
Page 59 and 60:
Page 61 and 62:
Page 64 and 65:
KEYNOTE LECTURE 1 SESSION 4: GENETI
Page 66 and 67:
SESSION 4: GENETICS OF HOSTS - PLAN
Page 68 and 69:
Page 70 and 71:
Page 72 and 73:
KEYNOTE LECTURE 2 SESSION 4: GENETI
Page 74 and 75:
Page 76 and 77:
Page 78 and 79:
KEYNOTE LECTURE SESSION 5: DISEASE
Page 80 and 81:
SESSION 5: DISEASE CONTROL AND FORE
Page 82 and 83:
Page 84 and 85:
Page 86:
Page 89 and 90:
SESSION 6: FUTURE CHALLENGE FOR EUR
Page 91 and 92:
SESSION 6: FUTURE CHALLENGE FOR EUR
Page 94:
POSTER PRESENTATIONS 91
Page 97 and 98:
Page 99 and 100:
Page 101 and 102:
Page 103 and 104:
Page 105 and 106:
Page 107 and 108:
Page 109 and 110:
Page 111 and 112:
Page 113 and 114:
Page 115 and 116:
Page 117 and 118:
Page 119 and 120:
Page 121 and 122:
Page 123 and 124:
Page 125 and 126:
Page 127 and 128:
Page 129 and 130:
Page 131 and 132:
Page 133 and 134:
Page 135 and 136:
Page 137 and 138:
134
Page 139 and 140:
Page 141 and 142:
Page 143 and 144:
Page 145 and 146:
Page 147 and 148:
Page 149 and 150:
Page 151 and 152:
Page 153 and 154: SESSION 3: PATHOGENESIS - EPIDEMIOL
Page 177 and 178: SESSION 4: GENETICS OF HOSTS - PLAN
Page 203: SESSION 4: GENETICS OF HOSTS - PLAN
Page 207 and 208: 204
Page 209 and 210: SESSION 5: DISEASE CONTROL AND FORE
Page 233 and 234: SESSION 6: FUTURE CHALLENGE FOR EUR
Page 241 and 242: de Hoog G. S., 89, 114, 236 de Kler
Page 243 and 244: Ponts N., 33, 36, 82, 94, 104, 106,
Page 245 and 246: 242
Page 247 and 248: BOKU-University of Natural Resource
Page 249 and 250: touatisihem03@yahoo.fr He Xinyao In
Page 251 and 252: steve.oliver@bioc.cam.ac. uk Ortega
Page 253 and 254: Subramaniam Gopal Agriculture and A
show all

EFS12- Book of abstracts - Contact

You also want an ePaper? Increase the reach of your titles

Delete template?

Save as template?