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2007, Piran, Slovenia

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Environmental Ergonomics XII<br />

Igor B. Mekjavic, Stelios N. Kounalakis & Nigel A.S. Taylor (Eds.), © BIOMED, Ljubljana <strong>2007</strong><br />

([glucose], mmol/L) was measured at the beginning of the experiment and in minute<br />

15 of the hypoxic exposure.<br />

Statistical analysis<br />

Thirty minutes of hypoxia were divided into thirds, and the first 10-minute hypoxic<br />

period was adopted as a stabilization period; the last two thirds of hypoxic exposure<br />

were used for statistical analysis. Differences in HR, SaO2, V, Tty, FeO2, FeCO2, and<br />

[glucose] were assessed with a two-factor analysis of variance (ANOVA) with<br />

repeated measures on both factors. Whenever ANOVA yielded significant<br />

differences, data were further analysed with a Tukey HSD post-hoc test. The level of<br />

0.05) in both trials: 24.9 (0.8)°C in the CHO<br />

trial, and 25.1 (0.9)°C in the control trial. Mean age, height and weight of the subjects<br />

were: 24 (2) years, 173 (10) cm, and 73 (18) kg, respectively. Tympanic temperature<br />

remained similar in both trials (p>0.05) throughout the experiment; it was 36.9<br />

(0.3)°C at the end of first normoxia period in the control, and 36.8 (0.3)°C in the CHO<br />

trial.<br />

Glucose concentration was 4.9 (0.8) mmol/L in the control, and 4.5 (0.8) mmol/L in<br />

the CHO trial at the beginning of the experiment; the difference between the two trials<br />

was not statistically significant (p>0.05). In the middle of hypoxic period, thus 55<br />

minutes following the ingestion, glucose concentration was significantly (p0.05) at 4.6 (1.1) mmol/L in the control trial. The difference between the two trials<br />

was statistically significant (p0.05) between the two trials during normoxia, and decreased in<br />

both trials during the hypoxic exposure. In the control trial, SaO2 decreased from 99<br />

(1) % to 89 (4) % in the second third of hypoxia, and to 86 (6) % in the last third of<br />

hypoxia. In the CHO trial, SaO2 decreased from 99 (1) % to 92 (4) % in the second<br />

third of hypoxia, and to 90 (5) % in the last third of hypoxia (Figure 1). The difference<br />

between the two trials was statistically significant (p

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