ANTI-NUTRITIONAL CONSTITUENT OF COLOCASIA ESCULENTA ...

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Nair P., Turjman N., Kessie G., CaIkins B. and Goodman G. (1984) Diet, nutrition and metabolism in populations at high and low risk for colon cancer. American Journal of Clinical Nutrition 40, 927-930. Normen L., Johnsson M, Andersson M, van Gameren Y. and Dutta P. (1999) Plant sterols in vegetables and fruits commonly consmned in Sweden. Eur Journal ofNutrition 38,84-89. Ntanios F., Jones P.J.H. and Frohlich J.J. (1998) Dietary sitostanol reduces plaque formation bnt not lecithin cholesterol acyl transferase activity in rabbits. Atherosclerosis 138, lOl-llO. Ojiako OA. and Nwanjo liU. (2006) Effect of coffee consmnption on cardiovascular risk factors in female volunteers in Owerri, Nigeria. Sci Afr 5, 17-22. Omornyi F.O. (1991) Metabolism in streptozotocin induced rats fed Dikanut (Irvingia gabonensis). Ph.D. thesis, University ofBenin, Benin City. p 53. Oregon State University: Micronutrient Information Centre. (2008) A simplified model of the sodium (Na+), potassium (lC)ATPase pump. [ouline]. Available from: [Accessed 25 February 2008]. Padmaja V., Thankamany V. and Hisham A (1993) Antibacterial, antifungal and anthelmintic activities ofroot barks of Uvaria narum. Journal ofEthnopharmacology 40, 181-186. Pegel K.H. (1997) The importance of sitosterol and sitosterolin in human and animal nutrition. S AftJ Sci 93, 263-268. 229
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ANTI-NUTRITIONAL CONSTITUENT OF COL
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ABSTRACT Colocasia esculenta 1. Sch
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• My colleagues Kayode, Stranger,
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CHAPTER A-2 : MATERIALS AND METHODS
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CHAPTER 8-1-4 : DISCUSSION Bl-A.l I
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C.3.3 EXPERIMENTAL TEST C.3.3.1 Foo
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MakP Makatini purple MakW Makatini
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Figures C3-9 Figure B-1 The activit
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Table C3-3 TableC3-4 Summary ofseru
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crop. Amadumbe is not extensively c
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section of the study therefore inve
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Figure AI-2: The aboveground portio
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Taro is produced in the Pacific Isl
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A.1.4 Anti-nutrients Foods are comp
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essential amino acids not available
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A.l.4.2 Amylase inhibitors In order
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A.l.4.4 Total polyphenols Polypheno
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A.1.4.7 when the levels are high en
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A.1.4.8 coordination sides to inhib
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However,ifsaponins have a triterpen
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A.l.6 nutritional factors naturally
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A.2.2.3 A.2.3 A.2.3.1 A.2.3.2 Speci
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A.2.3.4.2 A.2.3.4.3 A2.3.4.4 In ano
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A.2.3.6.3 \.2.3.6.3.1 \.2.3.6.3.2 L
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sulphuric acid. Absorbance was meas
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Table A3-1a: The moisture and ash c
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Table A3-1c Carbohydrate content (g
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and 4.13 mg 100 g-l DMin the unproc
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Table A3-3c: Fatty acids identified
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Table AJ-4b: The phenolic compounds
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The results of the analysis of alka
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MtW MtP EW EP MakW MakP Figure A3-3
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The crude fat analysis ofZululand C
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The relative proportion ofNa, Ca, K
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TIA indicated that the trypsin inhi
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samples in this study were also hig
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The cyanogen levels for Amadumbe tu
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CHAPTERA-5 CONCLUSION A.5.1 Nutriti
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In conclusion, Section A ofthis stu
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Alfaia S., Ribeiro G., Nobre A, Lui
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Bhattacharyya A, Rai S. and Babu CR
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Chavan U.D., Shahidi F. and Naczk M
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De la Cuadra C., Muzquiz M, Burbano
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Fatty acids. (2008) CyberlipidCente
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Grant G. (1991) Lectins. In: Toxic
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HofmanP., Vuthapanich S., Whiley A,
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Jobnson LT., Gee J.M, Price K.R., C
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MagaJA (1978) Simple phenols and ph
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Milton K. (2003) Micronutrient inta
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Noonan S.C. and Savage G.P. (1999)
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Pathirana C, Gibney MJ. and Taylor
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Quesada C., Bartolome B., Nieto 0.,
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Robertson RN., Highkin H.R., Smydzu
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Sheeba R. and Padmaja G. (1997) Mec
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Thompson L.U. (1988) Antinutrients
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Vijayakumari K, Siddhuraju P. and J
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PURIFICATION AND AMYLASE INHIBITOR
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calcium metalloenzymes, are complet
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Bl.1.2.4 Kunitz-Iike a-amylase inhi
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diabetes mellitus or obesity. Octiv
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B1.2.3.1 Extraction and purificatio
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BI.3.1 BI.3.2.1 Introduction CHAPTE
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B1.3.2.2 Kinetic studies The inhibi
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Bl.4.1 B1.4.2 Introduction CHAPTER
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Bl.4.3.3 results that amylase inhib
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The chemical structure ofits sapoge
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12.1.2.3 Steroidal saponin The trit
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(the in vitro haemolytic activity)
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82.2.3.2 82.2.3.2.1 B2.2.3.2.2 Air-
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82.2.3.3.4 • ion source temperatu
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Figure 82-3.3: Chromatogram ofsapon
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Gamma- and possibly beta-sitosterol
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whereas beta-sitosterol was. It was
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Cheeke PR. (1971) Nutritional and p
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Franco O.L., Rigden D.L., Melo F.R
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Kokiladevi E., Manickam A and Thayu
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Morton R.L., Schroeder RE., Bateman
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Price K.R, Eagles J. and Fenwick GR
Page 197 and 198: Ukpabi U.R and Ukpabi J.U. (2003) P
Page 199 and 200: SECTIONC EFFECTS OF INGESTED BETA-S
Page 201 and 202: C.1.2.2 and fungal cells contain on
Page 203 and 204: Beta-sitosterol has been successful
Page 205: Digestive enzymes do the all import
Page 209 and 210: C.2.1 Introduction CHAPTERC-2 MATER
Page 211 and 212: C.2.2.3.3 C.2.2.4 C.2.2.4.1 C.2.2.4
Page 214: C.3.! Introduction CHAPTERC-3 RESUL
Page 219 and 220: C.3.3.3 Clinical signs The appearan
Page 221 and 222: C.3.3.5 ALT and AST values were sig
Page 223 and 224: .. - Figure C3-5: Representative ph
Page 225: Figure C3-7: Representative photomi
Page 229 and 230: C.4.1 Introduction CHAPTERC-4 DISCU
Page 231: significantly, decreased after beta
Page 234 and 235: decreased by sitosterols as a resul
Page 237 and 238: Bhattacharyya AI
Page 239 and 240: Dablqvist A (1968) Assay ofintestin
Page 242 and 243: Ivorra MD., D'OconMP., Paya M and V
Page 245: Miettinen TA, Tilvis RS. and Kesani
Page 251 and 252: 2.1 Introduction CHAPTER 2 GENERAL
Page 253 and 254: 2.4 Suggestions for future work Fur
Page 255: A.A.3 Enzyme trypsin 10 mg bovine-p
Page 259 and 260: AppendixB DETAILS OF MEmOOOLOGY BA1
Page 261 and 262: sugars were estimated from standard
Page 263 and 264: B.A.l.5.4.5 Liquid Chromatography M
Page 266: B.A.2.4 Tannin [Van-Burden and Robi
Page 269 and 270: The mixture was stirred continuousl
Page 273 and 274: B.C.l Esterification ofbeta-sitoste
Page 275 and 276: AppendixC OLEIC ACID STANDARD 230 2
Page 279: 5 '11I;1 5 2 r - - - • j ,et [:=:
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