michigan hypertension core curriculum - State of Michigan
michigan hypertension core curriculum - State of Michigan
michigan hypertension core curriculum - State of Michigan
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E Circulation<br />
G Neurologic<br />
General appearance<br />
Inspection begins when the patient first enters the clinic. Observe the patient’s behavior<br />
and gait for signs <strong>of</strong> neurologic deficits. Observe respiratory pattern for signs <strong>of</strong> dyspnea on<br />
exertion. Determine the patient body habitus. Central weight gain (apple shape) correlates with<br />
metabolic risk factors for cardiovascular disease.<br />
Waist measurement<br />
Patient should be wearing gown and non-restrictive briefs or underwear. The<br />
measurement should not be made over clothing. The patient should stand erect with<br />
the abdomen relaxed, the arms at the sides and feet together. The measurer faces the<br />
subject and places an inelastic tape around the subject in a horizontal plane at the level<br />
<strong>of</strong> the natural waist. The measurement should be taken at the end <strong>of</strong> a normal expiration<br />
without the tape compressing the skin. Record to the nearest 0.1cm.<br />
Respiration<br />
Respiration affects the level <strong>of</strong> BP, with SBP falling during inspiration. The basis for<br />
this effect is tw<strong>of</strong>old. First, during inspiration, intrathoracic pressure falls, the lungs expand,<br />
and pulmonary venous capacitance increases. This results in increased venous return to the<br />
right ventricle and diminished blood flow to the left ventricle. The decreased left ventricular<br />
preload contributes to the lowered SBP. Second, changes in intrathoracic pressure are directly<br />
transmitted to the intrathoracic aorta. The degree <strong>of</strong> fall in systemic BP is proportional to the<br />
fall in intrathoracic pressure. During the normal respiratory cycle SBP falls between 5 to 10 mm/<br />
Hg from the end <strong>of</strong> expiration to the end <strong>of</strong> inspiration. In asthma, however, this drop is larger<br />
and may exceed 10mm/Hg. The excessive drop in systolic BP during the respiratory cycle<br />
is known as the pulsus paradoxus. Pulsus paradoxus has also been observed in pericardial<br />
tamponade.<br />
Pulse<br />
Manual determination <strong>of</strong> pulse rate is integral to BP evaluation. Pulse rate, rhythm<br />
and pulse contour are evaluated. Tachycardia or bradycardia may reflect intrinsic cardiac<br />
disease, medical illness or reaction to medication. Similarly, rhythm reflects cardiac arrythmias.<br />
The brachial pulse should be compared to the apical impulse (palpated or auscultated). Not<br />
all cardiac contractions are transmitted to the periphery – this is particularly true in special<br />
situations such as atrial fibrillation. Premature ventricular contractions (PVC) result in<br />
uncoordinated ventricular contraction and diminished stroke volume. Thus, only assessing the<br />
presence <strong>of</strong> peripheral pulses may underestimate the true heart rate.<br />
Similarly, patients with pulsus alternans, alternating weak and strong ventricular<br />
contractions result in only half <strong>of</strong> the cardiac contractions being effective. This discrepancy<br />
<strong>of</strong> peripheral and apical rate is known as pulse deficit. Pulsus bisferiens, a double peak in the<br />
pulse, is difficult to palpate, but may manifest as a “split” Korotk<strong>of</strong>f sound.<br />
Palpate pulses in both arms and one leg. Coarctation <strong>of</strong> the aorta usually presents with<br />
equal radial but diminished femoral pulses. Rarely (