EFS12- Book of abstracts - Contact

More documents

Recommendations

Info

SESSION 4: GENETICS OF HOSTS – PLANT RESISTANCE TO FUSARIUM, VARIETY DEVELOPMENT P84 - Evaluation of German winter wheat cultivars for resistance against Fusarium head blight and mycotoxin reduction B. Rodemann Julius Kühn-Institut, Institute of plant protection in field crops and grassland, Messeweg 11/12, 38104 Braunschweig, Germany, E-mail: bernd.rodemann@jki.bund.de Fusarium head blight (FHB) caused by Fusarium graminearum (Schwabe) and Fusarium culmorum (W.G. Smith) belongs to the most damaging disease in cereal crops. Both species were described to produce deoxynivalenol (DON) and DON derivatives and zearalenone depends on the proved isolates. Different studies revealed significant relations between DON content and characteristics of FHB infection. However the relationship between disease symptoms and DON content is not yet well understood in every case. This could be important for minimizing the risk of mycotoxin contamination of grains and foodstuffs. For official cultivar disease ranking, the susceptibility of German cultivars and new breeding lines against FHB was evaluated every year. Under field conditions the FHB nursery was investigated at six different sites in Germany. Furthermore, the FHB and AUDPC were calculated using both incidence and severity. In some cases the DON contamination of the grains was also analyzed. New registered winter wheat cultivars in 2010 and 2011 showed FHB symptoms between 5 to 55% assessed 28 days after inoculation. Investigated grain samples showed DON contents up to 7.4 mg/kg. A strong correlation coefficient (r=0.74**) was observed between visual disease symptoms and mycotoxin concentrations. In accordance to this data a ranking for FHB and DON on a scale from 1-9 (1= low susceptible / low toxin; 9= high susceptible / high toxin) could be made. By cultivation low fusarium susceptible varieties the DON contamination could be reduced by 50-70%. The performance of resistance cultivars in reduction of deoxynivalenol is comparable to the efficacy of fungicides used for fusarium head blight control. Keywords: Fusarium, cultivar, resistance, mycotoxins 177
SESSION 4: GENETICS OF HOSTS – PLANT RESISTANCE TO FUSARIUM, VARIETY DEVELOPMENT P85 - Promising Fusarium head blight resistance in durum wheat N. Prat 1,2,3 , B. Steiner 1 , T. Langin 2 , O. Robert 3 , H. Buerstmayr 1 1 BOKU-University of Natural Resources and Life Sciences Vienna, Department IFA-Tulln, Institute for Biotechnology in Plant Production, Konrad Lorenz Str. 20, 3430 Tulln, Austria; 2 INRA-Université Blaise Pascal, UMR 1095, Genetic, Diversity and Ecophysiology of Cereals, 5 chemin de Beaulieu, 63039 Clermont-Ferrand, Cedex 2, France; 3 Florimond-Desprez, 3 rue Florimond Desprez, BP 41, 59242 Cappelle-en-Pévèle, France Cultivated tetraploid wheat, especially durum wheat (Triticum durum), is highly susceptible to the wide-spread disease Fusarium head blight (FHB). While many resistance QTL have been reported in hexaploid wheat (Triticum aestivum) the QTL identified in tetraploid wheat do not provide satisfactory FHB resistance. To overcome T. durum susceptibility attempts have been made to introgress resistance alleles from wild and cultivated relatives. In this study, back-cross lines derived from crosses of T. durum and FHB resistance sources including Triticum dicoccum (cultivated emmer), Triticum dicoccoides (wild emmer) and Triticum aestivum (bread wheat) have been used as resistant parental lines in several bi- and multi-parental crosses with T.durum. A large population has been developed allowing the evaluation of FHB resistance derived from relatives in an agronomically acceptable durum background. This population was evaluated in 2012 in disease nursery through artificial inoculation at BOKU University in Tulln (Austria). FHB disease symptoms were visually scored, morphological (plant height) and phenological (flowering date) traits were recorded. Based on this first year trial, the population showed a large genetic variation for the different traits evaluated. More interestingly, a large spectrum of response for FHB resistance was observed among the lines ranging from highly resistant to susceptible. These first results are promising and need to be confirmed in 2013 trials. A subset of 500 lines will subsequently be analyzed through both linkage and genome-wide association mapping. The lines will be genotyped in high-density at INRA Clermont-Ferrand (France) using GENTYANE platform and phenotyped at two locations: Florimond-Desprez in Cappelle-en-Pévèle (France) and BOKU University in Tulln (Austria).Through this project, we expect to unveil QTL linked with resistance and/or increased susceptibility and to evaluate the importance of epistatic interactions for FHB resistance in durum wheat. We acknowledge funding of this project by CIFRE (Conventions Industrielles de Formation par la Recherche). Keywords: FHB, disease resistance, tetraploid wheat, durum 178
Page 1 and 2:
Bordeaux EFS12 12 th European Fusar
Page 4:
TABLE OF CONTENTS Welcome from the
Page 7:
4 SPONSORSHIP The 12 th European Fu
Page 11 and 12:
8 13:00 - 14:00 Lunch 14:00 - 15:00
Page 13 and 14:
10 Session 4: Genetics of Hosts - P
Page 15 and 16:
12 Thursday, May 16 th Session 6: F
Page 17 and 18:
Evidence for birth-and-death evolut
Page 19 and 20:
Future challenges of Fusarium and m
Page 21 and 22:
P35 - Screening deoxynivalenol in o
Page 23 and 24:
P76 - Fusarium verticillioides and
Page 25 and 26:
P113 - Agronomic practices and risk
Page 28:
ORAL PRESENTATIONS 25
Page 32 and 33:
KEYNOTE LECTURE SESSION 1: FUSARIUM
Page 34 and 35:
SESSION 1: FUSARIUM - GENETICS, GEN
Page 36 and 37:
Page 38 and 39:
Page 40 and 41:
Page 42 and 43:
KEYNOTE LECTURE SESSION 2: SECONDAR
Page 44 and 45:
SESSION 2: SECONDARY METABOLITES -
Page 46 and 47:
Page 48 and 49:
Page 50:
Page 53 and 54:
SESSION 3: PATHOGENESIS - EPIDEMIOL
Page 55 and 56:
Page 57 and 58:
Page 59 and 60:
Page 61 and 62:
Page 64 and 65:
KEYNOTE LECTURE 1 SESSION 4: GENETI
Page 66 and 67:
SESSION 4: GENETICS OF HOSTS - PLAN
Page 68 and 69:
Page 70 and 71:
Page 72 and 73:
KEYNOTE LECTURE 2 SESSION 4: GENETI
Page 74 and 75:
Page 76 and 77:
Page 78 and 79:
KEYNOTE LECTURE SESSION 5: DISEASE
Page 80 and 81:
SESSION 5: DISEASE CONTROL AND FORE
Page 82 and 83:
Page 84 and 85:
Page 86:
Page 89 and 90:
SESSION 6: FUTURE CHALLENGE FOR EUR
Page 91 and 92:
Page 94:
POSTER PRESENTATIONS 91
Page 97 and 98:
Page 99 and 100:
Page 101 and 102:
Page 103 and 104:
Page 105 and 106:
Page 107 and 108:
Page 109 and 110:
Page 111 and 112:
Page 113 and 114:
Page 115 and 116:
Page 117 and 118:
Page 119 and 120:
Page 121 and 122:
Page 123 and 124:
Page 125 and 126:
Page 127 and 128:
Page 129 and 130: SESSION 2: SECONDARY METABOLITES -
Page 137 and 138: 134
Page 139 and 140: SESSION 3: PATHOGENESIS - EPIDEMIOL
Page 177 and 178: SESSION 4: GENETICS OF HOSTS - PLAN
Page 179: SESSION 4: GENETICS OF HOSTS - PLAN
Page 207 and 208: 204
Page 209 and 210: SESSION 5: DISEASE CONTROL AND FORE
Page 231 and 232:
Page 233 and 234:
Page 235 and 236:
Page 237 and 238:
Page 239 and 240:
Page 241 and 242:
de Hoog G. S., 89, 114, 236 de Kler
Page 243 and 244:
Ponts N., 33, 36, 82, 94, 104, 106,
Page 245 and 246:
242
Page 247 and 248:
BOKU-University of Natural Resource
Page 249 and 250:
touatisihem03@yahoo.fr He Xinyao In
Page 251 and 252:
steve.oliver@bioc.cam.ac. uk Ortega
Page 253 and 254:
Subramaniam Gopal Agriculture and A
show all

EFS12- Book of abstracts - Contact

Create successful ePaper yourself

Delete template?

Save as template?