E. Coli

Review of Literature
K5 (37.5%), 15 strains O111: K4 (37.5%), 6 strains O128: K6
(15.0%) and 4 strains O88: K4 with a percentage of 10.0%.
Hirsh and Zee (1999) illustrated that colibacillosis of fowl is an
economically important disease caused by invasive strains of E.coli.
the disease takes many forms in fowl depending upon the age of the
host and mode of infection. The egg surface could be contaminated
with potentially pathogenic strains at the time of laying. The bacteria
penetrate the shell and infect the yolk sac. Embryo that survive may
die shortly after hatching, with losses occurring as late as 2 weeks
after hatching fowl may also be infected by the respiratory tract and
develop respiratory or septicemic disease. The course may be rapidly
fetal or chronic, manifested by debilitation, diarrhea and respiratory
distress. Other clinical syndromes seemingly caused by E. coli include
cellulites, synovitis, pericarditis, salpingitis and panophthalmitis.
Gomis et al. (2000) examined 241 broiler birds in Srilanka for
bacteriological examination from different gross lesions, 162 E.coli
were recovered. Twenty one percent of the birds had multiple lesions
due to E.coli. The frequency of detection of these lesions were, 162
(67%) pericarditis, 26 (11%) air saculitis, 24 (10%) hepatitis, 12 (5%)
perihepatitis and 16 (7%) polyserositis. Serogroups O:78, O:85 and O:
88 were distributed among the 32% of typable E.coli.
El-Sayed et al. (2001) isolated pathogenic E.coli from 50
samples of poultry carcasses at shops in Mansoura city, Dakahlia
province positive E.coli samples were 15 with an incidence rate
30.5%. E. coli isolates were serotyped as, 6 strains O55: K59, 3 strains
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