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Book of Medical Disorders in Pregnancy - Tintash

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120mmHg<br />

Aortic Value opens<br />

80mmHg<br />

Mitral Value Closes<br />

S4 S1 S2 S3<br />

Fig5.8: Shows heart sounds.<br />

Electrocardiography:<br />

The mean QRS axis shifts to the left <strong>in</strong><br />

the frontal plane\<strong>of</strong> the electrocardiogram.<br />

A "Q" wave may develop <strong>in</strong><br />

lead III, but disappear on deep <strong>in</strong>spiration.<br />

The electrocardiogram is quite useful <strong>in</strong><br />

assess<strong>in</strong>g the presence or absence <strong>of</strong><br />

ventricular hypertrophy and atrial enlargement.<br />

(LBBB) allow the P2 sound<br />

to be heard before the A2 sound dur<strong>in</strong>g<br />

expiration. With LBBB, <strong>in</strong>hala-tion<br />

br<strong>in</strong>gs A2 and P2 closer together where<br />

they cannot be audibly dist<strong>in</strong>-guished.<br />

PR segment<br />

P<br />

QRS Complex<br />

R<br />

PR <strong>in</strong>terval Q QT <strong>in</strong>terval<br />

S<br />

Fig5.9: Shows ECG.<br />

Aortic Value Closes<br />

Aortic Pressure<br />

Mitral Value opens<br />

LA Pressure<br />

LV Pressure<br />

ST segment<br />

Normal adult 12-lead ECG: The<br />

diagnosis <strong>of</strong> the normal electrocar-<br />

T<br />

9<br />

76<br />

diogram is made by exclud<strong>in</strong>g any<br />

recognized abnormality. Its description<br />

is therefore quite lengthy.<br />

Normal s<strong>in</strong>us rhythm:<br />

Each P wave is followed by a QRS, P<br />

waves normal for the subject, P wave<br />

rate 60 - 100 bpm with 10%<br />

= s<strong>in</strong>us arrhythmia.<br />

Normal QRS axis, normal P waves,<br />

height < 2.5 mm <strong>in</strong> lead II and width <<br />

0.11 s <strong>in</strong> lead II for abnormal P waves<br />

see right atrial hypertrophy, left atrial<br />

hypertrophy, atrial premature beat,<br />

hyperkaliemia, normal PR <strong>in</strong>terval and<br />

0.12 to 0.20 s (3 - 5 small squares) for<br />

short PR segment consider Wolff-<br />

Park<strong>in</strong>son-White syndrome or Lown-<br />

Ganong-Lev<strong>in</strong>e syndrome (other causes<br />

- Duchenne muscular dystrophy.<br />

Type II glycogen storage disease<br />

(Pompe's), HOCM), for long PR <strong>in</strong>terval<br />

see first degree heart block and<br />

'trifascicular' block, normal QRS complex,<br />

< 0.12 s duration (3 small squares),<br />

for abnormally wide QRS consider right<br />

or left bundle branch block, ventricular<br />

rhythm, hyperkaliemia, etc. No pathological<br />

Q waves, no evidence <strong>of</strong> left or<br />

right ventricular hypertrophy and normal<br />

QT <strong>in</strong>terval. Calculate the corrected QT<br />

<strong>in</strong>terval (Q TC) by divid<strong>in</strong>g the QT<br />

<strong>in</strong>terval by the square root <strong>of</strong> the<br />

proceed<strong>in</strong>g R - R <strong>in</strong>terval. Normal =<br />

0.42 s. Causes <strong>of</strong> long QT <strong>in</strong>terval, myocardial<br />

<strong>in</strong>farction, myocarditis, diffuse<br />

myocardial disease, hypocalcaemia, hypothyroidism<br />

, subar-achnoid hemorrhage,<br />

<strong>in</strong>tracerebral hemorrhage, drugs<br />

(e.g. sotalol, amiodarone), hereditary,<br />

Romano Ward syndrome (autosomique

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