Book of Medical Disorders in Pregnancy - Tintash
Book of Medical Disorders in Pregnancy - Tintash
Book of Medical Disorders in Pregnancy - Tintash
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120mmHg<br />
Aortic Value opens<br />
80mmHg<br />
Mitral Value Closes<br />
S4 S1 S2 S3<br />
Fig5.8: Shows heart sounds.<br />
Electrocardiography:<br />
The mean QRS axis shifts to the left <strong>in</strong><br />
the frontal plane\<strong>of</strong> the electrocardiogram.<br />
A "Q" wave may develop <strong>in</strong><br />
lead III, but disappear on deep <strong>in</strong>spiration.<br />
The electrocardiogram is quite useful <strong>in</strong><br />
assess<strong>in</strong>g the presence or absence <strong>of</strong><br />
ventricular hypertrophy and atrial enlargement.<br />
(LBBB) allow the P2 sound<br />
to be heard before the A2 sound dur<strong>in</strong>g<br />
expiration. With LBBB, <strong>in</strong>hala-tion<br />
br<strong>in</strong>gs A2 and P2 closer together where<br />
they cannot be audibly dist<strong>in</strong>-guished.<br />
PR segment<br />
P<br />
QRS Complex<br />
R<br />
PR <strong>in</strong>terval Q QT <strong>in</strong>terval<br />
S<br />
Fig5.9: Shows ECG.<br />
Aortic Value Closes<br />
Aortic Pressure<br />
Mitral Value opens<br />
LA Pressure<br />
LV Pressure<br />
ST segment<br />
Normal adult 12-lead ECG: The<br />
diagnosis <strong>of</strong> the normal electrocar-<br />
T<br />
9<br />
76<br />
diogram is made by exclud<strong>in</strong>g any<br />
recognized abnormality. Its description<br />
is therefore quite lengthy.<br />
Normal s<strong>in</strong>us rhythm:<br />
Each P wave is followed by a QRS, P<br />
waves normal for the subject, P wave<br />
rate 60 - 100 bpm with 10%<br />
= s<strong>in</strong>us arrhythmia.<br />
Normal QRS axis, normal P waves,<br />
height < 2.5 mm <strong>in</strong> lead II and width <<br />
0.11 s <strong>in</strong> lead II for abnormal P waves<br />
see right atrial hypertrophy, left atrial<br />
hypertrophy, atrial premature beat,<br />
hyperkaliemia, normal PR <strong>in</strong>terval and<br />
0.12 to 0.20 s (3 - 5 small squares) for<br />
short PR segment consider Wolff-<br />
Park<strong>in</strong>son-White syndrome or Lown-<br />
Ganong-Lev<strong>in</strong>e syndrome (other causes<br />
- Duchenne muscular dystrophy.<br />
Type II glycogen storage disease<br />
(Pompe's), HOCM), for long PR <strong>in</strong>terval<br />
see first degree heart block and<br />
'trifascicular' block, normal QRS complex,<br />
< 0.12 s duration (3 small squares),<br />
for abnormally wide QRS consider right<br />
or left bundle branch block, ventricular<br />
rhythm, hyperkaliemia, etc. No pathological<br />
Q waves, no evidence <strong>of</strong> left or<br />
right ventricular hypertrophy and normal<br />
QT <strong>in</strong>terval. Calculate the corrected QT<br />
<strong>in</strong>terval (Q TC) by divid<strong>in</strong>g the QT<br />
<strong>in</strong>terval by the square root <strong>of</strong> the<br />
proceed<strong>in</strong>g R - R <strong>in</strong>terval. Normal =<br />
0.42 s. Causes <strong>of</strong> long QT <strong>in</strong>terval, myocardial<br />
<strong>in</strong>farction, myocarditis, diffuse<br />
myocardial disease, hypocalcaemia, hypothyroidism<br />
, subar-achnoid hemorrhage,<br />
<strong>in</strong>tracerebral hemorrhage, drugs<br />
(e.g. sotalol, amiodarone), hereditary,<br />
Romano Ward syndrome (autosomique