E. Coli

Review of Literature
suggested that E.coli spread rapidly after hatching. Feed is often
contaminated with pathogenic coliform but these can be destroyed by
hot pelliting process. Rodent dropping often contain pathogenic
coliforms.
Blanco et al. (1997) established a study for detecting the
serogroups of E.coli that causes avian colibacillosis in spain. The
serogroups of 625 avian E.coli isolated between 1992-1993 were
determined. The 458 E.coli from chickens with septicemia belonged
to 62 different 0 serogroups; however, 59% were of 18 serogroups (O:
1, O:2, O:5, O:8, O:12, O:14, O:15, O:18, O:20, O:53, O:78, O:81,
O:83, O: 102, O:103, O:115, O:116 and O:132). The high prevalence
of O:18, O:81, O:115, O:116, O:132 isolates was not expected and
many indicate the emergence of five new serogroups associated with
avian colibacillosis not yet reported.
El-Morsi (1998) examined twenty five liver samples from
poultry and found that 5 samples were positive to E.coli by incidence
of 20%. The isolated serotypes of E.coli from liver samples were 2
untypable (40%), 2 belonged to O111:K58 (40%) and one was O126:
K71 (20%).
Fisher et al. (1998) induced E. coli septicemia in broilers in
order to determine if lesions of acute septicemia could be grossly
detected in visceral organs of broiler carcasses. Increased spleen and
liver weight were observed during the acute phase of septicemia. Air
saculitis, pericarditis and periphepatitis were observed also during the
acute phase.
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